Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine

The development of medications to treat alcohol hangover is important for a large number of people, especially those, who perform responsible and dangerous types of work. The severity of the hangover syndrome is determined by the toxic effect of acetaldehyde and its metabolic rate, which decreases w...

Full description

Saved in:
Bibliographic Details
Main Authors: D. V. Kurkin, E. I. Morkovin, N. A. Osadchenko, L. P. Knyshova, D. A. Bakulin, E. E. Abrosimova, Yu. V. Gorbunova, I. N. Tyurenkov
Format: Article
Language:Russian
Published: Volgograd State Medical University, Pyatigorsk Medical and Pharmaceutical Institute 2019-12-01
Series:Фармация и фармакология (Пятигорск)
Subjects:
Online Access:https://www.pharmpharm.ru/jour/article/view/515
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850024553309274112
author D. V. Kurkin
E. I. Morkovin
N. A. Osadchenko
L. P. Knyshova
D. A. Bakulin
E. E. Abrosimova
Yu. V. Gorbunova
I. N. Tyurenkov
author_facet D. V. Kurkin
E. I. Morkovin
N. A. Osadchenko
L. P. Knyshova
D. A. Bakulin
E. E. Abrosimova
Yu. V. Gorbunova
I. N. Tyurenkov
author_sort D. V. Kurkin
collection DOAJ
description The development of medications to treat alcohol hangover is important for a large number of people, especially those, who perform responsible and dangerous types of work. The severity of the hangover syndrome is determined by the toxic effect of acetaldehyde and its metabolic rate, which decreases with the depletion of glutathione, worsening and prolonging the hangover.The aim of the article is to provide an experimental basis for use of acetylcysteine as a precursor of glutathione in treatment of psychological, neurological and cognitive symptoms of ethanol hangover.Materials and methods. The study was performed in male Wistar rats. The ethanol hangover was modeled via an acute ethanol injection (i.p., 3 g/kg). After awakening, the animals were divided into 2 groups, receiving acetylcysteine (p.o., 1 mg/ kg) or saline. The intact group of the animals received saline only. Before and after acetylcysteine or saline administration, the animals were assessed according to Combs and D'Alecy scale. The adhesive test, the open field test, the passive avoidance test and Morris water maze test were also performed twice. The liver glutathione level was assessed in sacrificed animals. Results. The control group animals showed signs of neurological deficits and cognitive impairment, including a decreased locomotion, motor deficits and a memory decline. The rats administered with acetylcysteine after the ethanol intoxication, had a less severe impairment associated with an improved performance in the adhesive test, Morris water maze test and the passive avoidance test, and demonstrated an increased locomotion in the open field test. The liver glutathione content in the animals treated with acetylcysteine, was comparable to the glutathione content in the liver of the animals not exposed to the ethanol intoxication.Conclusion. Against the background of an acute ethanol intoxication, an oral administration of acetylcysteine in the rats, promoted an increase in liver glutathione levels and led to a decrease in severity of neurological and cognitive deficits in the animals.
format Article
id doaj-art-e31a1e548d1f4c528537ea6966cc5050
institution DOAJ
issn 2307-9266
2413-2241
language Russian
publishDate 2019-12-01
publisher Volgograd State Medical University, Pyatigorsk Medical and Pharmaceutical Institute
record_format Article
series Фармация и фармакология (Пятигорск)
spelling doaj-art-e31a1e548d1f4c528537ea6966cc50502025-08-20T03:01:05ZrusVolgograd State Medical University, Pyatigorsk Medical and Pharmaceutical InstituteФармация и фармакология (Пятигорск)2307-92662413-22412019-12-017529129910.19163/2307-9266-2019-7-5-291-299320Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteineD. V. Kurkin0E. I. Morkovin1N. A. Osadchenko2L. P. Knyshova3D. A. Bakulin4E. E. Abrosimova5Yu. V. Gorbunova6I. N. Tyurenkov7Volgograd State Medical UniversityVolgograd State Medical UniversityVolgograd State Medical UniversityVolgograd State Medical UniversityVolgograd State Medical UniversityVolgograd State Medical UniversityVolgograd State Medical UniversityVolgograd State Medical UniversityThe development of medications to treat alcohol hangover is important for a large number of people, especially those, who perform responsible and dangerous types of work. The severity of the hangover syndrome is determined by the toxic effect of acetaldehyde and its metabolic rate, which decreases with the depletion of glutathione, worsening and prolonging the hangover.The aim of the article is to provide an experimental basis for use of acetylcysteine as a precursor of glutathione in treatment of psychological, neurological and cognitive symptoms of ethanol hangover.Materials and methods. The study was performed in male Wistar rats. The ethanol hangover was modeled via an acute ethanol injection (i.p., 3 g/kg). After awakening, the animals were divided into 2 groups, receiving acetylcysteine (p.o., 1 mg/ kg) or saline. The intact group of the animals received saline only. Before and after acetylcysteine or saline administration, the animals were assessed according to Combs and D'Alecy scale. The adhesive test, the open field test, the passive avoidance test and Morris water maze test were also performed twice. The liver glutathione level was assessed in sacrificed animals. Results. The control group animals showed signs of neurological deficits and cognitive impairment, including a decreased locomotion, motor deficits and a memory decline. The rats administered with acetylcysteine after the ethanol intoxication, had a less severe impairment associated with an improved performance in the adhesive test, Morris water maze test and the passive avoidance test, and demonstrated an increased locomotion in the open field test. The liver glutathione content in the animals treated with acetylcysteine, was comparable to the glutathione content in the liver of the animals not exposed to the ethanol intoxication.Conclusion. Against the background of an acute ethanol intoxication, an oral administration of acetylcysteine in the rats, promoted an increase in liver glutathione levels and led to a decrease in severity of neurological and cognitive deficits in the animals.https://www.pharmpharm.ru/jour/article/view/515ethanolpost-toxic statehangoveracetylcysteineratpreclinical study
spellingShingle D. V. Kurkin
E. I. Morkovin
N. A. Osadchenko
L. P. Knyshova
D. A. Bakulin
E. E. Abrosimova
Yu. V. Gorbunova
I. N. Tyurenkov
Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine
Фармация и фармакология (Пятигорск)
ethanol
post-toxic state
hangover
acetylcysteine
rat
preclinical study
title Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine
title_full Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine
title_fullStr Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine
title_full_unstemmed Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine
title_short Correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine
title_sort correction of psychological and neurological signs of alcohol hangover in rats with acetylcysteine
topic ethanol
post-toxic state
hangover
acetylcysteine
rat
preclinical study
url https://www.pharmpharm.ru/jour/article/view/515
work_keys_str_mv AT dvkurkin correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine
AT eimorkovin correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine
AT naosadchenko correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine
AT lpknyshova correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine
AT dabakulin correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine
AT eeabrosimova correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine
AT yuvgorbunova correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine
AT intyurenkov correctionofpsychologicalandneurologicalsignsofalcoholhangoverinratswithacetylcysteine