Effects of Ire1 gene on virulence and pathogenicity of Candida albicans

With the extensive utilization of antifungal drugs, the drug resistance of Candida albicans is progressively intensifying, and the effect of empirical treatment for C. albicans infection is not evident. There is an urgent need for novel strategies and methods for the treatment of C. albicans infecti...

Full description

Saved in:
Bibliographic Details
Main Authors: Zhao Huihai, Qin Lixia, Li Mengyan, Jiang Mengyu, Cui Mengge, Wang Hua, Hou Baohua, Wang Fukun, Jia Keran
Format: Article
Language:English
Published: De Gruyter 2025-04-01
Series:Open Life Sciences
Subjects:
Online Access:https://doi.org/10.1515/biol-2022-1062
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850204992317685760
author Zhao Huihai
Qin Lixia
Li Mengyan
Jiang Mengyu
Cui Mengge
Wang Hua
Hou Baohua
Wang Fukun
Jia Keran
author_facet Zhao Huihai
Qin Lixia
Li Mengyan
Jiang Mengyu
Cui Mengge
Wang Hua
Hou Baohua
Wang Fukun
Jia Keran
author_sort Zhao Huihai
collection DOAJ
description With the extensive utilization of antifungal drugs, the drug resistance of Candida albicans is progressively intensifying, and the effect of empirical treatment for C. albicans infection is not evident. There is an urgent need for novel strategies and methods for the treatment of C. albicans infection. Our study utilized the previously constructed C. albicans Ire1 double gene deletion strain to explore the influence of the Ire1 on endoplasmic reticulum (ER) stress and pathogenicity of C. albicans through drug stress phenotype testing, biofilm and hyphomycete formation testing, and mouse systemic infection testing. The results indicate that Ire1 is involved in maintaining the integrity of the C. albicans cell wall and influencing the hyphal formation ability of C. albicans. Concurrently, the deletion of the Ire1 increased the sensitivity of C. albicans to the ER stress agents tunicamycin and dithiothreitol and diminished the biofilm formation ability of C. albicans in vitro, resulting in significant inhibition of the growth of C. albicans. In mouse models, the deletion of Ire1 completely nullified the virulence and pathogenicity of C. albicans in the tail vein infection. In conclusion, Ire1 might be a key target for the potential development of new therapeutic drugs and vaccines.
format Article
id doaj-art-e5250755eb5f4382b4c628686f98a8cf
institution OA Journals
issn 2391-5412
language English
publishDate 2025-04-01
publisher De Gruyter
record_format Article
series Open Life Sciences
spelling doaj-art-e5250755eb5f4382b4c628686f98a8cf2025-08-20T02:11:11ZengDe GruyterOpen Life Sciences2391-54122025-04-012011032410.1515/biol-2022-1062Effects of Ire1 gene on virulence and pathogenicity of Candida albicansZhao Huihai0Qin Lixia1Li Mengyan2Jiang Mengyu3Cui Mengge4Wang Hua5Hou Baohua6Wang Fukun7Jia Keran8Clinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaClinical Laboratory, The 980Th Hospital of PLA Joint Logistical Support Force (Bethune International Peace Hospital), 398 Zhongshan Road, Shijiazhuang, Hebei, 050082, People’s Republic of ChinaWith the extensive utilization of antifungal drugs, the drug resistance of Candida albicans is progressively intensifying, and the effect of empirical treatment for C. albicans infection is not evident. There is an urgent need for novel strategies and methods for the treatment of C. albicans infection. Our study utilized the previously constructed C. albicans Ire1 double gene deletion strain to explore the influence of the Ire1 on endoplasmic reticulum (ER) stress and pathogenicity of C. albicans through drug stress phenotype testing, biofilm and hyphomycete formation testing, and mouse systemic infection testing. The results indicate that Ire1 is involved in maintaining the integrity of the C. albicans cell wall and influencing the hyphal formation ability of C. albicans. Concurrently, the deletion of the Ire1 increased the sensitivity of C. albicans to the ER stress agents tunicamycin and dithiothreitol and diminished the biofilm formation ability of C. albicans in vitro, resulting in significant inhibition of the growth of C. albicans. In mouse models, the deletion of Ire1 completely nullified the virulence and pathogenicity of C. albicans in the tail vein infection. In conclusion, Ire1 might be a key target for the potential development of new therapeutic drugs and vaccines.https://doi.org/10.1515/biol-2022-1062candida albicansire1endoplasmic reticulumvirulence
spellingShingle Zhao Huihai
Qin Lixia
Li Mengyan
Jiang Mengyu
Cui Mengge
Wang Hua
Hou Baohua
Wang Fukun
Jia Keran
Effects of Ire1 gene on virulence and pathogenicity of Candida albicans
Open Life Sciences
candida albicans
ire1
endoplasmic reticulum
virulence
title Effects of Ire1 gene on virulence and pathogenicity of Candida albicans
title_full Effects of Ire1 gene on virulence and pathogenicity of Candida albicans
title_fullStr Effects of Ire1 gene on virulence and pathogenicity of Candida albicans
title_full_unstemmed Effects of Ire1 gene on virulence and pathogenicity of Candida albicans
title_short Effects of Ire1 gene on virulence and pathogenicity of Candida albicans
title_sort effects of ire1 gene on virulence and pathogenicity of candida albicans
topic candida albicans
ire1
endoplasmic reticulum
virulence
url https://doi.org/10.1515/biol-2022-1062
work_keys_str_mv AT zhaohuihai effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT qinlixia effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT limengyan effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT jiangmengyu effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT cuimengge effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT wanghua effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT houbaohua effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT wangfukun effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans
AT jiakeran effectsofire1geneonvirulenceandpathogenicityofcandidaalbicans