Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A

Xeroderma pigmentosum group A (XPA) is an inherited skin disorder characterized by sensitivity to ultraviolet radiation. In Maghrebi patients, a homozygous mutation in exon 6 of the XPA gene (c.682C>T) results in the introduction of a premature termination codon. Using CRISPR/Cas9-mediated gene e...

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Main Authors: Halida P. Widyastuti, Babet van der Vaart, Spyridon T. Pachis, Christian Freund, Xavier Gidrol, Karine Raymond
Format: Article
Language:English
Published: Elsevier 2024-12-01
Series:Stem Cell Research
Online Access:http://www.sciencedirect.com/science/article/pii/S1873506124002629
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author Halida P. Widyastuti
Babet van der Vaart
Spyridon T. Pachis
Christian Freund
Xavier Gidrol
Karine Raymond
author_facet Halida P. Widyastuti
Babet van der Vaart
Spyridon T. Pachis
Christian Freund
Xavier Gidrol
Karine Raymond
author_sort Halida P. Widyastuti
collection DOAJ
description Xeroderma pigmentosum group A (XPA) is an inherited skin disorder characterized by sensitivity to ultraviolet radiation. In Maghrebi patients, a homozygous mutation in exon 6 of the XPA gene (c.682C>T) results in the introduction of a premature termination codon. Using CRISPR/Cas9-mediated gene editing, this mutation was introduced into the well-characterized LUMCi004-A line. The resulting hiPSC line showed typical morphology, expressed markers of the undifferentiated state, was able to differentiate into the three germ layers in vitro and displayed a normal karyotype. When paired with its isogenic counterpart, this line represents a valuable resource to model the disease.
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issn 1873-5061
language English
publishDate 2024-12-01
publisher Elsevier
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series Stem Cell Research
spelling doaj-art-e2eba29f88c0494fae6345de55264eb72025-08-20T02:38:17ZengElsevierStem Cell Research1873-50612024-12-018110356410.1016/j.scr.2024.103564Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group AHalida P. Widyastuti0Babet van der Vaart1Spyridon T. Pachis2Christian Freund3Xavier Gidrol4Karine Raymond5Department of Anatomy and Embryology, Leiden University Medical Center, Leiden, Netherlands; The Novo Nordisk Foundation Center for Stem Cell Medicine (reNEW), Leiden University Medical Center, Leiden, NetherlandsDepartment of Anatomy and Embryology, Leiden University Medical Center, Leiden, Netherlands; The Novo Nordisk Foundation Center for Stem Cell Medicine (reNEW), Leiden University Medical Center, Leiden, Netherlands; Leiden hiPSC Centre, Leiden University Medical Center, Leiden, NetherlandsDepartment of Anatomy and Embryology, Leiden University Medical Center, Leiden, Netherlands; The Novo Nordisk Foundation Center for Stem Cell Medicine (reNEW), Leiden University Medical Center, Leiden, NetherlandsDepartment of Anatomy and Embryology, Leiden University Medical Center, Leiden, Netherlands; The Novo Nordisk Foundation Center for Stem Cell Medicine (reNEW), Leiden University Medical Center, Leiden, Netherlands; Leiden hiPSC Centre, Leiden University Medical Center, Leiden, NetherlandsUniversity of Grenoble Alpes, CEA, INSERM, IRIG, UA13 BGE, Biomics, Grenoble, FranceDepartment of Anatomy and Embryology, Leiden University Medical Center, Leiden, Netherlands; The Novo Nordisk Foundation Center for Stem Cell Medicine (reNEW), Leiden University Medical Center, Leiden, Netherlands; University of Grenoble Alpes, CEA, INSERM, IRIG, UA13 BGE, Biomics, Grenoble, France; Corresponding author at: Sylviusweg 62, 2333 BE Leiden, Netherlands.Xeroderma pigmentosum group A (XPA) is an inherited skin disorder characterized by sensitivity to ultraviolet radiation. In Maghrebi patients, a homozygous mutation in exon 6 of the XPA gene (c.682C>T) results in the introduction of a premature termination codon. Using CRISPR/Cas9-mediated gene editing, this mutation was introduced into the well-characterized LUMCi004-A line. The resulting hiPSC line showed typical morphology, expressed markers of the undifferentiated state, was able to differentiate into the three germ layers in vitro and displayed a normal karyotype. When paired with its isogenic counterpart, this line represents a valuable resource to model the disease.http://www.sciencedirect.com/science/article/pii/S1873506124002629
spellingShingle Halida P. Widyastuti
Babet van der Vaart
Spyridon T. Pachis
Christian Freund
Xavier Gidrol
Karine Raymond
Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A
Stem Cell Research
title Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A
title_full Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A
title_fullStr Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A
title_full_unstemmed Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A
title_short Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A
title_sort generation of xpa p arg228t mutant lumci004 a cell line for modeling xeroderma pigmentosum group a
url http://www.sciencedirect.com/science/article/pii/S1873506124002629
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