Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress

Abstract The study’s goals are to fabricate PLGA nanoparticles (PNPs) loaded with apigenin (AP) and adenosine (AD) using a microfluidic preparation method to a standard emulsification method and investigate the possible heart-protective effects of AP-AD PNPs made using the emulsification method. Com...

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Main Authors: Ruixuan Li, Aixia Xu, Ye Chen, Yihui Li, Ru Fu, Weihong Jiang, Xiaogang Li
Format: Article
Language:English
Published: BMC 2024-11-01
Series:BMC Biotechnology
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Online Access:https://doi.org/10.1186/s12896-024-00912-y
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author Ruixuan Li
Aixia Xu
Ye Chen
Yihui Li
Ru Fu
Weihong Jiang
Xiaogang Li
author_facet Ruixuan Li
Aixia Xu
Ye Chen
Yihui Li
Ru Fu
Weihong Jiang
Xiaogang Li
author_sort Ruixuan Li
collection DOAJ
description Abstract The study’s goals are to fabricate PLGA nanoparticles (PNPs) loaded with apigenin (AP) and adenosine (AD) using a microfluidic preparation method to a standard emulsification method and investigate the possible heart-protective effects of AP-AD PNPs made using the emulsification method. Compared to microfluidics, the emulsification method fabricated small-size nanoparticles, which are better at encapsulating drugs, retaining more drugs, and having a low viscosity for the myocardial infarction (MI) injection. TheMI model was developed using SD rats injected under the skin with 85 mg/kg doxorubicin (DOX) for 2 days. The metabolic results showed that our AP-AD PNPs accelerated the blood flow in rats with MI, which increased the amounts of AP and AD in the circulatory system. This led to significant improvements in the cardiac index and lower amounts of AST, LDH, and CK in the blood. A histopathological study using Hematoxylin&eosin, and TUNEL staining showed that cardiac function had improved and apoptosis had decreased. Moreover, tests that checked the amounts of IL-6, TNF-α, NO, GSH, MDA, and SOD showed that AP-AD PNPs may help treat MI by reducing oxidative stress and inflammation, making it a potentially useful therapeutic approach.
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issn 1472-6750
language English
publishDate 2024-11-01
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spelling doaj-art-e3a83ae6039e4778855fc53418516a392025-08-20T02:50:08ZengBMCBMC Biotechnology1472-67502024-11-0124111410.1186/s12896-024-00912-yFabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stressRuixuan Li0Aixia Xu1Ye Chen2Yihui Li3Ru Fu4Weihong Jiang5Xiaogang Li6Department of Cardiology, The Third Xiangya Hospital of Central South UniversityDepartment of Endocrinology, Changsha Central HospitalDepartment of Cardiology, The Third Xiangya Hospital of Central South UniversityDepartment of Cardiology, The Third Xiangya Hospital of Central South UniversityDepartment of Cardiology, The Third Xiangya Hospital of Central South UniversityDepartment of Cardiology, The Third Xiangya Hospital of Central South UniversityDepartment of Cardiology, The Third Xiangya Hospital of Central South UniversityAbstract The study’s goals are to fabricate PLGA nanoparticles (PNPs) loaded with apigenin (AP) and adenosine (AD) using a microfluidic preparation method to a standard emulsification method and investigate the possible heart-protective effects of AP-AD PNPs made using the emulsification method. Compared to microfluidics, the emulsification method fabricated small-size nanoparticles, which are better at encapsulating drugs, retaining more drugs, and having a low viscosity for the myocardial infarction (MI) injection. TheMI model was developed using SD rats injected under the skin with 85 mg/kg doxorubicin (DOX) for 2 days. The metabolic results showed that our AP-AD PNPs accelerated the blood flow in rats with MI, which increased the amounts of AP and AD in the circulatory system. This led to significant improvements in the cardiac index and lower amounts of AST, LDH, and CK in the blood. A histopathological study using Hematoxylin&eosin, and TUNEL staining showed that cardiac function had improved and apoptosis had decreased. Moreover, tests that checked the amounts of IL-6, TNF-α, NO, GSH, MDA, and SOD showed that AP-AD PNPs may help treat MI by reducing oxidative stress and inflammation, making it a potentially useful therapeutic approach.https://doi.org/10.1186/s12896-024-00912-yMyocardial infarctionApigeninAdenosinePLGA nanoparticlesNanomedicineEmulsification method
spellingShingle Ruixuan Li
Aixia Xu
Ye Chen
Yihui Li
Ru Fu
Weihong Jiang
Xiaogang Li
Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress
BMC Biotechnology
Myocardial infarction
Apigenin
Adenosine
PLGA nanoparticles
Nanomedicine
Emulsification method
title Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress
title_full Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress
title_fullStr Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress
title_full_unstemmed Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress
title_short Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress
title_sort fabrication of apigenin and adenosine loaded nanoparticles against doxorubicin induced myocardial infarction by reducing inflammation and oxidative stress
topic Myocardial infarction
Apigenin
Adenosine
PLGA nanoparticles
Nanomedicine
Emulsification method
url https://doi.org/10.1186/s12896-024-00912-y
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