The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity

Enteropathogenic E. coli (EPEC) is a significant cause of diarrhea, leading to high infant mortality rates. A key toxin produced by EPEC is the EspC autotransporter, which is regulated alongside genes from the locus of enterocyte effacement (LEE), which collectively result in the characteristic atta...

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Main Authors: Akila U. Pilapitiya, Lilian Hor, Jing Pan, Lakshmi C. Wijeyewickrema, Robert N. Pike, Denisse L. Leyton, Jason J Paxman, Begoña Heras
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Gut Microbes
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Online Access:https://www.tandfonline.com/doi/10.1080/19490976.2025.2483777
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author Akila U. Pilapitiya
Lilian Hor
Jing Pan
Lakshmi C. Wijeyewickrema
Robert N. Pike
Denisse L. Leyton
Jason J Paxman
Begoña Heras
author_facet Akila U. Pilapitiya
Lilian Hor
Jing Pan
Lakshmi C. Wijeyewickrema
Robert N. Pike
Denisse L. Leyton
Jason J Paxman
Begoña Heras
author_sort Akila U. Pilapitiya
collection DOAJ
description Enteropathogenic E. coli (EPEC) is a significant cause of diarrhea, leading to high infant mortality rates. A key toxin produced by EPEC is the EspC autotransporter, which is regulated alongside genes from the locus of enterocyte effacement (LEE), which collectively result in the characteristic attaching and effacing lesions on the intestinal epithelium. In this study, we present the crystal structure of the EspC passenger domain (αEspC) revealing a toxin comprised a serine protease attached to a large β-helix with additional subdomains. Using various modified EspC expression constructs, alongside type III secretion system-mediated cell internalization assays, we dissect how the αEspC structural features enable toxin entry into the intestinal epithelium to cause cell cytotoxicity.
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series Gut Microbes
spelling doaj-art-eac669355b7e4482815267027ffa8b012025-08-20T02:53:44ZengTaylor & Francis GroupGut Microbes1949-09761949-09842025-12-0117110.1080/19490976.2025.2483777The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicityAkila U. Pilapitiya0Lilian Hor1Jing Pan2Lakshmi C. Wijeyewickrema3Robert N. Pike4Denisse L. Leyton5Jason J Paxman6Begoña Heras7Department of Biochemistry and Chemistry, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, AustraliaDepartment of Biochemistry and Chemistry, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, AustraliaDepartment of Biochemistry and Chemistry, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, AustraliaDepartment of Biochemistry and Chemistry, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, AustraliaDepartment of Biochemistry and Chemistry, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, AustraliaResearch School of Biology, Australian National University, Canberra, AustraliaDepartment of Biochemistry and Chemistry, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, AustraliaDepartment of Biochemistry and Chemistry, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, AustraliaEnteropathogenic E. coli (EPEC) is a significant cause of diarrhea, leading to high infant mortality rates. A key toxin produced by EPEC is the EspC autotransporter, which is regulated alongside genes from the locus of enterocyte effacement (LEE), which collectively result in the characteristic attaching and effacing lesions on the intestinal epithelium. In this study, we present the crystal structure of the EspC passenger domain (αEspC) revealing a toxin comprised a serine protease attached to a large β-helix with additional subdomains. Using various modified EspC expression constructs, alongside type III secretion system-mediated cell internalization assays, we dissect how the αEspC structural features enable toxin entry into the intestinal epithelium to cause cell cytotoxicity.https://www.tandfonline.com/doi/10.1080/19490976.2025.2483777Enteropathogenic E. coliautotransporter proteinserine proteasetoxinsbacterial infectionssecretion system
spellingShingle Akila U. Pilapitiya
Lilian Hor
Jing Pan
Lakshmi C. Wijeyewickrema
Robert N. Pike
Denisse L. Leyton
Jason J Paxman
Begoña Heras
The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity
Gut Microbes
Enteropathogenic E. coli
autotransporter protein
serine protease
toxins
bacterial infections
secretion system
title The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity
title_full The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity
title_fullStr The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity
title_full_unstemmed The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity
title_short The crystal structure of the toxin EspC from enteropathogenic Escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity
title_sort crystal structure of the toxin espc from enteropathogenic escherichia coli reveals the mechanism that governs host cell entry and cytotoxicity
topic Enteropathogenic E. coli
autotransporter protein
serine protease
toxins
bacterial infections
secretion system
url https://www.tandfonline.com/doi/10.1080/19490976.2025.2483777
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