The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid Hemorrhage

Vascular endothelial growth factor (VEGF) stimulating angiogenesis was shown to be a potential novel therapeutic approach for the treatment of ischemic vascular diseases. The goal of the present study was to examine whether transfection of VEGF before occurrence of major stroke (part I) and cerebra...

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Main Authors: Sven Oliver Eicker, Moritz Hoppe, Nima Etminan, Stephan Macht, Jason Perrin, Hans-Jakob Steiger, Daniel Hänggi
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:Stroke Research and Treatment
Online Access:http://dx.doi.org/10.1155/2013/948783
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author Sven Oliver Eicker
Moritz Hoppe
Nima Etminan
Stephan Macht
Jason Perrin
Hans-Jakob Steiger
Daniel Hänggi
author_facet Sven Oliver Eicker
Moritz Hoppe
Nima Etminan
Stephan Macht
Jason Perrin
Hans-Jakob Steiger
Daniel Hänggi
author_sort Sven Oliver Eicker
collection DOAJ
description Vascular endothelial growth factor (VEGF) stimulating angiogenesis was shown to be a potential novel therapeutic approach for the treatment of ischemic vascular diseases. The goal of the present study was to examine whether transfection of VEGF before occurrence of major stroke (part I) and cerebral vasospasm after experimental subarachnoid hemorrhage (SAH; part II) develops neuroprotective qualities. A total of 25 (part I) and 26 (part II) brains were analyzed, respectively. In part one, a significant reduction of infarct volume in the VEGF-treated stroke animals (43% reduction, P<0.05) could be detected. In part two, significant vasospasm was induced in all hemorrhage groups (P<0.02). Analyzing microperfusion, a significant higher amount of perfused vessels could be detected (P<0.01), whereas no significant effect could be detected towards macroperfusion. Histologically, no infarctions were observed in the VEGF-treated SAH group and the sham-operated group. Minor infarction in terms of vasospasm-induced small lesions could be detected in the control vector transduced group (P=0.05) and saline-treated group (P=0.09). The present study demonstrates the preconditioning impact of systemic intramuscular VEGF injection in animals after major stroke and induced severe vasospasm after SAH.
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spelling doaj-art-e6cebd419dbd43dd9ef8372c357c104d2025-08-20T02:21:04ZengWileyStroke Research and Treatment2090-81052042-00562013-01-01201310.1155/2013/948783948783The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid HemorrhageSven Oliver Eicker0Moritz Hoppe1Nima Etminan2Stephan Macht3Jason Perrin4Hans-Jakob Steiger5Daniel Hänggi6Department of Neurosurgery, Heinrich-Heine-University, Medical Faculty, 40225 Düsseldorf, GermanyDepartment of Neurosurgery, Heinrich-Heine-University, Medical Faculty, 40225 Düsseldorf, GermanyDepartment of Neurosurgery, Heinrich-Heine-University, Medical Faculty, 40225 Düsseldorf, GermanyDepartment of Radiology, Heinrich-Heine-University, Medical Faculty, 40225 Düsseldorf, GermanyDepartment of Neurosurgery, Heinrich-Heine-University, Medical Faculty, 40225 Düsseldorf, GermanyDepartment of Neurosurgery, Heinrich-Heine-University, Medical Faculty, 40225 Düsseldorf, GermanyDepartment of Neurosurgery, Heinrich-Heine-University, Medical Faculty, 40225 Düsseldorf, GermanyVascular endothelial growth factor (VEGF) stimulating angiogenesis was shown to be a potential novel therapeutic approach for the treatment of ischemic vascular diseases. The goal of the present study was to examine whether transfection of VEGF before occurrence of major stroke (part I) and cerebral vasospasm after experimental subarachnoid hemorrhage (SAH; part II) develops neuroprotective qualities. A total of 25 (part I) and 26 (part II) brains were analyzed, respectively. In part one, a significant reduction of infarct volume in the VEGF-treated stroke animals (43% reduction, P<0.05) could be detected. In part two, significant vasospasm was induced in all hemorrhage groups (P<0.02). Analyzing microperfusion, a significant higher amount of perfused vessels could be detected (P<0.01), whereas no significant effect could be detected towards macroperfusion. Histologically, no infarctions were observed in the VEGF-treated SAH group and the sham-operated group. Minor infarction in terms of vasospasm-induced small lesions could be detected in the control vector transduced group (P=0.05) and saline-treated group (P=0.09). The present study demonstrates the preconditioning impact of systemic intramuscular VEGF injection in animals after major stroke and induced severe vasospasm after SAH.http://dx.doi.org/10.1155/2013/948783
spellingShingle Sven Oliver Eicker
Moritz Hoppe
Nima Etminan
Stephan Macht
Jason Perrin
Hans-Jakob Steiger
Daniel Hänggi
The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid Hemorrhage
Stroke Research and Treatment
title The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid Hemorrhage
title_full The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid Hemorrhage
title_fullStr The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid Hemorrhage
title_full_unstemmed The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid Hemorrhage
title_short The Impact of Experimental Preconditioning Using Vascular Endothelial Growth Factor in Stroke and Subarachnoid Hemorrhage
title_sort impact of experimental preconditioning using vascular endothelial growth factor in stroke and subarachnoid hemorrhage
url http://dx.doi.org/10.1155/2013/948783
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