Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis

Aim. To evaluate changes in the concentration of molecules that mark the neurodegenerative process, experimental spinal cord injuries (SCI) of various origins were studied.Materials and methods. SCI was modeled in six-month-old male Wistar rats by exposing the T10 vertebra to: carbon dioxide under a...

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Main Authors: D. I. Pozdnyakov, V. V. Kozlova, V. F. Reps
Format: Article
Language:Russian
Published: Federal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University) 2024-04-01
Series:Сеченовский вестник
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Online Access:https://www.sechenovmedj.com/jour/article/view/1058
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author D. I. Pozdnyakov
V. V. Kozlova
V. F. Reps
author_facet D. I. Pozdnyakov
V. V. Kozlova
V. F. Reps
author_sort D. I. Pozdnyakov
collection DOAJ
description Aim. To evaluate changes in the concentration of molecules that mark the neurodegenerative process, experimental spinal cord injuries (SCI) of various origins were studied.Materials and methods. SCI was modeled in six-month-old male Wistar rats by exposing the T10 vertebra to: carbon dioxide under a pressure of 2 N/cm2 (pneumocontusion); free-falling load of three weights of 1.12 N/cm2, 1.68 N/cm2, 1.96 N/cm2 (contusion injury); compression with forceps (compression injury); partial hemisection of the spinal cord; mechanical laminectomy using a mechanical drill. There were 6 rats in each group, including the intact control group. On the 28th day after a single application of SCI in rats, the concentrations of tumor necrosis factor-alpha (TNF-α), interleukin 6 (IL-6), albumin, thiobarbituric acid reactive substances (TBA-RS) and superoxide dismutase activity were assessed in the blood serum.Results. When modeling SCI of various origins in rats, the serum concentration of TNF-α increased (from 115.5% (p < 0.05) in mild contusion to 234.5% (p < 0.05) in compression trauma compared to intact control) as well as IL-6 (from 49.2% (p < 0.05) in mechanical laminectomy to 89.8% (p < 0.05) in hemisection compared with intact control), suggesting activation of inflammatory reactions. The concentration of albumin in the blood serum of rats with SCI was lower than that of intact animals, especially in the hemisection group – by 41.9% (p < 0.05). Animals with SCI had an increase in TBA-RS concentration ranging from 103.2% (p < 0.05) in mild contusion and compression to 135.5% (p < 0.05) in pneumocontusion, and a decrease in superoxide dismutase activity ranging from 26.3% (p < 0.05) in laminectomy to 31.7% (p < 0.05) in hemisection. At the same time, injuries caused by spinal compression and hemisection led to a more pronounced activation of the inflammatory process, as evidenced by the increased TNF-α content compared to other variants of SCI modeling.Conclusion. All SCI simulations resulted in equivalent activation of oxidative stress, while inflammation is more pronounced when reproducing compression injury and injury caused by spinal hemisection.
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spelling doaj-art-dde9e97d88764039bc65924d5431f4d72025-08-20T02:53:22ZrusFederal State Autonomous Educational Institution of Higher Education I.M. Sechenov First Moscow State Medical University of the Ministry of Health of the Russian Federation (Sechenov University)Сеченовский вестник2218-73322658-33482024-04-01151364610.47093/2218-7332.2024.15.1.36-46593Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesisD. I. Pozdnyakov0V. V. Kozlova1V. F. Reps2Pyatigorsk Medical and Pharmaceutical Institute; Pyatigorsk State Research Institute of Balneology, branch of the Federal Scientifi c and Clinical Center of Medical Rehabilitation and Balneology of the Federal Medical and Biological Agency of RussiaPyatigorsk Medical and Pharmaceutical Institute; Pyatigorsk State Research Institute of Balneology, branch of the Federal Scientifi c and Clinical Center of Medical Rehabilitation and Balneology of the Federal Medical and Biological Agency of RussiaPyatigorsk Medical and Pharmaceutical Institute; Pyatigorsk State Research Institute of Balneology, branch of the Federal Scientifi c and Clinical Center of Medical Rehabilitation and Balneology of the Federal Medical and Biological Agency of RussiaAim. To evaluate changes in the concentration of molecules that mark the neurodegenerative process, experimental spinal cord injuries (SCI) of various origins were studied.Materials and methods. SCI was modeled in six-month-old male Wistar rats by exposing the T10 vertebra to: carbon dioxide under a pressure of 2 N/cm2 (pneumocontusion); free-falling load of three weights of 1.12 N/cm2, 1.68 N/cm2, 1.96 N/cm2 (contusion injury); compression with forceps (compression injury); partial hemisection of the spinal cord; mechanical laminectomy using a mechanical drill. There were 6 rats in each group, including the intact control group. On the 28th day after a single application of SCI in rats, the concentrations of tumor necrosis factor-alpha (TNF-α), interleukin 6 (IL-6), albumin, thiobarbituric acid reactive substances (TBA-RS) and superoxide dismutase activity were assessed in the blood serum.Results. When modeling SCI of various origins in rats, the serum concentration of TNF-α increased (from 115.5% (p < 0.05) in mild contusion to 234.5% (p < 0.05) in compression trauma compared to intact control) as well as IL-6 (from 49.2% (p < 0.05) in mechanical laminectomy to 89.8% (p < 0.05) in hemisection compared with intact control), suggesting activation of inflammatory reactions. The concentration of albumin in the blood serum of rats with SCI was lower than that of intact animals, especially in the hemisection group – by 41.9% (p < 0.05). Animals with SCI had an increase in TBA-RS concentration ranging from 103.2% (p < 0.05) in mild contusion and compression to 135.5% (p < 0.05) in pneumocontusion, and a decrease in superoxide dismutase activity ranging from 26.3% (p < 0.05) in laminectomy to 31.7% (p < 0.05) in hemisection. At the same time, injuries caused by spinal compression and hemisection led to a more pronounced activation of the inflammatory process, as evidenced by the increased TNF-α content compared to other variants of SCI modeling.Conclusion. All SCI simulations resulted in equivalent activation of oxidative stress, while inflammation is more pronounced when reproducing compression injury and injury caused by spinal hemisection.https://www.sechenovmedj.com/jour/article/view/1058rehabilitationtumor necrosis factor-αinterleukin 6superoxide dismutasespinal trauma
spellingShingle D. I. Pozdnyakov
V. V. Kozlova
V. F. Reps
Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis
Сеченовский вестник
rehabilitation
tumor necrosis factor-α
interleukin 6
superoxide dismutase
spinal trauma
title Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis
title_full Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis
title_fullStr Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis
title_full_unstemmed Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis
title_short Serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis
title_sort serum markers of neuroinflammation and oxidative stress in modeling spinal injury of various genesis
topic rehabilitation
tumor necrosis factor-α
interleukin 6
superoxide dismutase
spinal trauma
url https://www.sechenovmedj.com/jour/article/view/1058
work_keys_str_mv AT dipozdnyakov serummarkersofneuroinflammationandoxidativestressinmodelingspinalinjuryofvariousgenesis
AT vvkozlova serummarkersofneuroinflammationandoxidativestressinmodelingspinalinjuryofvariousgenesis
AT vfreps serummarkersofneuroinflammationandoxidativestressinmodelingspinalinjuryofvariousgenesis