Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkers

Abstract Posttraumatic Stress Disorder (PTSD) is a heterogeneous mental health disorder that can develop following a traumatic experience. Understanding its neurobiological basis is crucial to advance early diagnosis and treatment. Electroencephalography (EEG) can be used to explore the neurobiologi...

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Main Authors: Ashritha Peddi, Mohammad S. E. Sendi, Sean T. Minton, Ryan Langhinrichsen-Rohling, Cecilia A. Hinojosa, Emma West, Kerry J. Ressler, Vince D. Calhoun, Sanne J. H. van Rooij
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Language:English
Published: Nature Portfolio 2025-02-01
Series:Scientific Reports
Online Access:https://doi.org/10.1038/s41598-025-88426-1
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author Ashritha Peddi
Mohammad S. E. Sendi
Sean T. Minton
Ryan Langhinrichsen-Rohling
Cecilia A. Hinojosa
Emma West
Kerry J. Ressler
Vince D. Calhoun
Sanne J. H. van Rooij
author_facet Ashritha Peddi
Mohammad S. E. Sendi
Sean T. Minton
Ryan Langhinrichsen-Rohling
Cecilia A. Hinojosa
Emma West
Kerry J. Ressler
Vince D. Calhoun
Sanne J. H. van Rooij
author_sort Ashritha Peddi
collection DOAJ
description Abstract Posttraumatic Stress Disorder (PTSD) is a heterogeneous mental health disorder that can develop following a traumatic experience. Understanding its neurobiological basis is crucial to advance early diagnosis and treatment. Electroencephalography (EEG) can be used to explore the neurobiological basis of PTSD. However, only limited research has explored mobile EEG, which is important for scalability. This proof-of-concept study delves into mobile EEG-derived biomarkers for posttraumatic stress (PTS) symptom severity and their potential implications. Participants with partial PTSD, defined as meeting for at least three out of four symptom clusters, including hyperarousal symptoms, were enrolled in the study. Over four weeks, we measured PTS symptom severity using the PTSD checklist for DSM-5 (PCL-5) at multiple timepoints, and we recorded multiple EEG sessions from 21 individuals using a mobile EEG device. In total, we captured 38 EEG sessions, each comprising two recordings (“Recording A” and “Recording B”) that lasted approximately 180 s, to evaluate reproducibility. Next, we extracted Shannon entropy, as a measure of the brain flexibility and complexity of the signal and spectral power for the fronto-temporal regions of interest, including electrodes at AF3, AF4, T7, and T8 for each EEG recording session. We calculated the partial correlation between the EEG variables and PCL- 5 measured closest to the EEG session, using age, sex, and the grouping variable ‘batch’ as covariates. We observed a significant negative correlation between Shannon entropy in fronto- temporal regions and PCL-5 scores. Specifically, this association was evident in the AF3 (r = -0.456, FDR-corrected p = 0.01), AF4 (r = -0.362, FDR-corrected p = 0.04), and T7 (r = -0.472, FDR-corrected p = 0.01) regions. Additionally, we found a significant negative association between the alpha power estimated from AF4 and PCL-5 (r = -0.429, FDR-corrected p = 0.04). Our findings suggest that EEG markers acquired using a mobile EEG device are associated with PTS symptom severity, offering valuable insights into the neurobiological mechanisms underlying PTSD and highlighting the potential benefits of this innovative technology in assessing and monitoring PTSD.
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spelling doaj-art-ee2a7ba9544b4ac1ae0e8f60bbb1e8a02025-08-20T02:43:15ZengNature PortfolioScientific Reports2045-23222025-02-011511910.1038/s41598-025-88426-1Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkersAshritha Peddi0Mohammad S. E. Sendi1Sean T. Minton2Ryan Langhinrichsen-Rohling3Cecilia A. Hinojosa4Emma West5Kerry J. Ressler6Vince D. Calhoun7Sanne J. H. van Rooij8Georgia State UniversityTri-Institutional Center for Translational Research in Neuroimaging and Data Science, Georgia State UniversityGeorgia Institute of TechnologyEmory UniversityDepartment of Psychiatry and Behavioral Sciences, Emory University School of MedicineDepartment of Psychiatry and Behavioral Sciences, Emory University School of MedicineDepartment of Psychiatry and Behavioral Sciences, Emory University School of MedicineDepartment of Psychiatry and Behavioral Sciences, Emory University School of MedicineDivision of Depression and Anxiety, McLean HospitalGeorgia State UniversityDepartment of Psychiatry and Behavioral Sciences, Emory University School of MedicineAbstract Posttraumatic Stress Disorder (PTSD) is a heterogeneous mental health disorder that can develop following a traumatic experience. Understanding its neurobiological basis is crucial to advance early diagnosis and treatment. Electroencephalography (EEG) can be used to explore the neurobiological basis of PTSD. However, only limited research has explored mobile EEG, which is important for scalability. This proof-of-concept study delves into mobile EEG-derived biomarkers for posttraumatic stress (PTS) symptom severity and their potential implications. Participants with partial PTSD, defined as meeting for at least three out of four symptom clusters, including hyperarousal symptoms, were enrolled in the study. Over four weeks, we measured PTS symptom severity using the PTSD checklist for DSM-5 (PCL-5) at multiple timepoints, and we recorded multiple EEG sessions from 21 individuals using a mobile EEG device. In total, we captured 38 EEG sessions, each comprising two recordings (“Recording A” and “Recording B”) that lasted approximately 180 s, to evaluate reproducibility. Next, we extracted Shannon entropy, as a measure of the brain flexibility and complexity of the signal and spectral power for the fronto-temporal regions of interest, including electrodes at AF3, AF4, T7, and T8 for each EEG recording session. We calculated the partial correlation between the EEG variables and PCL- 5 measured closest to the EEG session, using age, sex, and the grouping variable ‘batch’ as covariates. We observed a significant negative correlation between Shannon entropy in fronto- temporal regions and PCL-5 scores. Specifically, this association was evident in the AF3 (r = -0.456, FDR-corrected p = 0.01), AF4 (r = -0.362, FDR-corrected p = 0.04), and T7 (r = -0.472, FDR-corrected p = 0.01) regions. Additionally, we found a significant negative association between the alpha power estimated from AF4 and PCL-5 (r = -0.429, FDR-corrected p = 0.04). Our findings suggest that EEG markers acquired using a mobile EEG device are associated with PTS symptom severity, offering valuable insights into the neurobiological mechanisms underlying PTSD and highlighting the potential benefits of this innovative technology in assessing and monitoring PTSD.https://doi.org/10.1038/s41598-025-88426-1
spellingShingle Ashritha Peddi
Mohammad S. E. Sendi
Sean T. Minton
Ryan Langhinrichsen-Rohling
Cecilia A. Hinojosa
Emma West
Kerry J. Ressler
Vince D. Calhoun
Sanne J. H. van Rooij
Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkers
Scientific Reports
title Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkers
title_full Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkers
title_fullStr Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkers
title_full_unstemmed Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkers
title_short Towards predicting posttraumatic stress symptom severity using portable EEG-derived biomarkers
title_sort towards predicting posttraumatic stress symptom severity using portable eeg derived biomarkers
url https://doi.org/10.1038/s41598-025-88426-1
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