Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature Review

Chronic myeloid leukemia (CML) is a myeloproliferative neoplasm driven by the BCR::ABL1 fusion gene and typically progresses through chronic, accelerated, and blast crisis phases. While blast crises often present with myeloid or lymphoid phenotypes, transformation to acute undifferentiated leukemia...

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Main Authors: Oussama Grari, Assya Khermach, Nouhaila Chahid, Nisma Douzi, Amina Himri, Abdelilah Berhili, Mounia Slaoui, Nabiha Trougouty, Mohammed Bensalah, Rachid Seddik
Format: Article
Language:English
Published: Wolters Kluwer Medknow Publications 2025-04-01
Series:Journal of Applied Hematology
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Online Access:https://journals.lww.com/10.4103/joah.joah_53_25
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author Oussama Grari
Assya Khermach
Nouhaila Chahid
Nisma Douzi
Amina Himri
Abdelilah Berhili
Mounia Slaoui
Nabiha Trougouty
Mohammed Bensalah
Rachid Seddik
author_facet Oussama Grari
Assya Khermach
Nouhaila Chahid
Nisma Douzi
Amina Himri
Abdelilah Berhili
Mounia Slaoui
Nabiha Trougouty
Mohammed Bensalah
Rachid Seddik
author_sort Oussama Grari
collection DOAJ
description Chronic myeloid leukemia (CML) is a myeloproliferative neoplasm driven by the BCR::ABL1 fusion gene and typically progresses through chronic, accelerated, and blast crisis phases. While blast crises often present with myeloid or lymphoid phenotypes, transformation to acute undifferentiated leukemia (AUL) is exceedingly rare and presents significant diagnostic and therapeutic challenges. We describe a 41-year-old female with CML whose disease transformed to AUL during blast crisis. Initially treated with imatinib, therapy was discontinued due to loss of response, followed by a brief, intolerant course of nilotinib. After 2 years without treatment, she presented with general deterioration, anemia, and splenomegaly. Laboratory findings revealed hyperleukocytosis, severe thrombocytopenia, and 27% undifferentiated blasts on peripheral blood smear; bone marrow aspiration confirmed 53% blasts with negative myeloperoxidase staining. Immunophenotyping confirmed AUL, showing blasts with weak CD45 expression, CD34 positivity, HLA-DR positivity, and absence of lineage-specific markers, whereas cytogenetic analysis revealed a complex karyotype. The patient was treated with a 3 + 7 chemotherapy regimen but developed infectious complications, ultimately succumbing to cardiovascular arrest. This case highlights the complexity and poor prognosis of atypical CML transformations. It underscores the need for a deeper understanding of blast crisis mechanisms and innovative therapeutic strategies to improve patient care and outcomes.
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spelling doaj-art-ed8defb9295043bb990cda5c6d90bae32025-08-20T02:44:53ZengWolters Kluwer Medknow PublicationsJournal of Applied Hematology1658-51272454-69762025-04-0116217117510.4103/joah.joah_53_25Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature ReviewOussama GrariAssya KhermachNouhaila ChahidNisma DouziAmina HimriAbdelilah BerhiliMounia SlaouiNabiha TrougoutyMohammed BensalahRachid SeddikChronic myeloid leukemia (CML) is a myeloproliferative neoplasm driven by the BCR::ABL1 fusion gene and typically progresses through chronic, accelerated, and blast crisis phases. While blast crises often present with myeloid or lymphoid phenotypes, transformation to acute undifferentiated leukemia (AUL) is exceedingly rare and presents significant diagnostic and therapeutic challenges. We describe a 41-year-old female with CML whose disease transformed to AUL during blast crisis. Initially treated with imatinib, therapy was discontinued due to loss of response, followed by a brief, intolerant course of nilotinib. After 2 years without treatment, she presented with general deterioration, anemia, and splenomegaly. Laboratory findings revealed hyperleukocytosis, severe thrombocytopenia, and 27% undifferentiated blasts on peripheral blood smear; bone marrow aspiration confirmed 53% blasts with negative myeloperoxidase staining. Immunophenotyping confirmed AUL, showing blasts with weak CD45 expression, CD34 positivity, HLA-DR positivity, and absence of lineage-specific markers, whereas cytogenetic analysis revealed a complex karyotype. The patient was treated with a 3 + 7 chemotherapy regimen but developed infectious complications, ultimately succumbing to cardiovascular arrest. This case highlights the complexity and poor prognosis of atypical CML transformations. It underscores the need for a deeper understanding of blast crisis mechanisms and innovative therapeutic strategies to improve patient care and outcomes.https://journals.lww.com/10.4103/joah.joah_53_25acute undifferentiated leukemiablast crisischronic myeloid leukemiatyrosine kinase inhibitors
spellingShingle Oussama Grari
Assya Khermach
Nouhaila Chahid
Nisma Douzi
Amina Himri
Abdelilah Berhili
Mounia Slaoui
Nabiha Trougouty
Mohammed Bensalah
Rachid Seddik
Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature Review
Journal of Applied Hematology
acute undifferentiated leukemia
blast crisis
chronic myeloid leukemia
tyrosine kinase inhibitors
title Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature Review
title_full Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature Review
title_fullStr Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature Review
title_full_unstemmed Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature Review
title_short Transformation of Chronic Myeloid Leukemia to Acute Undifferentiated Leukemia: Case Report and Literature Review
title_sort transformation of chronic myeloid leukemia to acute undifferentiated leukemia case report and literature review
topic acute undifferentiated leukemia
blast crisis
chronic myeloid leukemia
tyrosine kinase inhibitors
url https://journals.lww.com/10.4103/joah.joah_53_25
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