Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c mice

Abstract Galectin-3 regulates cell-cell and cell-extracellular matrix interactions in distinct tissues, including intestinal epithelial cells interfering with inflammatory responses. In the gut, galectin-3 stabilizes epithelial junctions and intestinal permeability. Here, it was investigated whether...

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Main Authors: PAULA C.P. SILVA, VICTOR F.S. FERREIRA, NATÁLIA G. MAÇÃO, RAFAEL M.S. LIMA, FELIPE S. LEMOS, FELIPE L. OLIVEIRA
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Language:English
Published: Academia Brasileira de Ciências 2025-03-01
Series:Anais da Academia Brasileira de Ciências
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Online Access:http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0001-37652025000100605&lng=en&tlng=en
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author PAULA C.P. SILVA
VICTOR F.S. FERREIRA
NATÁLIA G. MAÇÃO
RAFAEL M.S. LIMA
FELIPE S. LEMOS
FELIPE L. OLIVEIRA
author_facet PAULA C.P. SILVA
VICTOR F.S. FERREIRA
NATÁLIA G. MAÇÃO
RAFAEL M.S. LIMA
FELIPE S. LEMOS
FELIPE L. OLIVEIRA
author_sort PAULA C.P. SILVA
collection DOAJ
description Abstract Galectin-3 regulates cell-cell and cell-extracellular matrix interactions in distinct tissues, including intestinal epithelial cells interfering with inflammatory responses. In the gut, galectin-3 stabilizes epithelial junctions and intestinal permeability. Here, it was investigated whether gliadin (protein of gluten) can induce inflammatory reactions in the digestive system in the presence or absence of galectin-3. BALB/c wild-type (Lgals3+/+) and genetically mutated in LGALS3 (Lgals3-/-) mice were divided into controls (water) or orally supplemented with gliadin (100mg/day) for 30 days. Fragments of duodenum, jejunum, ileum, colon and rectum were processed for histological analysis and immunohistochemistry. Gliadin induced an inflammatory response in the gut of Lgals3+/+ and Lgals3-/- mice, but with high severity in the Lgals3-/- mice. Galectin-3 was significantly expressed by enterocytes in all fragments. However, it was drastically reduced after gliadin supplementation and directly associated with severe inflammation in all compartments of the gut. In parallel, gliadin supplemented Lgals3-/- mice showed significant inflammatory signals in the mucosa, including leukocyte infiltration in mucosal sites, hyperplasia of crypts, enhance of intraepithelial lymphocytes, and hypertrophy linked to accumulation of apoptotic bodies in the Peyer’s patches. These data suggested that galectin-3 plays protective roles in the gut submitted an inflammatory stress after gluten intake.
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spelling doaj-art-e28f3175cba64f60be39b7753405db3d2025-08-20T02:46:29ZengAcademia Brasileira de CiênciasAnais da Academia Brasileira de Ciências1678-26902025-03-0197110.1590/0001-3765202520240900Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c micePAULA C.P. SILVAhttps://orcid.org/0009-0004-4818-7043VICTOR F.S. FERREIRAhttps://orcid.org/0009-0009-7168-6429NATÁLIA G. MAÇÃOhttps://orcid.org/0009-0007-7155-8406RAFAEL M.S. LIMAhttps://orcid.org/0009-0007-7856-7732FELIPE S. LEMOShttps://orcid.org/0000-0003-3133-8373FELIPE L. OLIVEIRAhttps://orcid.org/0000-0001-6193-1616Abstract Galectin-3 regulates cell-cell and cell-extracellular matrix interactions in distinct tissues, including intestinal epithelial cells interfering with inflammatory responses. In the gut, galectin-3 stabilizes epithelial junctions and intestinal permeability. Here, it was investigated whether gliadin (protein of gluten) can induce inflammatory reactions in the digestive system in the presence or absence of galectin-3. BALB/c wild-type (Lgals3+/+) and genetically mutated in LGALS3 (Lgals3-/-) mice were divided into controls (water) or orally supplemented with gliadin (100mg/day) for 30 days. Fragments of duodenum, jejunum, ileum, colon and rectum were processed for histological analysis and immunohistochemistry. Gliadin induced an inflammatory response in the gut of Lgals3+/+ and Lgals3-/- mice, but with high severity in the Lgals3-/- mice. Galectin-3 was significantly expressed by enterocytes in all fragments. However, it was drastically reduced after gliadin supplementation and directly associated with severe inflammation in all compartments of the gut. In parallel, gliadin supplemented Lgals3-/- mice showed significant inflammatory signals in the mucosa, including leukocyte infiltration in mucosal sites, hyperplasia of crypts, enhance of intraepithelial lymphocytes, and hypertrophy linked to accumulation of apoptotic bodies in the Peyer’s patches. These data suggested that galectin-3 plays protective roles in the gut submitted an inflammatory stress after gluten intake.http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0001-37652025000100605&lng=en&tlng=enGalectin-3gliadinglutenintestinal mucosaceliac diseasegut inflammation
spellingShingle PAULA C.P. SILVA
VICTOR F.S. FERREIRA
NATÁLIA G. MAÇÃO
RAFAEL M.S. LIMA
FELIPE S. LEMOS
FELIPE L. OLIVEIRA
Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c mice
Anais da Academia Brasileira de Ciências
Galectin-3
gliadin
gluten
intestinal mucosa
celiac disease
gut inflammation
title Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c mice
title_full Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c mice
title_fullStr Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c mice
title_full_unstemmed Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c mice
title_short Galectin-3 organizes histological compartments associated with inflammatory reaction induced by gliadin in BALB/c mice
title_sort galectin 3 organizes histological compartments associated with inflammatory reaction induced by gliadin in balb c mice
topic Galectin-3
gliadin
gluten
intestinal mucosa
celiac disease
gut inflammation
url http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0001-37652025000100605&lng=en&tlng=en
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