Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice

The specific role that prolactin plays in lactational infertility, as distinct from other suckling or metabolic cues, remains unresolved. Here, deletion of the prolactin receptor (Prlr) from forebrain neurons or arcuate kisspeptin neurons resulted in failure to maintain normal lactation-induced supp...

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Main Authors: Eleni CR Hackwell, Sharon R Ladyman, Jenny Clarkson, H James McQuillan, Ulrich Boehm, Allan E Herbison, Rosemary SE Brown, David R Grattan
Format: Article
Language:English
Published: eLife Sciences Publications Ltd 2025-01-01
Series:eLife
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Online Access:https://elifesciences.org/articles/94570
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author Eleni CR Hackwell
Sharon R Ladyman
Jenny Clarkson
H James McQuillan
Ulrich Boehm
Allan E Herbison
Rosemary SE Brown
David R Grattan
author_facet Eleni CR Hackwell
Sharon R Ladyman
Jenny Clarkson
H James McQuillan
Ulrich Boehm
Allan E Herbison
Rosemary SE Brown
David R Grattan
author_sort Eleni CR Hackwell
collection DOAJ
description The specific role that prolactin plays in lactational infertility, as distinct from other suckling or metabolic cues, remains unresolved. Here, deletion of the prolactin receptor (Prlr) from forebrain neurons or arcuate kisspeptin neurons resulted in failure to maintain normal lactation-induced suppression of estrous cycles. Kisspeptin immunoreactivity and pulsatile LH secretion were increased in these mice, even in the presence of ongoing suckling stimulation and lactation. GCaMP fibre photometry of arcuate kisspeptin neurons revealed that the normal episodic activity of these neurons is rapidly suppressed in pregnancy and this was maintained throughout early lactation. Deletion of Prlr from arcuate kisspeptin neurons resulted in early reactivation of episodic activity of kisspeptin neurons prior to a premature return of reproductive cycles in early lactation. These observations show dynamic variation in arcuate kisspeptin neuronal activity associated with the hormonal changes of pregnancy and lactation, and provide direct evidence that prolactin action on arcuate kisspeptin neurons is necessary for suppressing fertility during lactation in mice.
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institution Kabale University
issn 2050-084X
language English
publishDate 2025-01-01
publisher eLife Sciences Publications Ltd
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series eLife
spelling doaj-art-e4c6951ee09c4bed8efe01cce9246f072025-08-20T03:31:16ZengeLife Sciences Publications LtdeLife2050-084X2025-01-011310.7554/eLife.94570Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in miceEleni CR Hackwell0Sharon R Ladyman1Jenny Clarkson2H James McQuillan3Ulrich Boehm4https://orcid.org/0000-0003-2436-6907Allan E Herbison5https://orcid.org/0000-0002-9615-3022Rosemary SE Brown6David R Grattan7https://orcid.org/0000-0001-5606-2559Centre for Neuroendocrinology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Department of Anatomy, School of Biomedical Sciences, University of Otago, Dunedin, New ZealandCentre for Neuroendocrinology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Department of Anatomy, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Maurice Wilkins Centre for Molecular Biodiscovery, Auckland, New ZealandCentre for Neuroendocrinology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New ZealandCentre for Neuroendocrinology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Department of Anatomy, School of Biomedical Sciences, University of Otago, Dunedin, New ZealandSaarland University School of Medicine, Centre for Molecular Signalling (PZMS), Experimental Pharmacology, Homburg, GermanyDepartment of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, United KingdomCentre for Neuroendocrinology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New ZealandCentre for Neuroendocrinology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Department of Anatomy, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand; Maurice Wilkins Centre for Molecular Biodiscovery, Auckland, New ZealandThe specific role that prolactin plays in lactational infertility, as distinct from other suckling or metabolic cues, remains unresolved. Here, deletion of the prolactin receptor (Prlr) from forebrain neurons or arcuate kisspeptin neurons resulted in failure to maintain normal lactation-induced suppression of estrous cycles. Kisspeptin immunoreactivity and pulsatile LH secretion were increased in these mice, even in the presence of ongoing suckling stimulation and lactation. GCaMP fibre photometry of arcuate kisspeptin neurons revealed that the normal episodic activity of these neurons is rapidly suppressed in pregnancy and this was maintained throughout early lactation. Deletion of Prlr from arcuate kisspeptin neurons resulted in early reactivation of episodic activity of kisspeptin neurons prior to a premature return of reproductive cycles in early lactation. These observations show dynamic variation in arcuate kisspeptin neuronal activity associated with the hormonal changes of pregnancy and lactation, and provide direct evidence that prolactin action on arcuate kisspeptin neurons is necessary for suppressing fertility during lactation in mice.https://elifesciences.org/articles/94570neuroendocrinologyhypothalamuskisspeptinprolactinlactationinfertility
spellingShingle Eleni CR Hackwell
Sharon R Ladyman
Jenny Clarkson
H James McQuillan
Ulrich Boehm
Allan E Herbison
Rosemary SE Brown
David R Grattan
Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
eLife
neuroendocrinology
hypothalamus
kisspeptin
prolactin
lactation
infertility
title Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
title_full Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
title_fullStr Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
title_full_unstemmed Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
title_short Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
title_sort prolactin mediates a lactation induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
topic neuroendocrinology
hypothalamus
kisspeptin
prolactin
lactation
infertility
url https://elifesciences.org/articles/94570
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