Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neurons

Transient Receptor Potential (TRP) ion channels like Vanilloid 1 (TRPV1) and Melastatin 3 (TRPM3) are nonselective cation channels expressed in primary sensory neurons and peripheral nerve endings, which are located in cholesterol- and sphingolipid-rich membrane lipid raft regions and have important...

Full description

Saved in:
Bibliographic Details
Main Authors: Maja Payrits, Balázs Zoltán Zsidó, Andrea Kinga Nehr-Majoros, Rita Börzsei, Zsuzsanna Helyes, Csaba Hetényi, Éva Szőke
Format: Article
Language:English
Published: Frontiers Media S.A. 2024-11-01
Series:Frontiers in Cell and Developmental Biology
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fcell.2024.1452306/full
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850220539383119872
author Maja Payrits
Maja Payrits
Maja Payrits
Balázs Zoltán Zsidó
Balázs Zoltán Zsidó
Andrea Kinga Nehr-Majoros
Andrea Kinga Nehr-Majoros
Andrea Kinga Nehr-Majoros
Rita Börzsei
Rita Börzsei
Zsuzsanna Helyes
Zsuzsanna Helyes
Zsuzsanna Helyes
Csaba Hetényi
Csaba Hetényi
Éva Szőke
Éva Szőke
Éva Szőke
author_facet Maja Payrits
Maja Payrits
Maja Payrits
Balázs Zoltán Zsidó
Balázs Zoltán Zsidó
Andrea Kinga Nehr-Majoros
Andrea Kinga Nehr-Majoros
Andrea Kinga Nehr-Majoros
Rita Börzsei
Rita Börzsei
Zsuzsanna Helyes
Zsuzsanna Helyes
Zsuzsanna Helyes
Csaba Hetényi
Csaba Hetényi
Éva Szőke
Éva Szőke
Éva Szőke
author_sort Maja Payrits
collection DOAJ
description Transient Receptor Potential (TRP) ion channels like Vanilloid 1 (TRPV1) and Melastatin 3 (TRPM3) are nonselective cation channels expressed in primary sensory neurons and peripheral nerve endings, which are located in cholesterol- and sphingolipid-rich membrane lipid raft regions and have important roles in pain processing. Besides TRP ion channels a wide variety of voltage-gated ion channels were also described in the membrane raft regions of neuronal cells. Here we investigated the effects of lipid raft disruption by methyl-beta-cyclodextrin (MCD) and sphingomyelinase (SMase) on TRPV1, TRPM3 and voltage-gated L-type Ca2+ channel activation in cultured trigeminal neurons and sensory nerve terminals of the trachea. We also examined the mechanism of action of MCD by in silico modeling. Disruption of lipid rafts by MCD or SMase did not alter CIM0216-induced TRPM3 cation channel activation and the voltage-gated L-type Ca2+ channel activation by FPL 64126 or veratridine neither on trigeminal sensory neurons nor sensory nerve terminals. We provided the first structural explanation with in silico modeling that the activation of TRPV1, TRPM3 and voltage-gated L-type Ca2+ channels is affected differently by the cholesterol content surrounding them in the plasma membrane. It is concluded that modifying the hydrophobic interactions between lipid rafts and ion channels might provide a selective novel mechanism for peripheral analgesia.
format Article
id doaj-art-e9f2aa3183e84344a3e132c11e562e75
institution OA Journals
issn 2296-634X
language English
publishDate 2024-11-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Cell and Developmental Biology
spelling doaj-art-e9f2aa3183e84344a3e132c11e562e752025-08-20T02:07:02ZengFrontiers Media S.A.Frontiers in Cell and Developmental Biology2296-634X2024-11-011210.3389/fcell.2024.14523061452306Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neuronsMaja Payrits0Maja Payrits1Maja Payrits2Balázs Zoltán Zsidó3Balázs Zoltán Zsidó4Andrea Kinga Nehr-Majoros5Andrea Kinga Nehr-Majoros6Andrea Kinga Nehr-Majoros7Rita Börzsei8Rita Börzsei9Zsuzsanna Helyes10Zsuzsanna Helyes11Zsuzsanna Helyes12Csaba Hetényi13Csaba Hetényi14Éva Szőke15Éva Szőke16Éva Szőke17Department of Pharmacology and Pharmacotherapy, Medical School and Centre for Neuroscience, University of Pécs, Pécs, HungaryNational Laboratory for Drug Research and Development, Budapest, HungaryHungarian Research Network, Chronic Pain Research Group, Pécs, HungaryNational Laboratory for Drug Research and Development, Budapest, HungaryPharmacoinformatics Unit, Department of Pharmacology and Pharmacotherapy, Medical School, University of Pécs, Pécs, HungaryDepartment of Pharmacology and Pharmacotherapy, Medical School and Centre for Neuroscience, University of Pécs, Pécs, HungaryNational Laboratory for Drug Research and Development, Budapest, HungaryHungarian Research Network, Chronic Pain Research Group, Pécs, HungaryNational Laboratory for Drug Research and Development, Budapest, HungaryPharmacoinformatics Unit, Department of Pharmacology and Pharmacotherapy, Medical School, University of Pécs, Pécs, HungaryDepartment of Pharmacology and Pharmacotherapy, Medical School and Centre for Neuroscience, University of Pécs, Pécs, HungaryNational Laboratory for Drug Research and Development, Budapest, HungaryHungarian Research Network, Chronic Pain Research Group, Pécs, HungaryNational Laboratory for Drug Research and Development, Budapest, HungaryPharmacoinformatics Unit, Department of Pharmacology and Pharmacotherapy, Medical School, University of Pécs, Pécs, HungaryDepartment of Pharmacology and Pharmacotherapy, Medical School and Centre for Neuroscience, University of Pécs, Pécs, HungaryNational Laboratory for Drug Research and Development, Budapest, HungaryHungarian Research Network, Chronic Pain Research Group, Pécs, HungaryTransient Receptor Potential (TRP) ion channels like Vanilloid 1 (TRPV1) and Melastatin 3 (TRPM3) are nonselective cation channels expressed in primary sensory neurons and peripheral nerve endings, which are located in cholesterol- and sphingolipid-rich membrane lipid raft regions and have important roles in pain processing. Besides TRP ion channels a wide variety of voltage-gated ion channels were also described in the membrane raft regions of neuronal cells. Here we investigated the effects of lipid raft disruption by methyl-beta-cyclodextrin (MCD) and sphingomyelinase (SMase) on TRPV1, TRPM3 and voltage-gated L-type Ca2+ channel activation in cultured trigeminal neurons and sensory nerve terminals of the trachea. We also examined the mechanism of action of MCD by in silico modeling. Disruption of lipid rafts by MCD or SMase did not alter CIM0216-induced TRPM3 cation channel activation and the voltage-gated L-type Ca2+ channel activation by FPL 64126 or veratridine neither on trigeminal sensory neurons nor sensory nerve terminals. We provided the first structural explanation with in silico modeling that the activation of TRPV1, TRPM3 and voltage-gated L-type Ca2+ channels is affected differently by the cholesterol content surrounding them in the plasma membrane. It is concluded that modifying the hydrophobic interactions between lipid rafts and ion channels might provide a selective novel mechanism for peripheral analgesia.https://www.frontiersin.org/articles/10.3389/fcell.2024.1452306/fullTRPV1TRPM3L-type voltage-gated Ca2+ channelCIM0216FPL 64176veratridine
spellingShingle Maja Payrits
Maja Payrits
Maja Payrits
Balázs Zoltán Zsidó
Balázs Zoltán Zsidó
Andrea Kinga Nehr-Majoros
Andrea Kinga Nehr-Majoros
Andrea Kinga Nehr-Majoros
Rita Börzsei
Rita Börzsei
Zsuzsanna Helyes
Zsuzsanna Helyes
Zsuzsanna Helyes
Csaba Hetényi
Csaba Hetényi
Éva Szőke
Éva Szőke
Éva Szőke
Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neurons
Frontiers in Cell and Developmental Biology
TRPV1
TRPM3
L-type voltage-gated Ca2+ channel
CIM0216
FPL 64176
veratridine
title Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neurons
title_full Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neurons
title_fullStr Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neurons
title_full_unstemmed Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neurons
title_short Lipid raft disruption inhibits the activation of Transient Receptor Potential Vanilloid 1, but not TRP Melastatin 3 and the voltage-gated L-type calcium channels in sensory neurons
title_sort lipid raft disruption inhibits the activation of transient receptor potential vanilloid 1 but not trp melastatin 3 and the voltage gated l type calcium channels in sensory neurons
topic TRPV1
TRPM3
L-type voltage-gated Ca2+ channel
CIM0216
FPL 64176
veratridine
url https://www.frontiersin.org/articles/10.3389/fcell.2024.1452306/full
work_keys_str_mv AT majapayrits lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT majapayrits lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT majapayrits lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT balazszoltanzsido lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT balazszoltanzsido lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT andreakinganehrmajoros lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT andreakinganehrmajoros lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT andreakinganehrmajoros lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT ritaborzsei lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT ritaborzsei lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT zsuzsannahelyes lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT zsuzsannahelyes lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT zsuzsannahelyes lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT csabahetenyi lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT csabahetenyi lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT evaszoke lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT evaszoke lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons
AT evaszoke lipidraftdisruptioninhibitstheactivationoftransientreceptorpotentialvanilloid1butnottrpmelastatin3andthevoltagegatedltypecalciumchannelsinsensoryneurons