Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with Hypercalciuria

Hereditary hypophosphatemic rickets with hypercalciuria (HHRH) is a rare fibroblast growth factor-23-independent disorder caused by biallelic variants in the SLC34A3 gene. The disease severity varies, and patients have an increased risk of developing renal complications. Phosphate supplementation is...

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Main Authors: Luciana Pinto Valadares, Daniel Rocha de Carvalho
Format: Article
Language:English
Published: Galenos Yayincilik 2025-09-01
Series:JCRPE
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Online Access:https://www.jcrpe.org/articles/novel-variant-of-lessemgreaterslc34a3lessemgreater-in-a-compound-heterozygous-brazilian-girl-with-hereditary-hypophosphatemic-rickets-with-hypercalciuria/doi/jcrpe.galenos.2023.2023-5-2
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author Luciana Pinto Valadares
Daniel Rocha de Carvalho
author_facet Luciana Pinto Valadares
Daniel Rocha de Carvalho
author_sort Luciana Pinto Valadares
collection DOAJ
description Hereditary hypophosphatemic rickets with hypercalciuria (HHRH) is a rare fibroblast growth factor-23-independent disorder caused by biallelic variants in the SLC34A3 gene. The disease severity varies, and patients have an increased risk of developing renal complications. Phosphate supplementation is the standard of care and active vitamin D analogs are not indicated as they could worsen the hypercalciuria. We report a Brazilian girl with HHRH who presented with knee pain and progressive genu valgum deformity that became apparent from the age of eight years onwards. Nephrocalcinosis was also identified at age 13 years. Targeted next-generation sequencing for hereditary forms of rickets detected compound heterozygous pathogenic variants in SLC34A3, including a novel missense variant c.1217G>T (p.Gly406Val). Compliance to oral phosphorus therapy was suboptimal and adjunctive chlorthalidone therapy improved hypercalciuria. This report highlights the phenotypic variability and also expands the list of SLC34A3 variants associated with HHRH. An accurate diagnosis is key for optimal treatment. Of note, thiazide diuretics may be useful as adjunctive therapy for controlling hypercalciuria.
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publishDate 2025-09-01
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spelling doaj-art-e2ffb020458c4b3da6ccd0406fd066d32025-08-25T06:35:31ZengGalenos YayincilikJCRPE1308-57271308-57352025-09-0117332633110.4274/jcrpe.galenos.2023.2023-5-2Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with HypercalciuriaLuciana Pinto Valadares0https://orcid.org/0000-0002-4848-306XDaniel Rocha de Carvalho1https://orcid.org/0000-0002-3410-9704SARAH Network of Rehabilitation Hospitals, Clinic of Osteometabolic Disorders, Brasília, BrazilSARAH Network of Rehabilitation Hospitals, Clinic of Medical Genetics, Brasília, BrazilHereditary hypophosphatemic rickets with hypercalciuria (HHRH) is a rare fibroblast growth factor-23-independent disorder caused by biallelic variants in the SLC34A3 gene. The disease severity varies, and patients have an increased risk of developing renal complications. Phosphate supplementation is the standard of care and active vitamin D analogs are not indicated as they could worsen the hypercalciuria. We report a Brazilian girl with HHRH who presented with knee pain and progressive genu valgum deformity that became apparent from the age of eight years onwards. Nephrocalcinosis was also identified at age 13 years. Targeted next-generation sequencing for hereditary forms of rickets detected compound heterozygous pathogenic variants in SLC34A3, including a novel missense variant c.1217G>T (p.Gly406Val). Compliance to oral phosphorus therapy was suboptimal and adjunctive chlorthalidone therapy improved hypercalciuria. This report highlights the phenotypic variability and also expands the list of SLC34A3 variants associated with HHRH. An accurate diagnosis is key for optimal treatment. Of note, thiazide diuretics may be useful as adjunctive therapy for controlling hypercalciuria.https://www.jcrpe.org/articles/novel-variant-of-lessemgreaterslc34a3lessemgreater-in-a-compound-heterozygous-brazilian-girl-with-hereditary-hypophosphatemic-rickets-with-hypercalciuria/doi/jcrpe.galenos.2023.2023-5-2hereditary hypophosphatemic rickets with hypercalciuriascl34a3 pathogenic variantshypercalciuria
spellingShingle Luciana Pinto Valadares
Daniel Rocha de Carvalho
Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with Hypercalciuria
JCRPE
hereditary hypophosphatemic rickets with hypercalciuria
scl34a3 pathogenic variants
hypercalciuria
title Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with Hypercalciuria
title_full Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with Hypercalciuria
title_fullStr Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with Hypercalciuria
title_full_unstemmed Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with Hypercalciuria
title_short Novel Variant of SLC34A3 in a Compound Heterozygous Brazilian Girl with Hereditary Hypophosphatemic Rickets with Hypercalciuria
title_sort novel variant of slc34a3 in a compound heterozygous brazilian girl with hereditary hypophosphatemic rickets with hypercalciuria
topic hereditary hypophosphatemic rickets with hypercalciuria
scl34a3 pathogenic variants
hypercalciuria
url https://www.jcrpe.org/articles/novel-variant-of-lessemgreaterslc34a3lessemgreater-in-a-compound-heterozygous-brazilian-girl-with-hereditary-hypophosphatemic-rickets-with-hypercalciuria/doi/jcrpe.galenos.2023.2023-5-2
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AT danielrochadecarvalho novelvariantofslc34a3inacompoundheterozygousbraziliangirlwithhereditaryhypophosphatemicricketswithhypercalciuria