The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacy

Abstract AT‐IAP (1‐{6‐[(4‐fluorophenyl)methyl]‐3,3‐dimethyl‐1H,2H,3H‐pyrrolo[3,2‐b]pyridin‐1‐yl}‐2‐[(2R,5R)‐5‐methyl‐2‐{[(3R)‐3‐methylmorpholin‐4‐yl]methyl}piperazin‐1‐yl]ethan‐1‐one) was identified as a novel potent non‐alanine small molecule dual inhibitor of cIAP1/XIAP protein. AT‐IAP was assesse...

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Main Authors: Vanessa Martins, George Ward, Tomoko Smyth, Mike Reader, Gianni Chessari, Chris Johnson, Nicola Wilsher
Format: Article
Language:English
Published: Wiley 2024-12-01
Series:Pharmacology Research & Perspectives
Online Access:https://doi.org/10.1002/prp2.70030
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author Vanessa Martins
George Ward
Tomoko Smyth
Mike Reader
Gianni Chessari
Chris Johnson
Nicola Wilsher
author_facet Vanessa Martins
George Ward
Tomoko Smyth
Mike Reader
Gianni Chessari
Chris Johnson
Nicola Wilsher
author_sort Vanessa Martins
collection DOAJ
description Abstract AT‐IAP (1‐{6‐[(4‐fluorophenyl)methyl]‐3,3‐dimethyl‐1H,2H,3H‐pyrrolo[3,2‐b]pyridin‐1‐yl}‐2‐[(2R,5R)‐5‐methyl‐2‐{[(3R)‐3‐methylmorpholin‐4‐yl]methyl}piperazin‐1‐yl]ethan‐1‐one) was identified as a novel potent non‐alanine small molecule dual inhibitor of cIAP1/XIAP protein. AT‐IAP was assessed in preclinical species, demonstrating favorable bioavailability in rodent species and oral efficacy at 30 mg/kg in MDA‐MB‐231 mouse xenograft models. The major metabolic route of AT‐IAP was identified to be CYP3A driven, resulting in low oral exposure in non‐human primate (NHP) studies, given the comparatively high expression of equivalent CYP3A. AT‐IAP metabolite identification studies determined ring opening of the morpholino or piperazine moiety. An extensive campaign of optimisation resulted in increased polarity by the addition of a hydroxymethyl, which led to the identification of tolinapant (1‐(6‐[(4‐Fluorophenyl)methyl]‐5‐(hydroxymethyl)‐3,3‐dimethyl‐1 H,2 H,3 H‐pyrrolo[3,2‐ b]pyridin‐1‐yl)‐2‐[(2 R,5 R)‐5‐methyl‐2‐([(3R)‐3‐methylmorpholin‐4‐yl]methyl)piperazin‐1‐yl]ethan‐1‐one) with reduced CYP3A metabolism. Tolinapant showed oral bioavailability in rodents and NHP in the range 12–34% at 5 mg/kg. Non‐linear pharmacokinetics in NHP were observed at oral doses in the range 5–75 mg/kg. Pharmacodynamic modulation and efficacy were demonstrated in A375 mouse xenograft models at dose ranges between 5 and 20 mg/kg. On‐target engagement, as measured by reduction of cIAP 1 protein levels, was confirmed in NHP surrogate tissues and applied to target activity assessments in tolinapant phase1/2 clinical trials.
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spelling doaj-art-e8c8c3ab0275432482ed48a188db291c2025-08-20T02:38:03ZengWileyPharmacology Research & Perspectives2052-17072024-12-01126n/an/a10.1002/prp2.70030The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacyVanessa Martins0George Ward1Tomoko Smyth2Mike Reader3Gianni Chessari4Chris Johnson5Nicola Wilsher6Astex Therapeutics Limited Cambridge UKAstex Therapeutics Limited Cambridge UKAstex Therapeutics Limited Cambridge UKAstex Therapeutics Limited Cambridge UKAstex Therapeutics Limited Cambridge UKAstex Therapeutics Limited Cambridge UKAstex Therapeutics Limited Cambridge UKAbstract AT‐IAP (1‐{6‐[(4‐fluorophenyl)methyl]‐3,3‐dimethyl‐1H,2H,3H‐pyrrolo[3,2‐b]pyridin‐1‐yl}‐2‐[(2R,5R)‐5‐methyl‐2‐{[(3R)‐3‐methylmorpholin‐4‐yl]methyl}piperazin‐1‐yl]ethan‐1‐one) was identified as a novel potent non‐alanine small molecule dual inhibitor of cIAP1/XIAP protein. AT‐IAP was assessed in preclinical species, demonstrating favorable bioavailability in rodent species and oral efficacy at 30 mg/kg in MDA‐MB‐231 mouse xenograft models. The major metabolic route of AT‐IAP was identified to be CYP3A driven, resulting in low oral exposure in non‐human primate (NHP) studies, given the comparatively high expression of equivalent CYP3A. AT‐IAP metabolite identification studies determined ring opening of the morpholino or piperazine moiety. An extensive campaign of optimisation resulted in increased polarity by the addition of a hydroxymethyl, which led to the identification of tolinapant (1‐(6‐[(4‐Fluorophenyl)methyl]‐5‐(hydroxymethyl)‐3,3‐dimethyl‐1 H,2 H,3 H‐pyrrolo[3,2‐ b]pyridin‐1‐yl)‐2‐[(2 R,5 R)‐5‐methyl‐2‐([(3R)‐3‐methylmorpholin‐4‐yl]methyl)piperazin‐1‐yl]ethan‐1‐one) with reduced CYP3A metabolism. Tolinapant showed oral bioavailability in rodents and NHP in the range 12–34% at 5 mg/kg. Non‐linear pharmacokinetics in NHP were observed at oral doses in the range 5–75 mg/kg. Pharmacodynamic modulation and efficacy were demonstrated in A375 mouse xenograft models at dose ranges between 5 and 20 mg/kg. On‐target engagement, as measured by reduction of cIAP 1 protein levels, was confirmed in NHP surrogate tissues and applied to target activity assessments in tolinapant phase1/2 clinical trials.https://doi.org/10.1002/prp2.70030
spellingShingle Vanessa Martins
George Ward
Tomoko Smyth
Mike Reader
Gianni Chessari
Chris Johnson
Nicola Wilsher
The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacy
Pharmacology Research & Perspectives
title The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacy
title_full The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacy
title_fullStr The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacy
title_full_unstemmed The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacy
title_short The preclinical pharmacokinetics of Tolinapant—A dual cIAP1/XIAP antagonist with in vivo efficacy
title_sort preclinical pharmacokinetics of tolinapant a dual ciap1 xiap antagonist with in vivo efficacy
url https://doi.org/10.1002/prp2.70030
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