LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106b

LncRNA PTENP1 is a competitive endogenous RNA (ceRNA) involved in decoying miR-106b in multiple diseases. This study investigates the interaction of PTENP1 and miR-106b in cell proliferation, apoptosis and epithelial–mesenchymal transition (EMT) in cervical cancer. The expressions of PTENP1, miR-106...

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Main Authors: Yingrui Fan, Weiwei Sheng, Yi Meng, Yundi Cao, Rong Li
Format: Article
Language:English
Published: Taylor & Francis Group 2020-01-01
Series:Artificial Cells, Nanomedicine, and Biotechnology
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Online Access:https://www.tandfonline.com/doi/10.1080/21691401.2019.1709852
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author Yingrui Fan
Weiwei Sheng
Yi Meng
Yundi Cao
Rong Li
author_facet Yingrui Fan
Weiwei Sheng
Yi Meng
Yundi Cao
Rong Li
author_sort Yingrui Fan
collection DOAJ
description LncRNA PTENP1 is a competitive endogenous RNA (ceRNA) involved in decoying miR-106b in multiple diseases. This study investigates the interaction of PTENP1 and miR-106b in cell proliferation, apoptosis and epithelial–mesenchymal transition (EMT) in cervical cancer. The expressions of PTENP1, miR-106b and PTEN were determined in cervical cancer tissues, adjacent normal tissues, cervical cancer cells (HeLa, SiHa, C33A and CasKi) and normal cervical epithelial H8 cells. Up-regulation of PTENP1 and down-regulation of miR-106b were conducted in HeLa and CasKi cells by transfecting cells with corresponding miRNA mimics and inhibitors. Bioinformatics analysis, luciferase reporter assay and RNA-pull down assay were performed to verify the association of miR-106b, PTEN, and PTENP1. Cell growth and cell apoptosis were determined by CCK-8 and flow cytometry analysis. It was found that the expressions of PTENP1 and PTEN were up-regulated and that of miR-106b were down-regulated in cervical cancer tissues and cells. PTENP1 localized in cytoplasm and competitively bound to miR-106b. Up-regulation of PTENP1 and down-regulation of miR-106b contributed to increased expressions of PTEN and E-cadherin. Decreased expression of miR-106b, ZEB1, Snail and Vimentin, resulted in inhibiting cell proliferation and promoting cell apoptosis. Over-expression of PTENP1 and miR-106b accelerated cell proliferation and slowed down cell apoptosis. miR-106b inhibited the expression of PTEN. Our results suggest that LncRNA PTENP1 inhibits cervical cancer progression by competitively binding to miR-106b, leading to promote PTEN expression, inhibit cell proliferation and EMT and induce cell apoptosis in cervical cancer cells
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spelling doaj-art-e56145d719f64a1d83b4a62057f1678e2025-08-20T03:41:21ZengTaylor & Francis GroupArtificial Cells, Nanomedicine, and Biotechnology2169-14012169-141X2020-01-0148139340710.1080/21691401.2019.1709852LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106bYingrui Fan0Weiwei Sheng1Yi Meng2Yundi Cao3Rong Li4Department of Oncology, Taikang Xianlin Drum Tower Hospital, Nanjing University School of Medicine, Nanjing, Jiangsu, P.R. ChinaDepartment of Oncology, Taikang Xianlin Drum Tower Hospital, Nanjing University School of Medicine, Nanjing, Jiangsu, P.R. ChinaDepartment of Oncology, Taikang Xianlin Drum Tower Hospital, Nanjing University School of Medicine, Nanjing, Jiangsu, P.R. ChinaDepartment of Oncology, Taikang Xianlin Drum Tower Hospital, Nanjing University School of Medicine, Nanjing, Jiangsu, P.R. ChinaDepartment of Oncology, Taikang Xianlin Drum Tower Hospital, Nanjing University School of Medicine, Nanjing, Jiangsu, P.R. ChinaLncRNA PTENP1 is a competitive endogenous RNA (ceRNA) involved in decoying miR-106b in multiple diseases. This study investigates the interaction of PTENP1 and miR-106b in cell proliferation, apoptosis and epithelial–mesenchymal transition (EMT) in cervical cancer. The expressions of PTENP1, miR-106b and PTEN were determined in cervical cancer tissues, adjacent normal tissues, cervical cancer cells (HeLa, SiHa, C33A and CasKi) and normal cervical epithelial H8 cells. Up-regulation of PTENP1 and down-regulation of miR-106b were conducted in HeLa and CasKi cells by transfecting cells with corresponding miRNA mimics and inhibitors. Bioinformatics analysis, luciferase reporter assay and RNA-pull down assay were performed to verify the association of miR-106b, PTEN, and PTENP1. Cell growth and cell apoptosis were determined by CCK-8 and flow cytometry analysis. It was found that the expressions of PTENP1 and PTEN were up-regulated and that of miR-106b were down-regulated in cervical cancer tissues and cells. PTENP1 localized in cytoplasm and competitively bound to miR-106b. Up-regulation of PTENP1 and down-regulation of miR-106b contributed to increased expressions of PTEN and E-cadherin. Decreased expression of miR-106b, ZEB1, Snail and Vimentin, resulted in inhibiting cell proliferation and promoting cell apoptosis. Over-expression of PTENP1 and miR-106b accelerated cell proliferation and slowed down cell apoptosis. miR-106b inhibited the expression of PTEN. Our results suggest that LncRNA PTENP1 inhibits cervical cancer progression by competitively binding to miR-106b, leading to promote PTEN expression, inhibit cell proliferation and EMT and induce cell apoptosis in cervical cancer cellshttps://www.tandfonline.com/doi/10.1080/21691401.2019.1709852PTENP1miR-106bceRNAPTENcervical cancer
spellingShingle Yingrui Fan
Weiwei Sheng
Yi Meng
Yundi Cao
Rong Li
LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106b
Artificial Cells, Nanomedicine, and Biotechnology
PTENP1
miR-106b
ceRNA
PTEN
cervical cancer
title LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106b
title_full LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106b
title_fullStr LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106b
title_full_unstemmed LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106b
title_short LncRNA PTENP1 inhibits cervical cancer progression by suppressing miR-106b
title_sort lncrna ptenp1 inhibits cervical cancer progression by suppressing mir 106b
topic PTENP1
miR-106b
ceRNA
PTEN
cervical cancer
url https://www.tandfonline.com/doi/10.1080/21691401.2019.1709852
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AT yimeng lncrnaptenp1inhibitscervicalcancerprogressionbysuppressingmir106b
AT yundicao lncrnaptenp1inhibitscervicalcancerprogressionbysuppressingmir106b
AT rongli lncrnaptenp1inhibitscervicalcancerprogressionbysuppressingmir106b