Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibition

Abstract Myotonic dystrophy type 2 (DM2), caused by CCTG repeat expansion, is a common adult-onset disorder characterized by myotonia and progressive muscle degeneration with no effective treatment. Here, we identified Tyrosyl-DNA phosphodiesterase 1 (TDP1) as a novel modifier for DM2 therapeutic in...

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Main Authors: Yingbao Zhu, Shengwei Xiao, Xinxin Guan, Haitao Deng, Liqiang Ai, Kaijing Fan, Jin Xue, Guangxu Li, Xiaoxue Bi, Qiao Xiao, Yuanjiang Huang, Lin Jiang, Wen Huang, Peng Jin, Ranhui Duan
Format: Article
Language:English
Published: Springer Nature 2025-03-01
Series:EMBO Molecular Medicine
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Online Access:https://doi.org/10.1038/s44321-025-00217-3
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author Yingbao Zhu
Shengwei Xiao
Xinxin Guan
Haitao Deng
Liqiang Ai
Kaijing Fan
Jin Xue
Guangxu Li
Xiaoxue Bi
Qiao Xiao
Yuanjiang Huang
Lin Jiang
Wen Huang
Peng Jin
Ranhui Duan
author_facet Yingbao Zhu
Shengwei Xiao
Xinxin Guan
Haitao Deng
Liqiang Ai
Kaijing Fan
Jin Xue
Guangxu Li
Xiaoxue Bi
Qiao Xiao
Yuanjiang Huang
Lin Jiang
Wen Huang
Peng Jin
Ranhui Duan
author_sort Yingbao Zhu
collection DOAJ
description Abstract Myotonic dystrophy type 2 (DM2), caused by CCTG repeat expansion, is a common adult-onset disorder characterized by myotonia and progressive muscle degeneration with no effective treatment. Here, we identified Tyrosyl-DNA phosphodiesterase 1 (TDP1) as a novel modifier for DM2 therapeutic intervention through a high-throughput chemical screening of 2160 compounds. Moreover, we detailed how both genetic and pharmacological inhibition of TDP1 translates to a cascade of beneficial effects, including improved motor functions, amelioration of progressive muscle degeneration, repair of muscle fiber damage, and normalization of aberrant molecular pathology. Remarkably, the TDP1 inhibition led to substantial CCTG repeat contractions, a mechanism that underlies the observed muscle toxicity and neurodegeneration. Our results highlighted the potential of TDP1 as a molecular target for addressing the complex interplay between repeat expansions and neuromuscular degeneration in DM2, hinting at broader applicability in a spectrum of repeat expansion disorders.
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institution Kabale University
issn 1757-4684
language English
publishDate 2025-03-01
publisher Springer Nature
record_format Article
series EMBO Molecular Medicine
spelling doaj-art-e09bd7e2b6c74f3ea6a6866dd08119b82025-08-20T03:53:58ZengSpringer NatureEMBO Molecular Medicine1757-46842025-03-0117596799210.1038/s44321-025-00217-3Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibitionYingbao Zhu0Shengwei Xiao1Xinxin Guan2Haitao Deng3Liqiang Ai4Kaijing Fan5Jin Xue6Guangxu Li7Xiaoxue Bi8Qiao Xiao9Yuanjiang Huang10Lin Jiang11Wen Huang12Peng Jin13Ranhui Duan14Furong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityDepartment of Human Genetics, Emory University School of MedicineFurong Laboratory, Center for Medical Genetics, School of Life Sciences, Central South UniversityAbstract Myotonic dystrophy type 2 (DM2), caused by CCTG repeat expansion, is a common adult-onset disorder characterized by myotonia and progressive muscle degeneration with no effective treatment. Here, we identified Tyrosyl-DNA phosphodiesterase 1 (TDP1) as a novel modifier for DM2 therapeutic intervention through a high-throughput chemical screening of 2160 compounds. Moreover, we detailed how both genetic and pharmacological inhibition of TDP1 translates to a cascade of beneficial effects, including improved motor functions, amelioration of progressive muscle degeneration, repair of muscle fiber damage, and normalization of aberrant molecular pathology. Remarkably, the TDP1 inhibition led to substantial CCTG repeat contractions, a mechanism that underlies the observed muscle toxicity and neurodegeneration. Our results highlighted the potential of TDP1 as a molecular target for addressing the complex interplay between repeat expansions and neuromuscular degeneration in DM2, hinting at broader applicability in a spectrum of repeat expansion disorders.https://doi.org/10.1038/s44321-025-00217-3DM2CCTG Repeat ExpansionChemical ScreenTDP1Repeat Instability
spellingShingle Yingbao Zhu
Shengwei Xiao
Xinxin Guan
Haitao Deng
Liqiang Ai
Kaijing Fan
Jin Xue
Guangxu Li
Xiaoxue Bi
Qiao Xiao
Yuanjiang Huang
Lin Jiang
Wen Huang
Peng Jin
Ranhui Duan
Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibition
EMBO Molecular Medicine
DM2
CCTG Repeat Expansion
Chemical Screen
TDP1
Repeat Instability
title Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibition
title_full Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibition
title_fullStr Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibition
title_full_unstemmed Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibition
title_short Modulating CCTG repeat expansion toxicity in DM2 Drosophila model through TDP1 inhibition
title_sort modulating cctg repeat expansion toxicity in dm2 drosophila model through tdp1 inhibition
topic DM2
CCTG Repeat Expansion
Chemical Screen
TDP1
Repeat Instability
url https://doi.org/10.1038/s44321-025-00217-3
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