Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutations

Propionic acidemia (PA) is an inherited metabolic disease caused by the deficiency of one of the four biotin-dependent enzymes propionyl-CoA carboxylase (PCC), and is characterized by coma and death in unrecognized patients, additionally late diagnosis leads to severe developmental delay and neurol...

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Main Authors: Deniz Kör, Berna Şeker-Yılmaz, Fatma Derya Bulut, Sebile Kılavuz, Murat Öktem, Serdar Ceylaner, Dinçer Yıldızdaş, Neslihan Önenli-Mungan
Format: Article
Language:English
Published: Hacettepe University Institute of Child Health 2019-06-01
Series:The Turkish Journal of Pediatrics
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Online Access:https://turkjpediatr.org/article/view/699
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author Deniz Kör
Berna Şeker-Yılmaz
Fatma Derya Bulut
Sebile Kılavuz
Murat Öktem
Serdar Ceylaner
Dinçer Yıldızdaş
Neslihan Önenli-Mungan
author_facet Deniz Kör
Berna Şeker-Yılmaz
Fatma Derya Bulut
Sebile Kılavuz
Murat Öktem
Serdar Ceylaner
Dinçer Yıldızdaş
Neslihan Önenli-Mungan
author_sort Deniz Kör
collection DOAJ
description Propionic acidemia (PA) is an inherited metabolic disease caused by the deficiency of one of the four biotin-dependent enzymes propionyl-CoA carboxylase (PCC), and is characterized by coma and death in unrecognized patients, additionally late diagnosis leads to severe developmental delay and neurological sequels. Manifestations of PA over time can include growth impairment, intellectual disability, seizures, basal ganglia lesions, pancreatitis, and cardiomyopathy. Other rarely reported complications include optic atrophy, hearing loss, premature ovarian insufficiency, and chronic renal failure. Mutations in PCCA-PCCB genes cause the clinically heterogeneous disease of PA. In this study, we investigate the mutation spectrum of PCCAPCCB genes and phenotypic features of 27 Turkish patients with PA from the South and Southeast parts of Turkey. We report 12 novel PA mutations, five affecting the PCCA gene and 7 affecting the PCCB gene.
format Article
id doaj-art-edee5b5b8b6a4d3b9e352bed1702ff0c
institution DOAJ
issn 0041-4301
2791-6421
language English
publishDate 2019-06-01
publisher Hacettepe University Institute of Child Health
record_format Article
series The Turkish Journal of Pediatrics
spelling doaj-art-edee5b5b8b6a4d3b9e352bed1702ff0c2025-08-20T03:00:53ZengHacettepe University Institute of Child HealthThe Turkish Journal of Pediatrics0041-43012791-64212019-06-0161310.24953/turkjped.2019.03.003Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutationsDeniz Kör0Berna Şeker-Yılmaz1Fatma Derya Bulut2Sebile Kılavuz3Murat Öktem4Serdar Ceylaner5Dinçer Yıldızdaş6Neslihan Önenli-Mungan7Divisions of Pediatric Metabolism and Nutrition, Adana, Turkey.Department of Pediatric Metabolism, Mersin City Hospital, Mersin, Turkey.Department of Pediatric Metabolism, Adana City Hospital, Adana, Turkey.Divisions of Pediatric Metabolism and Nutrition, Adana, Turkey.Düzen Biochemistry Laboratories, Ankara, Turkey.Intergen Genetics Laboratories, Ankara, Turkey.Divisions of Pediatric Intensive Care Unit, Çukurova University Faculty of Medicine, Adana, Turkey.Divisions of Pediatric Metabolism and Nutrition, Adana, Turkey. Propionic acidemia (PA) is an inherited metabolic disease caused by the deficiency of one of the four biotin-dependent enzymes propionyl-CoA carboxylase (PCC), and is characterized by coma and death in unrecognized patients, additionally late diagnosis leads to severe developmental delay and neurological sequels. Manifestations of PA over time can include growth impairment, intellectual disability, seizures, basal ganglia lesions, pancreatitis, and cardiomyopathy. Other rarely reported complications include optic atrophy, hearing loss, premature ovarian insufficiency, and chronic renal failure. Mutations in PCCA-PCCB genes cause the clinically heterogeneous disease of PA. In this study, we investigate the mutation spectrum of PCCAPCCB genes and phenotypic features of 27 Turkish patients with PA from the South and Southeast parts of Turkey. We report 12 novel PA mutations, five affecting the PCCA gene and 7 affecting the PCCB gene. https://turkjpediatr.org/article/view/699PCCAPCCBPropionic acidemiaclinical featuresnovel mutation
spellingShingle Deniz Kör
Berna Şeker-Yılmaz
Fatma Derya Bulut
Sebile Kılavuz
Murat Öktem
Serdar Ceylaner
Dinçer Yıldızdaş
Neslihan Önenli-Mungan
Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutations
The Turkish Journal of Pediatrics
PCCA
PCCB
Propionic acidemia
clinical features
novel mutation
title Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutations
title_full Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutations
title_fullStr Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutations
title_full_unstemmed Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutations
title_short Clinical features of 27 Turkish Propionic acidemia patients with 12 novel mutations
title_sort clinical features of 27 turkish propionic acidemia patients with 12 novel mutations
topic PCCA
PCCB
Propionic acidemia
clinical features
novel mutation
url https://turkjpediatr.org/article/view/699
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AT sebilekılavuz clinicalfeaturesof27turkishpropionicacidemiapatientswith12novelmutations
AT muratoktem clinicalfeaturesof27turkishpropionicacidemiapatientswith12novelmutations
AT serdarceylaner clinicalfeaturesof27turkishpropionicacidemiapatientswith12novelmutations
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