Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya Disease

Negative remodeling, characterized by a decrease in the outer diameter of the terminal (C1) segment of the internal carotid artery and the proximal (M1) segment of the middle cerebral artery, is a hallmark of moyamoya disease. However, the role of the disease-susceptibility gene RNF213 in negative r...

Full description

Saved in:
Bibliographic Details
Main Authors: Haruto UCHINO, Masaki ITO, Taku SUGIYAMA, Kota KURISU, Noriyuki FUJIMA, Miki FUJIMURA
Format: Article
Language:English
Published: The Japan Neurosurgical Society 2025-06-01
Series:Neurologia Medico-Chirurgica
Subjects:
Online Access:https://www.jstage.jst.go.jp/article/nmc/65/6/65_2025-0019/_pdf/-char/en
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849703297217200128
author Haruto UCHINO
Masaki ITO
Taku SUGIYAMA
Kota KURISU
Noriyuki FUJIMA
Miki FUJIMURA
author_facet Haruto UCHINO
Masaki ITO
Taku SUGIYAMA
Kota KURISU
Noriyuki FUJIMA
Miki FUJIMURA
author_sort Haruto UCHINO
collection DOAJ
description Negative remodeling, characterized by a decrease in the outer diameter of the terminal (C1) segment of the internal carotid artery and the proximal (M1) segment of the middle cerebral artery, is a hallmark of moyamoya disease. However, the role of the disease-susceptibility gene RNF213 in negative remodeling in moyamoya disease remains unclear. This study investigated the effect of RNF213 p.R4810K polymorphism on the degree of negative remodeling in moyamoya disease. We analyzed 70 hemispheres of 38 adult patients with moyamoya disease who underwent RNF213 p.R4810K gene analysis. Vascular outer diameters of the distal C1 and proximal M1 segments were measured using constructive interference in steady-state images obtained from 3-tesla magnetic resonance imaging. Suzuki stages were determined via cerebral angiography, and comparisons were made between RNF213-mutant and wild-type hemispheres. Among the analyzed hemispheres, 39 (56%) were RNF213-mutant, and 31 were wild-type. Suzuki stages were distributed as follows: 0 in 8 hemispheres, 1-2 in 15, 3-4 in 40, and 5-6 in 7. At stage 3-4, the C1 outer diameter was significantly smaller in RNF213-mutant hemispheres compared to wild-type (median 2.1 vs 2.6 mm, p < 0.05). A significant reduction in vascular outer diameters in the advanced disease stage was observed only in the mutant group between stages 0 and 3-4 (C1: median 3.0 vs 2.1 mm, p < 0.05; M1: median 2.2 vs 1.5 mm, p < 0.001). These findings suggest the association between RNF213 p.R4810K polymorphism and the progression of negative remodeling at the carotid fork in advanced disease stages of moyamoya disease.
format Article
id doaj-art-ee5aa237b39e476db0c0ab5d6fa4d614
institution DOAJ
issn 1349-8029
language English
publishDate 2025-06-01
publisher The Japan Neurosurgical Society
record_format Article
series Neurologia Medico-Chirurgica
spelling doaj-art-ee5aa237b39e476db0c0ab5d6fa4d6142025-08-20T03:17:19ZengThe Japan Neurosurgical SocietyNeurologia Medico-Chirurgica1349-80292025-06-0165629029510.2176/jns-nmc.2025-00192025-0019Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya DiseaseHaruto UCHINO0Masaki ITO1Taku SUGIYAMA2Kota KURISU3Noriyuki FUJIMA4Miki FUJIMURA5Department of Neurosurgery, Hokkaido University Graduate School of MedicineDepartment of Neurosurgery, Hokkaido University Graduate School of MedicineDepartment of Neurosurgery, Hokkaido University Graduate School of MedicineDepartment of Neurosurgery, Hokkaido University Graduate School of MedicineDepartment of Radiology, Hokkaido University Graduate School of MedicineDepartment of Neurosurgery, Hokkaido University Graduate School of MedicineNegative remodeling, characterized by a decrease in the outer diameter of the terminal (C1) segment of the internal carotid artery and the proximal (M1) segment of the middle cerebral artery, is a hallmark of moyamoya disease. However, the role of the disease-susceptibility gene RNF213 in negative remodeling in moyamoya disease remains unclear. This study investigated the effect of RNF213 p.R4810K polymorphism on the degree of negative remodeling in moyamoya disease. We analyzed 70 hemispheres of 38 adult patients with moyamoya disease who underwent RNF213 p.R4810K gene analysis. Vascular outer diameters of the distal C1 and proximal M1 segments were measured using constructive interference in steady-state images obtained from 3-tesla magnetic resonance imaging. Suzuki stages were determined via cerebral angiography, and comparisons were made between RNF213-mutant and wild-type hemispheres. Among the analyzed hemispheres, 39 (56%) were RNF213-mutant, and 31 were wild-type. Suzuki stages were distributed as follows: 0 in 8 hemispheres, 1-2 in 15, 3-4 in 40, and 5-6 in 7. At stage 3-4, the C1 outer diameter was significantly smaller in RNF213-mutant hemispheres compared to wild-type (median 2.1 vs 2.6 mm, p < 0.05). A significant reduction in vascular outer diameters in the advanced disease stage was observed only in the mutant group between stages 0 and 3-4 (C1: median 3.0 vs 2.1 mm, p < 0.05; M1: median 2.2 vs 1.5 mm, p < 0.001). These findings suggest the association between RNF213 p.R4810K polymorphism and the progression of negative remodeling at the carotid fork in advanced disease stages of moyamoya disease.https://www.jstage.jst.go.jp/article/nmc/65/6/65_2025-0019/_pdf/-char/enmoyamoya diseasenegative remodelingrnf213shrinkage
spellingShingle Haruto UCHINO
Masaki ITO
Taku SUGIYAMA
Kota KURISU
Noriyuki FUJIMA
Miki FUJIMURA
Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya Disease
Neurologia Medico-Chirurgica
moyamoya disease
negative remodeling
rnf213
shrinkage
title Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya Disease
title_full Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya Disease
title_fullStr Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya Disease
title_full_unstemmed Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya Disease
title_short Association between RNF213 p.R4810K and Progression of Cerebral Artery Negative Remodeling in Moyamoya Disease
title_sort association between rnf213 p r4810k and progression of cerebral artery negative remodeling in moyamoya disease
topic moyamoya disease
negative remodeling
rnf213
shrinkage
url https://www.jstage.jst.go.jp/article/nmc/65/6/65_2025-0019/_pdf/-char/en
work_keys_str_mv AT harutouchino associationbetweenrnf213pr4810kandprogressionofcerebralarterynegativeremodelinginmoyamoyadisease
AT masakiito associationbetweenrnf213pr4810kandprogressionofcerebralarterynegativeremodelinginmoyamoyadisease
AT takusugiyama associationbetweenrnf213pr4810kandprogressionofcerebralarterynegativeremodelinginmoyamoyadisease
AT kotakurisu associationbetweenrnf213pr4810kandprogressionofcerebralarterynegativeremodelinginmoyamoyadisease
AT noriyukifujima associationbetweenrnf213pr4810kandprogressionofcerebralarterynegativeremodelinginmoyamoyadisease
AT mikifujimura associationbetweenrnf213pr4810kandprogressionofcerebralarterynegativeremodelinginmoyamoyadisease