Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescent

Gitelman syndrome is a renal tubular salt-wasting disorder characterized by hypokalemic metabolic alkalosis with hypomagnesemia and hypocalciuria. Patients occasionally have symptoms in childhood, while diagnosis is often in adulthood. It is inherited by an autosomal recessive manner through SLC12A...

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Main Authors: Bahriye Atmış, Rabia Miray Kışla-Ekinci, Engin Melek, Atıl Bişgin, Mustafa Yılmaz, Ali Anarat, Aysun Karabay-Bayazıt
Format: Article
Language:English
Published: Hacettepe University Institute of Child Health 2019-06-01
Series:The Turkish Journal of Pediatrics
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Online Access:https://turkjpediatr.org/article/view/717
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author Bahriye Atmış
Rabia Miray Kışla-Ekinci
Engin Melek
Atıl Bişgin
Mustafa Yılmaz
Ali Anarat
Aysun Karabay-Bayazıt
author_facet Bahriye Atmış
Rabia Miray Kışla-Ekinci
Engin Melek
Atıl Bişgin
Mustafa Yılmaz
Ali Anarat
Aysun Karabay-Bayazıt
author_sort Bahriye Atmış
collection DOAJ
description Gitelman syndrome is a renal tubular salt-wasting disorder characterized by hypokalemic metabolic alkalosis with hypomagnesemia and hypocalciuria. Patients occasionally have symptoms in childhood, while diagnosis is often in adulthood. It is inherited by an autosomal recessive manner through SLC12A3 gene mutations. Familial Mediterranean Fever (FMF) is the most common autoinflammatory disorder, inherited by an autosomal recessive manner and characterized by recurrent fever and pleuritis, peritonitis, and synovitis. Mutations in MEditerrenean FeVer (MEFV) gene, coding pyrin protein are responsible for FMF. Both MEFV and SCL12A3 genes were located on chromosome 16. A 9-year-old boy was admitted to our department because of recurrent abdominal pain, fever, joint pain and swelling since he was three years old. He was diagnosed as FMF and MEFV gene sequencing revealed homozygous M694V (c.2080A > G) mutation. At the age of 14 years, polyuria, polydipsia, hypokalemia and mild hypomagnesemia had occurred. Patient was successfully treated with oral supplementation of potassium and magnesium along with colchicine. Molecular genetic analysis including SCL12A3 gene sequencing revealed homozygote IVS4-16G > A (c.602-16G > A) intronic splicing site mutation.
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publisher Hacettepe University Institute of Child Health
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spelling doaj-art-eb3b5e013bc348af9d2a762354b3bcfe2025-08-20T02:01:51ZengHacettepe University Institute of Child HealthThe Turkish Journal of Pediatrics0041-43012791-64212019-06-0161310.24953/turkjped.2019.03.021Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescentBahriye Atmış0Rabia Miray Kışla-Ekinci1Engin Melek2Atıl Bişgin3Mustafa Yılmaz4Ali Anarat5Aysun Karabay-Bayazıt6Departments of Pediatric Nephrology, Çukurova University Faculty of Medicine, Adana, Turkey.Departments ofPediatric Rheumatology, Çukurova University Faculty of Medicine, Adana, Turkey.Departments of Pediatric Nephrology, Çukurova University Faculty of Medicine, Adana, Turkey.Departments of Medical Genetics, Çukurova University Faculty of Medicine, Adana, Turkey.Departments ofPediatric Rheumatology, Çukurova University Faculty of Medicine, Adana, Turkey.Departments of Pediatric Nephrology, Çukurova University Faculty of Medicine, Adana, Turkey.Departments of Pediatric Nephrology, Çukurova University Faculty of Medicine, Adana, Turkey. Gitelman syndrome is a renal tubular salt-wasting disorder characterized by hypokalemic metabolic alkalosis with hypomagnesemia and hypocalciuria. Patients occasionally have symptoms in childhood, while diagnosis is often in adulthood. It is inherited by an autosomal recessive manner through SLC12A3 gene mutations. Familial Mediterranean Fever (FMF) is the most common autoinflammatory disorder, inherited by an autosomal recessive manner and characterized by recurrent fever and pleuritis, peritonitis, and synovitis. Mutations in MEditerrenean FeVer (MEFV) gene, coding pyrin protein are responsible for FMF. Both MEFV and SCL12A3 genes were located on chromosome 16. A 9-year-old boy was admitted to our department because of recurrent abdominal pain, fever, joint pain and swelling since he was three years old. He was diagnosed as FMF and MEFV gene sequencing revealed homozygous M694V (c.2080A > G) mutation. At the age of 14 years, polyuria, polydipsia, hypokalemia and mild hypomagnesemia had occurred. Patient was successfully treated with oral supplementation of potassium and magnesium along with colchicine. Molecular genetic analysis including SCL12A3 gene sequencing revealed homozygote IVS4-16G > A (c.602-16G > A) intronic splicing site mutation. https://turkjpediatr.org/article/view/717Familial Mediterranean FeverGitelman syndromechild
spellingShingle Bahriye Atmış
Rabia Miray Kışla-Ekinci
Engin Melek
Atıl Bişgin
Mustafa Yılmaz
Ali Anarat
Aysun Karabay-Bayazıt
Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescent
The Turkish Journal of Pediatrics
Familial Mediterranean Fever
Gitelman syndrome
child
title Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescent
title_full Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescent
title_fullStr Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescent
title_full_unstemmed Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescent
title_short Concomitance of Familial Mediterranean Fever and Gitelman syndrome in an adolescent
title_sort concomitance of familial mediterranean fever and gitelman syndrome in an adolescent
topic Familial Mediterranean Fever
Gitelman syndrome
child
url https://turkjpediatr.org/article/view/717
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