Engineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injury

Abstract Small interfering RNA (siRNA) therapies hold great potential for treating myocardial ischemia-reperfusion injury (MIRI); while their practical application is limited by the low bioavailability, off-target effects, and poor therapeutic efficacy. Here, we present an innovative engineered neut...

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Main Authors: Yaohui Jiang, Rongyan Jiang, Zequn Xia, Meng Guo, Yanan Fu, Xiaocheng Wang, Jun Xie
Format: Article
Language:English
Published: BMC 2025-02-01
Series:Journal of Nanobiotechnology
Subjects:
Online Access:https://doi.org/10.1186/s12951-025-03172-w
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author Yaohui Jiang
Rongyan Jiang
Zequn Xia
Meng Guo
Yanan Fu
Xiaocheng Wang
Jun Xie
author_facet Yaohui Jiang
Rongyan Jiang
Zequn Xia
Meng Guo
Yanan Fu
Xiaocheng Wang
Jun Xie
author_sort Yaohui Jiang
collection DOAJ
description Abstract Small interfering RNA (siRNA) therapies hold great potential for treating myocardial ischemia-reperfusion injury (MIRI); while their practical application is limited by the low bioavailability, off-target effects, and poor therapeutic efficacy. Here, we present an innovative engineered neutrophil membrane-camouflaged nanocomplex for targeted siRNA delivery and effective MIRI therapy. A nanoparticle (NP)-based siRNA delivery system, namely MNM/siRNA NPs, is camouflaged with neutrophil membranes modified by hemagglutinin (HA) and integrins. Our comprehensive in vitro studies show that MNM/siRNA NPs effectively facilitate endosomal escape through HA, achieve excellent targeting via integrins, and significantly reduce integrin α9 expression. Furthermore, in MIRI mice, we identify integrin α9 as a potential target for MIRI therapy and demonstrate that MNM/siRNA NPs significantly decrease myocardial infarction area and improve cardiac function by reducing neutrophil recruitment, neutrophil extracellular trap (NET) and microthrombus formation. These findings highlight the engineered membrane-camouflaged NPs as a promising siRNA delivery platform, offering an effective treatment strategy for MIRI.
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id doaj-art-e2e5d255a09c4167bdfc87b4c50770f0
institution OA Journals
issn 1477-3155
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publishDate 2025-02-01
publisher BMC
record_format Article
series Journal of Nanobiotechnology
spelling doaj-art-e2e5d255a09c4167bdfc87b4c50770f02025-08-20T02:15:02ZengBMCJournal of Nanobiotechnology1477-31552025-02-0123111910.1186/s12951-025-03172-wEngineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injuryYaohui Jiang0Rongyan Jiang1Zequn Xia2Meng Guo3Yanan Fu4Xiaocheng Wang5Jun Xie6Department of Cardiology, Nanjing Drum Tower Hospital, Affiliated Hospital of Medical School, Nanjing UniversityDepartment of Cardiology, Affiliated Bozhou Hospital of Anhui Medical UniversityDepartment of Cardiology, Nanjing Drum Tower Hospital, Affiliated Hospital of Medical School, Nanjing UniversityDepartment of Cardiology, Nanjing Drum Tower Hospital, Affiliated Hospital of Medical School, Nanjing UniversityDepartment of Cardiology, National Cardiovascular Disease Regional Center for Anhui, The First Affiliated Hospital of Anhui Medical UniversityDepartment of Rheumatology and Immunology, The First Affiliated Hospital of Anhui Medical UniversityDepartment of Cardiology, Nanjing Drum Tower Hospital, Affiliated Hospital of Medical School, Nanjing UniversityAbstract Small interfering RNA (siRNA) therapies hold great potential for treating myocardial ischemia-reperfusion injury (MIRI); while their practical application is limited by the low bioavailability, off-target effects, and poor therapeutic efficacy. Here, we present an innovative engineered neutrophil membrane-camouflaged nanocomplex for targeted siRNA delivery and effective MIRI therapy. A nanoparticle (NP)-based siRNA delivery system, namely MNM/siRNA NPs, is camouflaged with neutrophil membranes modified by hemagglutinin (HA) and integrins. Our comprehensive in vitro studies show that MNM/siRNA NPs effectively facilitate endosomal escape through HA, achieve excellent targeting via integrins, and significantly reduce integrin α9 expression. Furthermore, in MIRI mice, we identify integrin α9 as a potential target for MIRI therapy and demonstrate that MNM/siRNA NPs significantly decrease myocardial infarction area and improve cardiac function by reducing neutrophil recruitment, neutrophil extracellular trap (NET) and microthrombus formation. These findings highlight the engineered membrane-camouflaged NPs as a promising siRNA delivery platform, offering an effective treatment strategy for MIRI.https://doi.org/10.1186/s12951-025-03172-wEngineered neutrophil membraneMyocardial ischemia-reperfusion injuryNeutrophilsIntegrin α9siRNA
spellingShingle Yaohui Jiang
Rongyan Jiang
Zequn Xia
Meng Guo
Yanan Fu
Xiaocheng Wang
Jun Xie
Engineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injury
Journal of Nanobiotechnology
Engineered neutrophil membrane
Myocardial ischemia-reperfusion injury
Neutrophils
Integrin α9
siRNA
title Engineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injury
title_full Engineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injury
title_fullStr Engineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injury
title_full_unstemmed Engineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injury
title_short Engineered neutrophil membrane-camouflaged nanocomplexes for targeted siRNA delivery against myocardial ischemia reperfusion injury
title_sort engineered neutrophil membrane camouflaged nanocomplexes for targeted sirna delivery against myocardial ischemia reperfusion injury
topic Engineered neutrophil membrane
Myocardial ischemia-reperfusion injury
Neutrophils
Integrin α9
siRNA
url https://doi.org/10.1186/s12951-025-03172-w
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