Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.

Aspirin is a prototypic cyclooxygenase inhibitor with a variety of beneficial effects on human health. It prevents age-related diseases and delays the aging process. Previous research has shown that aspirin might act through a dietary restriction-like mechanism to extend lifespan. To explore the mec...

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Main Authors: Xiao-Bing Huang, Xiao-Hui Mu, Qin-Li Wan, Xiao-Ming He, Gui-Sheng Wu, Huai-Rong Luo
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2017-01-01
Series:PLoS ONE
Online Access:https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0184027&type=printable
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author Xiao-Bing Huang
Xiao-Hui Mu
Qin-Li Wan
Xiao-Ming He
Gui-Sheng Wu
Huai-Rong Luo
author_facet Xiao-Bing Huang
Xiao-Hui Mu
Qin-Li Wan
Xiao-Ming He
Gui-Sheng Wu
Huai-Rong Luo
author_sort Xiao-Bing Huang
collection DOAJ
description Aspirin is a prototypic cyclooxygenase inhibitor with a variety of beneficial effects on human health. It prevents age-related diseases and delays the aging process. Previous research has shown that aspirin might act through a dietary restriction-like mechanism to extend lifespan. To explore the mechanism of action of aspirin on aging, we determined the whole-genome expression profile of Caenorhabditis elegans treated with aspirin. Transcriptome analysis revealed the RNA levels of genes involved in metabolism were primarily increased. Reproduction has been reported to be associated with metabolism. We found that aspirin did not extend the lifespan or improve the heat stress resistance of germline mutants of glp-1. Furthermore, Oil Red O staining showed that aspirin treatment decreased lipid deposition and increased expression of lipid hydrolysis and fatty acid β-oxidation-related genes. The effect of germline ablation on lifespan was mainly mediated by DAF-12 and DAF-16. Next, we performed genetic analysis with a series of worm mutants and found that aspirin did not further extend the lifespans of daf-12 and daf-16 single mutants, glp-1;daf-12 and glp-1;daf-16 double mutants, or glp-1;daf-12;daf-16 triple mutants. The results suggest that aspirin increase metabolism and regulate germline signalling to activate downstream DAF-12 and DAF-16 to extend lifespan.
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spelling doaj-art-ea5d40a1ce3440d2a53446e706eeac4e2025-08-20T02:46:01ZengPublic Library of Science (PLoS)PLoS ONE1932-62032017-01-01129e018402710.1371/journal.pone.0184027Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.Xiao-Bing HuangXiao-Hui MuQin-Li WanXiao-Ming HeGui-Sheng WuHuai-Rong LuoAspirin is a prototypic cyclooxygenase inhibitor with a variety of beneficial effects on human health. It prevents age-related diseases and delays the aging process. Previous research has shown that aspirin might act through a dietary restriction-like mechanism to extend lifespan. To explore the mechanism of action of aspirin on aging, we determined the whole-genome expression profile of Caenorhabditis elegans treated with aspirin. Transcriptome analysis revealed the RNA levels of genes involved in metabolism were primarily increased. Reproduction has been reported to be associated with metabolism. We found that aspirin did not extend the lifespan or improve the heat stress resistance of germline mutants of glp-1. Furthermore, Oil Red O staining showed that aspirin treatment decreased lipid deposition and increased expression of lipid hydrolysis and fatty acid β-oxidation-related genes. The effect of germline ablation on lifespan was mainly mediated by DAF-12 and DAF-16. Next, we performed genetic analysis with a series of worm mutants and found that aspirin did not further extend the lifespans of daf-12 and daf-16 single mutants, glp-1;daf-12 and glp-1;daf-16 double mutants, or glp-1;daf-12;daf-16 triple mutants. The results suggest that aspirin increase metabolism and regulate germline signalling to activate downstream DAF-12 and DAF-16 to extend lifespan.https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0184027&type=printable
spellingShingle Xiao-Bing Huang
Xiao-Hui Mu
Qin-Li Wan
Xiao-Ming He
Gui-Sheng Wu
Huai-Rong Luo
Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.
PLoS ONE
title Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.
title_full Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.
title_fullStr Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.
title_full_unstemmed Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.
title_short Aspirin increases metabolism through germline signalling to extend the lifespan of Caenorhabditis elegans.
title_sort aspirin increases metabolism through germline signalling to extend the lifespan of caenorhabditis elegans
url https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0184027&type=printable
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AT xiaominghe aspirinincreasesmetabolismthroughgermlinesignallingtoextendthelifespanofcaenorhabditiselegans
AT guishengwu aspirinincreasesmetabolismthroughgermlinesignallingtoextendthelifespanofcaenorhabditiselegans
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