Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7

Conclusion: EXE can modulate the miR-30d-5p/GALNT7 axis to inhibit the expression of TSHR, thereby regulating the polarization of macrophages to the M2 phenotype and ultimately alleviating DOX-induced myocardial injury, which provides new targets and strategies for the clinical treatment of myocardi...

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Main Authors: Haiyan Wu, Ruoyu Zhou, Hanxin Kong, Jieqiong Yang, Suijuan Liu, Xiaolin Wei, Kunzhi Li, Yunmei Zhang
Format: Article
Language:English
Published: Wiley 2024-01-01
Series:Journal of Immunology Research
Online Access:http://dx.doi.org/10.1155/2024/5562293
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author Haiyan Wu
Ruoyu Zhou
Hanxin Kong
Jieqiong Yang
Suijuan Liu
Xiaolin Wei
Kunzhi Li
Yunmei Zhang
author_facet Haiyan Wu
Ruoyu Zhou
Hanxin Kong
Jieqiong Yang
Suijuan Liu
Xiaolin Wei
Kunzhi Li
Yunmei Zhang
author_sort Haiyan Wu
collection DOAJ
description Conclusion: EXE can modulate the miR-30d-5p/GALNT7 axis to inhibit the expression of TSHR, thereby regulating the polarization of macrophages to the M2 phenotype and ultimately alleviating DOX-induced myocardial injury, which provides new targets and strategies for the clinical treatment of myocardial injury.
format Article
id doaj-art-e7296b1167534264b164cc0d41eebb87
institution Kabale University
issn 2314-7156
language English
publishDate 2024-01-01
publisher Wiley
record_format Article
series Journal of Immunology Research
spelling doaj-art-e7296b1167534264b164cc0d41eebb872025-08-20T03:26:11ZengWileyJournal of Immunology Research2314-71562024-01-01202410.1155/2024/5562293Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7Haiyan Wu0Ruoyu Zhou1Hanxin Kong2Jieqiong Yang3Suijuan Liu4Xiaolin Wei5Kunzhi Li6Yunmei Zhang7Faculty of Life Science and TechnologyMedical SchoolDepartment of CardiologyDepartment of CardiologyDepartment of CardiologyDepartment of CardiologyFaculty of Life Science and TechnologyDepartment of CardiologyConclusion: EXE can modulate the miR-30d-5p/GALNT7 axis to inhibit the expression of TSHR, thereby regulating the polarization of macrophages to the M2 phenotype and ultimately alleviating DOX-induced myocardial injury, which provides new targets and strategies for the clinical treatment of myocardial injury.http://dx.doi.org/10.1155/2024/5562293
spellingShingle Haiyan Wu
Ruoyu Zhou
Hanxin Kong
Jieqiong Yang
Suijuan Liu
Xiaolin Wei
Kunzhi Li
Yunmei Zhang
Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7
Journal of Immunology Research
title Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7
title_full Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7
title_fullStr Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7
title_full_unstemmed Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7
title_short Exercise Attenuates Doxorubicin-Induced Myocardial Injury by Inhibiting TSHR and Regulating Macrophage Polarization Through miR-30d-5p/GALNT7
title_sort exercise attenuates doxorubicin induced myocardial injury by inhibiting tshr and regulating macrophage polarization through mir 30d 5p galnt7
url http://dx.doi.org/10.1155/2024/5562293
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