Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancer

This study is to elucidate the effect of the LINC00663/EBF1/NR2F1 axis on inflammation and angiogenesis in bladder cancer (BC) and related molecular mechanisms. After transfection, functional experiments were conducted to test cell proliferation and invasion, tube formation ability, and content of i...

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Main Authors: Xiulong Zhong, Lijiang Sun, Junxiang Liu, Xiaokun Yang, Minghui Hou, Xinning Wang, Huifeng Diao
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:RNA Biology
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Online Access:https://www.tandfonline.com/doi/10.1080/15476286.2024.2368304
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author Xiulong Zhong
Lijiang Sun
Junxiang Liu
Xiaokun Yang
Minghui Hou
Xinning Wang
Huifeng Diao
author_facet Xiulong Zhong
Lijiang Sun
Junxiang Liu
Xiaokun Yang
Minghui Hou
Xinning Wang
Huifeng Diao
author_sort Xiulong Zhong
collection DOAJ
description This study is to elucidate the effect of the LINC00663/EBF1/NR2F1 axis on inflammation and angiogenesis in bladder cancer (BC) and related molecular mechanisms. After transfection, functional experiments were conducted to test cell proliferation and invasion, tube formation ability, and content of inflammatory factors, Snail, E-cadherin, and VEGFA. Meanwhile, the relationships among LINC00663, EBF1, and NR2F1 were predicted and verified. In addition, xenograft experiments in nude mice were performed to observe the oncogenicity of 5637 BC cells in vivo. In BC tissues and cells, LINC00663 and NR2F1 were upregulated. Silencing NR2F1 or LINC00663 repressed cell proliferation and invasion, weakened vascular mimicry in vitro, decreased inflammatory factor, Snail, and VEGFA levels, and increased expression of E-cadherin. LINC00663 positively regulated NR2F1 expression through EBF1. Additionally, in vivo experiments showed that NR2F1 upregulation reversed the suppression effects of LINC00663 silencing on tumour growth, inflammation, and angiogenesis. Silencing LINC00663 decreased NR2F1 expression by mediating EBF1, thereby inhibiting BC inflammation and angiogenesis.
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issn 1547-6286
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publishDate 2024-12-01
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record_format Article
series RNA Biology
spelling doaj-art-ea161a9f31ee454aad8d52b5d91e39ea2025-08-20T01:54:57ZengTaylor & Francis GroupRNA Biology1547-62861555-85842024-12-0121167368610.1080/15476286.2024.2368304Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancerXiulong Zhong0Lijiang Sun1Junxiang Liu2Xiaokun Yang3Minghui Hou4Xinning Wang5Huifeng Diao6Department of Urology Surgery, Affiliated Hospital of Qingdao University, Qingdao, Shandong, P.R. ChinaDepartment of Urology Surgery, Affiliated Hospital of Qingdao University, Qingdao, Shandong, P.R. ChinaDepartment of Urology Surgery, Affiliated Hospital of Qingdao University, Qingdao, Shandong, P.R. ChinaDepartment of Urology Surgery, Affiliated Hospital of Qingdao University, Qingdao, Shandong, P.R. ChinaDepartment of Urology Surgery, Affiliated Hospital of Qingdao University, Qingdao, Shandong, P.R. ChinaMedical Record Management Center, Affiliated Hospital of Qingdao University, Qingdao, Shandong, P.R. ChinaDepartment of Urology Surgery, Affiliated Hospital of Qingdao University, Qingdao, Shandong, P.R. ChinaThis study is to elucidate the effect of the LINC00663/EBF1/NR2F1 axis on inflammation and angiogenesis in bladder cancer (BC) and related molecular mechanisms. After transfection, functional experiments were conducted to test cell proliferation and invasion, tube formation ability, and content of inflammatory factors, Snail, E-cadherin, and VEGFA. Meanwhile, the relationships among LINC00663, EBF1, and NR2F1 were predicted and verified. In addition, xenograft experiments in nude mice were performed to observe the oncogenicity of 5637 BC cells in vivo. In BC tissues and cells, LINC00663 and NR2F1 were upregulated. Silencing NR2F1 or LINC00663 repressed cell proliferation and invasion, weakened vascular mimicry in vitro, decreased inflammatory factor, Snail, and VEGFA levels, and increased expression of E-cadherin. LINC00663 positively regulated NR2F1 expression through EBF1. Additionally, in vivo experiments showed that NR2F1 upregulation reversed the suppression effects of LINC00663 silencing on tumour growth, inflammation, and angiogenesis. Silencing LINC00663 decreased NR2F1 expression by mediating EBF1, thereby inhibiting BC inflammation and angiogenesis.https://www.tandfonline.com/doi/10.1080/15476286.2024.2368304LINC00663EBF1nuclear receptor subfamily 2 group F member 1bladder cancerinflammationangiogenesis
spellingShingle Xiulong Zhong
Lijiang Sun
Junxiang Liu
Xiaokun Yang
Minghui Hou
Xinning Wang
Huifeng Diao
Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancer
RNA Biology
LINC00663
EBF1
nuclear receptor subfamily 2 group F member 1
bladder cancer
inflammation
angiogenesis
title Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancer
title_full Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancer
title_fullStr Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancer
title_full_unstemmed Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancer
title_short Silencing LINC00663 inhibits inflammation and angiogenesis through downregulation of NR2F1 via EBF1 in bladder cancer
title_sort silencing linc00663 inhibits inflammation and angiogenesis through downregulation of nr2f1 via ebf1 in bladder cancer
topic LINC00663
EBF1
nuclear receptor subfamily 2 group F member 1
bladder cancer
inflammation
angiogenesis
url https://www.tandfonline.com/doi/10.1080/15476286.2024.2368304
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