LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary Explants

Acting at the level of the brain, interleukin- (IL-)1β is considered to be one of the most potent downregulators of reproduction processes during immune/inflammatory challenge. IL-1β suppresses gonadotropin-releasing hormone (GnRH) secretion from the hypothalamus resulting in the inhibition of the l...

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Main Authors: Andrzej Przemysław Herman, Agata Krawczyńska, Joanna Bochenek, Elżbieta Dobek, Anna Herman, Dorota Tomaszewska-Zaremba
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:Clinical and Developmental Immunology
Online Access:http://dx.doi.org/10.1155/2013/926937
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author Andrzej Przemysław Herman
Agata Krawczyńska
Joanna Bochenek
Elżbieta Dobek
Anna Herman
Dorota Tomaszewska-Zaremba
author_facet Andrzej Przemysław Herman
Agata Krawczyńska
Joanna Bochenek
Elżbieta Dobek
Anna Herman
Dorota Tomaszewska-Zaremba
author_sort Andrzej Przemysław Herman
collection DOAJ
description Acting at the level of the brain, interleukin- (IL-)1β is considered to be one of the most potent downregulators of reproduction processes during immune/inflammatory challenge. IL-1β suppresses gonadotropin-releasing hormone (GnRH) secretion from the hypothalamus resulting in the inhibition of the luteinizing hormone (LH) release from the anterior pituitary (AP). However, the presence of IL-1β receptors in the AP suggests the possible direct action of this cytokine on LH secretion. The study was designed to determine the effect of IL-1β on the LH secretion from the AP explants collected from saline and LPS-treated ewes in the follicular phase. It was found that IL-1β suppressed () GnRH-stimulated LH release and LHβ gene expression in AP explants in both groups. However, IL-1β action was more potent in the explants collected from LPS-treated animals. Pituitaries from LPS-treated animals were characterized by increased () IL-1 type I receptor and decreased () GnRH receptor gene expression level compared to the saline-treated group. IL-1β also affected the GnRH-R gene expression in explants collected from LPS-treated animals. Our results show that direct action of IL-1β on the pituitary gonadotropes could be one of the reasons of the reproductive processes disorders accompanying an inflammatory state.
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spelling doaj-art-e6ae73f44c264827b077ad33138b06a62025-08-20T03:26:03ZengWileyClinical and Developmental Immunology1740-25221740-25302013-01-01201310.1155/2013/926937926937LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary ExplantsAndrzej Przemysław Herman0Agata Krawczyńska1Joanna Bochenek2Elżbieta Dobek3Anna Herman4Dorota Tomaszewska-Zaremba5Polish Academy of Sciences, The Kielanowski Institute of Animal Physiology and Nutrition, 05-110 Jabłonna, PolandPolish Academy of Sciences, The Kielanowski Institute of Animal Physiology and Nutrition, 05-110 Jabłonna, PolandPolish Academy of Sciences, The Kielanowski Institute of Animal Physiology and Nutrition, 05-110 Jabłonna, PolandPolish Academy of Sciences, The Kielanowski Institute of Animal Physiology and Nutrition, 05-110 Jabłonna, PolandThe Academy of Cosmetics and Health Care, 13 Podwale Street, 00-252 Warsaw, PolandPolish Academy of Sciences, The Kielanowski Institute of Animal Physiology and Nutrition, 05-110 Jabłonna, PolandActing at the level of the brain, interleukin- (IL-)1β is considered to be one of the most potent downregulators of reproduction processes during immune/inflammatory challenge. IL-1β suppresses gonadotropin-releasing hormone (GnRH) secretion from the hypothalamus resulting in the inhibition of the luteinizing hormone (LH) release from the anterior pituitary (AP). However, the presence of IL-1β receptors in the AP suggests the possible direct action of this cytokine on LH secretion. The study was designed to determine the effect of IL-1β on the LH secretion from the AP explants collected from saline and LPS-treated ewes in the follicular phase. It was found that IL-1β suppressed () GnRH-stimulated LH release and LHβ gene expression in AP explants in both groups. However, IL-1β action was more potent in the explants collected from LPS-treated animals. Pituitaries from LPS-treated animals were characterized by increased () IL-1 type I receptor and decreased () GnRH receptor gene expression level compared to the saline-treated group. IL-1β also affected the GnRH-R gene expression in explants collected from LPS-treated animals. Our results show that direct action of IL-1β on the pituitary gonadotropes could be one of the reasons of the reproductive processes disorders accompanying an inflammatory state.http://dx.doi.org/10.1155/2013/926937
spellingShingle Andrzej Przemysław Herman
Agata Krawczyńska
Joanna Bochenek
Elżbieta Dobek
Anna Herman
Dorota Tomaszewska-Zaremba
LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary Explants
Clinical and Developmental Immunology
title LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary Explants
title_full LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary Explants
title_fullStr LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary Explants
title_full_unstemmed LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary Explants
title_short LPS-Induced Inflammation Potentiates the IL-1-Mediated Reduction of LH Secretion from the Anterior Pituitary Explants
title_sort lps induced inflammation potentiates the il 1 mediated reduction of lh secretion from the anterior pituitary explants
url http://dx.doi.org/10.1155/2013/926937
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