Role of immune inflammation in the pathogenesis of osteoarthrosis and possibilities of correction of immune impairments

A great role in the pathogenesis of osteoarthrosis has been recently assigned to immune impairments, particularly to the involvement of proinflammatory cytokines (interleukin 1 (IL-1), tumor necrosis factor-α (TNF-α)) that activate catabolic processes not only in cartilage tissue, but also in the su...

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Bibliographic Details
Main Author: R M Balabanova
Format: Article
Language:Russian
Published: IMA-PRESS LLC 2011-12-01
Series:Современная ревматология
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Online Access:https://mrj.ima-press.net/mrj/article/view/390
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Summary:A great role in the pathogenesis of osteoarthrosis has been recently assigned to immune impairments, particularly to the involvement of proinflammatory cytokines (interleukin 1 (IL-1), tumor necrosis factor-α (TNF-α)) that activate catabolic processes not only in cartilage tissue, but also in the subchodral bone and other articular structures. Non-steroidal anti-inflammatory drugs, symptomatic slow-acting agents (chondroitin and glucosamine sulfate), as well as diacerein that blocks IL-1 directly and TNF-α indirectly, reduce the activity of cytokines to some extent.
ISSN:1996-7012
2310-158X