A rare cause of mental motor retardation: recessive congenital methemoglobinemia type II

Recessive congenital methemoglobinemia (RCM) is a very rare disorder caused by NADH- cytochrome b5 reductase (cytb5r) deficiency. It has been classified into four types. Type I presents with mild cyanosis due to a significant deficiency of cytb5r in erythrocytes only. In type II, the deficien...

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Main Authors: Deniz Yüksel, Nesrin Senbil, Deniz Yilmaz, Neşe Yarali, Y K Yavuz Gürer
Format: Article
Language:English
Published: Hacettepe University Institute of Child Health 2009-04-01
Series:The Turkish Journal of Pediatrics
Online Access:https://turkjpediatr.org/article/view/2285
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author Deniz Yüksel
Nesrin Senbil
Deniz Yilmaz
Neşe Yarali
Y K Yavuz Gürer
author_facet Deniz Yüksel
Nesrin Senbil
Deniz Yilmaz
Neşe Yarali
Y K Yavuz Gürer
author_sort Deniz Yüksel
collection DOAJ
description Recessive congenital methemoglobinemia (RCM) is a very rare disorder caused by NADH- cytochrome b5 reductase (cytb5r) deficiency. It has been classified into four types. Type I presents with mild cyanosis due to a significant deficiency of cytb5r in erythrocytes only. In type II, the deficiency occurs in all tissues and causes growth and mental retardation and other neurological impairments. RCM types I and II are caused by a defect in a single gene, which is located on chromosome 22 (locus DIA 1: q 13.31-qter). Prenatal diagnosis is possible. Cyanosis can be well treated by 200-500 mg of ascorbic acid daily; there is no effective therapy for the progressive neurological impairments. This report presents two siblings with central cyanosis, growth retardation, mental retardation, microcephaly, dystonia and hypertonia diagnosed as RCM type II.
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issn 0041-4301
2791-6421
language English
publishDate 2009-04-01
publisher Hacettepe University Institute of Child Health
record_format Article
series The Turkish Journal of Pediatrics
spelling doaj-art-df0ce3d0bcc8457cbd4cbf9abf19119e2025-08-20T02:01:49ZengHacettepe University Institute of Child HealthThe Turkish Journal of Pediatrics0041-43012791-64212009-04-01512A rare cause of mental motor retardation: recessive congenital methemoglobinemia type IIDeniz Yüksel0Nesrin SenbilDeniz YilmazNeşe YaraliY K Yavuz GürerDr. Sami Ulus Child Health and Diseases Training and Research Hospital, Ankara, Turkey. Recessive congenital methemoglobinemia (RCM) is a very rare disorder caused by NADH- cytochrome b5 reductase (cytb5r) deficiency. It has been classified into four types. Type I presents with mild cyanosis due to a significant deficiency of cytb5r in erythrocytes only. In type II, the deficiency occurs in all tissues and causes growth and mental retardation and other neurological impairments. RCM types I and II are caused by a defect in a single gene, which is located on chromosome 22 (locus DIA 1: q 13.31-qter). Prenatal diagnosis is possible. Cyanosis can be well treated by 200-500 mg of ascorbic acid daily; there is no effective therapy for the progressive neurological impairments. This report presents two siblings with central cyanosis, growth retardation, mental retardation, microcephaly, dystonia and hypertonia diagnosed as RCM type II. https://turkjpediatr.org/article/view/2285
spellingShingle Deniz Yüksel
Nesrin Senbil
Deniz Yilmaz
Neşe Yarali
Y K Yavuz Gürer
A rare cause of mental motor retardation: recessive congenital methemoglobinemia type II
The Turkish Journal of Pediatrics
title A rare cause of mental motor retardation: recessive congenital methemoglobinemia type II
title_full A rare cause of mental motor retardation: recessive congenital methemoglobinemia type II
title_fullStr A rare cause of mental motor retardation: recessive congenital methemoglobinemia type II
title_full_unstemmed A rare cause of mental motor retardation: recessive congenital methemoglobinemia type II
title_short A rare cause of mental motor retardation: recessive congenital methemoglobinemia type II
title_sort rare cause of mental motor retardation recessive congenital methemoglobinemia type ii
url https://turkjpediatr.org/article/view/2285
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