mGem: The complexity of viral entry—one virus, many receptors

ABSTRACT Binding to cellular receptors initiates viral replication and dictates sites in the host infected by the virus. As illustrated by mammalian orthoreovirus (reovirus), viruses can bind several types of receptors using distinct capsid components to facilitate the viral entry steps of attachmen...

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Main Authors: Terence S. Dermody, Danica M. Sutherland
Format: Article
Language:English
Published: American Society for Microbiology 2025-03-01
Series:mBio
Subjects:
Online Access:https://journals.asm.org/doi/10.1128/mbio.02964-24
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author Terence S. Dermody
Danica M. Sutherland
author_facet Terence S. Dermody
Danica M. Sutherland
author_sort Terence S. Dermody
collection DOAJ
description ABSTRACT Binding to cellular receptors initiates viral replication and dictates sites in the host infected by the virus. As illustrated by mammalian orthoreovirus (reovirus), viruses can bind several types of receptors using distinct capsid components to facilitate the viral entry steps of attachment, internalization, and disassembly. The outer of the two concentric capsids of reovirus virions is formed by four viral proteins, three of which bind receptors. These capsid–receptor interactions mediate stepwise entry of reovirus, dictate viral tropism in infected animals, and expand the viral host range. Engagement of independent receptors by different capsid proteins is a property of many pathogenic viruses and illustrates common themes of receptor use in viral entry and disease.
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publisher American Society for Microbiology
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spelling doaj-art-e2904cdd7ca34186a42685f47999d7722025-08-20T02:52:41ZengAmerican Society for MicrobiologymBio2150-75112025-03-0116310.1128/mbio.02964-24mGem: The complexity of viral entry—one virus, many receptorsTerence S. Dermody0Danica M. Sutherland1Department of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, USADepartment of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, USAABSTRACT Binding to cellular receptors initiates viral replication and dictates sites in the host infected by the virus. As illustrated by mammalian orthoreovirus (reovirus), viruses can bind several types of receptors using distinct capsid components to facilitate the viral entry steps of attachment, internalization, and disassembly. The outer of the two concentric capsids of reovirus virions is formed by four viral proteins, three of which bind receptors. These capsid–receptor interactions mediate stepwise entry of reovirus, dictate viral tropism in infected animals, and expand the viral host range. Engagement of independent receptors by different capsid proteins is a property of many pathogenic viruses and illustrates common themes of receptor use in viral entry and disease.https://journals.asm.org/doi/10.1128/mbio.02964-24virusreceptorsneurotropismviral pathogenesis
spellingShingle Terence S. Dermody
Danica M. Sutherland
mGem: The complexity of viral entry—one virus, many receptors
mBio
virus
receptors
neurotropism
viral pathogenesis
title mGem: The complexity of viral entry—one virus, many receptors
title_full mGem: The complexity of viral entry—one virus, many receptors
title_fullStr mGem: The complexity of viral entry—one virus, many receptors
title_full_unstemmed mGem: The complexity of viral entry—one virus, many receptors
title_short mGem: The complexity of viral entry—one virus, many receptors
title_sort mgem the complexity of viral entry one virus many receptors
topic virus
receptors
neurotropism
viral pathogenesis
url https://journals.asm.org/doi/10.1128/mbio.02964-24
work_keys_str_mv AT terencesdermody mgemthecomplexityofviralentryonevirusmanyreceptors
AT danicamsutherland mgemthecomplexityofviralentryonevirusmanyreceptors