CSE-Induced ER-Mitochondria Crosstalk Promotes Oxidative Stress and Impairs Bronchial Contractile Response

CSE-Induced ER-Mitochondria Crosstalk Promotes Oxidative Stress and Impairs Bronchial Contractile Response

Chronic obstructive pulmonary disease (COPD), whose main risk factor is cigarette smoking, is among the most prevalent diseases worldwide. Previous studies have shown that cigarette smoke extract (CSE) can directly affect pulmonary artery function independently of hypoxia resulting from the airway o...

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Bibliographic Details
Main Authors: Jorge Rodríguez-Pérez, Rosa Andreu-Martínez, Leila Pérez-Sánchez, Ana Hernández-García, Cecilia Muñoz-Calleja, Ángel Cogolludo, María J. Calzada
Format: Article
Language:English
Published: MDPI AG 2025-06-01
Series:Antioxidants
Subjects:
COPD
cigarette smoke
contractility
mitochondria
calcium
ROS
Online Access:https://www.mdpi.com/2076-3921/14/6/703
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