Genetic and biochemical characterizations of Zika virus NS2A protein

Zika virus (ZIKV) can cause devastating congenital Zika syndromes in pregnant women and Guillain-Barre syndrome in adults. Understanding the molecular mechanism of ZIKV replication is essential for antiviral and vaccine development. Here we report the structural and functional characterization of ZI...

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Main Authors: Xianwen Zhang, Xuping Xie, Jing Zou, Hongjie Xia, Chao Shan, Xinwen Chen, Pei-Yong Shi
Format: Article
Language:English
Published: Taylor & Francis Group 2019-01-01
Series:Emerging Microbes and Infections
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Online Access:https://www.tandfonline.com/doi/10.1080/22221751.2019.1598291
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author Xianwen Zhang
Xuping Xie
Jing Zou
Hongjie Xia
Chao Shan
Xinwen Chen
Pei-Yong Shi
author_facet Xianwen Zhang
Xuping Xie
Jing Zou
Hongjie Xia
Chao Shan
Xinwen Chen
Pei-Yong Shi
author_sort Xianwen Zhang
collection DOAJ
description Zika virus (ZIKV) can cause devastating congenital Zika syndromes in pregnant women and Guillain-Barre syndrome in adults. Understanding the molecular mechanism of ZIKV replication is essential for antiviral and vaccine development. Here we report the structural and functional characterization of ZIKV NS2A protein. Biochemical structural probing suggests that ZIKV NS2A has a single segment that traverses the ER membrane and six segments that peripherally associate with the ER membrane. Functional analysis has defined distinct NS2A residues essential for viral RNA synthesis or virion assembly. Only the virion assembly-defective mutants, but not the RNA synthesis-defective mutants, could be rescued through trans complementation with a wide-type NS2A protein. These results suggest that the NS2A molecules in virion assembly complex could be recruited in trans, whereas the NS2A molecules in viral replication complex must be recruited in cis. Together with previous results, we propose a flavivirus assembly model where NS2A plays a central role in modulating viral structural and nonstructural proteins as well as genomic RNA during virion assembly.
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spelling doaj-art-e8e69cdc6b144a2b8620755dbd7f4a382025-08-20T02:07:59ZengTaylor & Francis GroupEmerging Microbes and Infections2222-17512019-01-018158560210.1080/22221751.2019.1598291Genetic and biochemical characterizations of Zika virus NS2A proteinXianwen Zhang0Xuping Xie1Jing Zou2Hongjie Xia3Chao Shan4Xinwen Chen5Pei-Yong Shi6State Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, People’s Republic of ChinaDepartment of Biochemistry & Molecular Biology, University of Texas Medical Branch, Galveston, TX, USADepartment of Biochemistry & Molecular Biology, University of Texas Medical Branch, Galveston, TX, USADepartment of Biochemistry & Molecular Biology, University of Texas Medical Branch, Galveston, TX, USADepartment of Biochemistry & Molecular Biology, University of Texas Medical Branch, Galveston, TX, USAState Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, People’s Republic of ChinaDepartment of Biochemistry & Molecular Biology, University of Texas Medical Branch, Galveston, TX, USAZika virus (ZIKV) can cause devastating congenital Zika syndromes in pregnant women and Guillain-Barre syndrome in adults. Understanding the molecular mechanism of ZIKV replication is essential for antiviral and vaccine development. Here we report the structural and functional characterization of ZIKV NS2A protein. Biochemical structural probing suggests that ZIKV NS2A has a single segment that traverses the ER membrane and six segments that peripherally associate with the ER membrane. Functional analysis has defined distinct NS2A residues essential for viral RNA synthesis or virion assembly. Only the virion assembly-defective mutants, but not the RNA synthesis-defective mutants, could be rescued through trans complementation with a wide-type NS2A protein. These results suggest that the NS2A molecules in virion assembly complex could be recruited in trans, whereas the NS2A molecules in viral replication complex must be recruited in cis. Together with previous results, we propose a flavivirus assembly model where NS2A plays a central role in modulating viral structural and nonstructural proteins as well as genomic RNA during virion assembly.https://www.tandfonline.com/doi/10.1080/22221751.2019.1598291Zika virusflavivirus replicationvirion assemblyflavivirus NS2Amembrane topology
spellingShingle Xianwen Zhang
Xuping Xie
Jing Zou
Hongjie Xia
Chao Shan
Xinwen Chen
Pei-Yong Shi
Genetic and biochemical characterizations of Zika virus NS2A protein
Emerging Microbes and Infections
Zika virus
flavivirus replication
virion assembly
flavivirus NS2A
membrane topology
title Genetic and biochemical characterizations of Zika virus NS2A protein
title_full Genetic and biochemical characterizations of Zika virus NS2A protein
title_fullStr Genetic and biochemical characterizations of Zika virus NS2A protein
title_full_unstemmed Genetic and biochemical characterizations of Zika virus NS2A protein
title_short Genetic and biochemical characterizations of Zika virus NS2A protein
title_sort genetic and biochemical characterizations of zika virus ns2a protein
topic Zika virus
flavivirus replication
virion assembly
flavivirus NS2A
membrane topology
url https://www.tandfonline.com/doi/10.1080/22221751.2019.1598291
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