Effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta

Background: Adequate placental perfusion is important for fetal development and well-being, but the effect of tocolysis on placental perfusion is unclear. The aim of this study was to evaluate changes in fetal cardiac output distribution to the placenta following ritodrine tocolysis. Methods: This r...

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Main Authors: Ryuichi Shimaoka, Tomomi Shiga, Ken-ichirou Morishige
Format: Article
Language:English
Published: IMR Press 2021-10-01
Series:Clinical and Experimental Obstetrics & Gynecology
Subjects:
Online Access:https://www.imrpress.com/journal/CEOG/48/5/10.31083/j.ceog4805181
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author Ryuichi Shimaoka
Tomomi Shiga
Ken-ichirou Morishige
author_facet Ryuichi Shimaoka
Tomomi Shiga
Ken-ichirou Morishige
author_sort Ryuichi Shimaoka
collection DOAJ
description Background: Adequate placental perfusion is important for fetal development and well-being, but the effect of tocolysis on placental perfusion is unclear. The aim of this study was to evaluate changes in fetal cardiac output distribution to the placenta following ritodrine tocolysis. Methods: This retrospective study involved 244 ultrasound findings in 142 singleton cases of appropriate for gestational age fetuses. The fetal cardiac output distribution to the placenta was defined and calculated as the percentage of umbilical vein flow volume (UVFV) based on the combined cardiac output (CCO). Ultrasound findings of 28 patients in the ritodrine group and 114 patients in the control group were compared using the unpaired t-test and Mann-Whitney U-test. Results: The CCO and UVFV increased as gestation progressed. On the other hand, distribution to the placenta was constant at approximately 15% from 28 to 35 weeks of gestation. Compared with the control group, the ritodrine group showed a significant increase in fetal heart rate, and the CCO also increased. The increase in UVFV in the ritodrine group was attributed to a significant increase in both the umbilical vessel diameter and blood flow velocity. As a result, distribution to the placenta in the ritodrine group was constant at about 20% and had a 5% increase at each week of gestation compared to the control group. Conclusions: To conclude, ritodrine tocolysis increased the fetal cardiac output distribution to the placenta. Additional research is required to determine whether tocolysis improves the placental perfusion in fetal growth restriction due to reduced placental perfusion.
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spelling doaj-art-e3a0418ec7ba45e9bc15e7c65eb787bf2025-08-20T03:48:32ZengIMR PressClinical and Experimental Obstetrics & Gynecology0390-66632021-10-014851126113110.31083/j.ceog4805181S0390-6663(21)00020-8Effect of ritodrine tocolysis on fetal cardiac output distribution to the placentaRyuichi Shimaoka0Tomomi Shiga1Ken-ichirou Morishige2Department of Obstetrics & Gynecology, Gifu University Graduate School of Medicine, 1-1, Yanagido, Gifu-shi, Gifu-prefecture, 501-1194 Gifu, JapanDepartment of Obstetrics & Gynecology, Gifu University Graduate School of Medicine, 1-1, Yanagido, Gifu-shi, Gifu-prefecture, 501-1194 Gifu, JapanDepartment of Obstetrics & Gynecology, Gifu University Graduate School of Medicine, 1-1, Yanagido, Gifu-shi, Gifu-prefecture, 501-1194 Gifu, JapanBackground: Adequate placental perfusion is important for fetal development and well-being, but the effect of tocolysis on placental perfusion is unclear. The aim of this study was to evaluate changes in fetal cardiac output distribution to the placenta following ritodrine tocolysis. Methods: This retrospective study involved 244 ultrasound findings in 142 singleton cases of appropriate for gestational age fetuses. The fetal cardiac output distribution to the placenta was defined and calculated as the percentage of umbilical vein flow volume (UVFV) based on the combined cardiac output (CCO). Ultrasound findings of 28 patients in the ritodrine group and 114 patients in the control group were compared using the unpaired t-test and Mann-Whitney U-test. Results: The CCO and UVFV increased as gestation progressed. On the other hand, distribution to the placenta was constant at approximately 15% from 28 to 35 weeks of gestation. Compared with the control group, the ritodrine group showed a significant increase in fetal heart rate, and the CCO also increased. The increase in UVFV in the ritodrine group was attributed to a significant increase in both the umbilical vessel diameter and blood flow velocity. As a result, distribution to the placenta in the ritodrine group was constant at about 20% and had a 5% increase at each week of gestation compared to the control group. Conclusions: To conclude, ritodrine tocolysis increased the fetal cardiac output distribution to the placenta. Additional research is required to determine whether tocolysis improves the placental perfusion in fetal growth restriction due to reduced placental perfusion.https://www.imrpress.com/journal/CEOG/48/5/10.31083/j.ceog4805181distribution to the placentadoppler ultrasoundfetal cardiac outputritodrineumbilical vein flow volume
spellingShingle Ryuichi Shimaoka
Tomomi Shiga
Ken-ichirou Morishige
Effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta
Clinical and Experimental Obstetrics & Gynecology
distribution to the placenta
doppler ultrasound
fetal cardiac output
ritodrine
umbilical vein flow volume
title Effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta
title_full Effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta
title_fullStr Effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta
title_full_unstemmed Effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta
title_short Effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta
title_sort effect of ritodrine tocolysis on fetal cardiac output distribution to the placenta
topic distribution to the placenta
doppler ultrasound
fetal cardiac output
ritodrine
umbilical vein flow volume
url https://www.imrpress.com/journal/CEOG/48/5/10.31083/j.ceog4805181
work_keys_str_mv AT ryuichishimaoka effectofritodrinetocolysisonfetalcardiacoutputdistributiontotheplacenta
AT tomomishiga effectofritodrinetocolysisonfetalcardiacoutputdistributiontotheplacenta
AT kenichiroumorishige effectofritodrinetocolysisonfetalcardiacoutputdistributiontotheplacenta