Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkers

As a multidimensional metabolic disorder, the disability and death rate of type 2 diabetes mellitus (T2DM) has increased over time. T2DM covers a wide range of pathological manifestations ranging from hyperglycemia to multi-organ failure, and it has the potential to evolve into acute complications,...

Full description

Saved in:
Bibliographic Details
Main Authors: Chuanxin Liu, Hetao Chen, Yujin Ma, Lei Zhang, Lulu Chen, Jiarui Huang, Zizhe Zhao, Hongwei Jiang, Jiao Kong
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-02-01
Series:Frontiers in Endocrinology
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fendo.2025.1501305/full
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850189429974499328
author Chuanxin Liu
Hetao Chen
Hetao Chen
Yujin Ma
Lei Zhang
Lulu Chen
Lulu Chen
Jiarui Huang
Zizhe Zhao
Hongwei Jiang
Jiao Kong
author_facet Chuanxin Liu
Hetao Chen
Hetao Chen
Yujin Ma
Lei Zhang
Lulu Chen
Lulu Chen
Jiarui Huang
Zizhe Zhao
Hongwei Jiang
Jiao Kong
author_sort Chuanxin Liu
collection DOAJ
description As a multidimensional metabolic disorder, the disability and death rate of type 2 diabetes mellitus (T2DM) has increased over time. T2DM covers a wide range of pathological manifestations ranging from hyperglycemia to multi-organ failure, and it has the potential to evolve into acute complications, including ketosis and chronic complications such as peripheral neuropathy, retinopathy, and nephropathy. T2DM mainly occurs in microvascular and large vessels and thus it is restricted for the clinician to diagnose and prescribe. However, the pathological mechanism and clinical diagnosis are inadequate. High-throughput metabolomics, characterized by non-invasive diagnostic techniques to identify potential biomarkers and distinct stages of T2DM, has been increasingly recognized as a vigorous tool with latent capacity for clinical translation. The pathological stratification of T2DM can significantly reduce disability and mortality rates. By tracing the metabolome and associated pathways from impaired fasting blood glucose or impaired glucose tolerance to severe organ failure, the chief contributions of large, independent population-based cohorts are summarized herein. These results facilitate understanding the pathophysiology and mechanism and supports research in accurate diagnosis, risk prediction, curative effect, distinct stages, and prognosis judgment of T2DM.
format Article
id doaj-art-dc3b744fc73148dfa35b97e35c870119
institution OA Journals
issn 1664-2392
language English
publishDate 2025-02-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Endocrinology
spelling doaj-art-dc3b744fc73148dfa35b97e35c8701192025-08-20T02:15:37ZengFrontiers Media S.A.Frontiers in Endocrinology1664-23922025-02-011610.3389/fendo.2025.15013051501305Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkersChuanxin Liu0Hetao Chen1Hetao Chen2Yujin Ma3Lei Zhang4Lulu Chen5Lulu Chen6Jiarui Huang7Zizhe Zhao8Hongwei Jiang9Jiao Kong10Luoyang Key Laboratory of Clinical Multiomics and Translational Medicine, Henan Key Laboratory of Rare Diseases, Endocrinology and Metabolism Center, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaLuoyang Key Laboratory of Clinical Multiomics and Translational Medicine, Henan Key Laboratory of Rare Diseases, Endocrinology and Metabolism Center, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaDepartment of Clinical Laboratory, The First Affiliated Hospital, College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaLuoyang Key Laboratory of Clinical Multiomics and Translational Medicine, Henan Key Laboratory of Rare Diseases, Endocrinology and Metabolism Center, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaDepartment of Integrative Medicine, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaLuoyang Key Laboratory of Clinical Multiomics and Translational Medicine, Henan Key Laboratory of Rare Diseases, Endocrinology and Metabolism Center, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaDepartment of Clinical Laboratory, The First Affiliated Hospital, College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaDepartment of Critical Care Medicine, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaLuoyang Key Laboratory of Clinical Multiomics and Translational Medicine, Henan Key Laboratory of Rare Diseases, Endocrinology and Metabolism Center, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaLuoyang Key Laboratory of Clinical Multiomics and Translational Medicine, Henan Key Laboratory of Rare Diseases, Endocrinology and Metabolism Center, The First Affiliated Hospital, and College of Clinical Medicine of Henan University of Science and Technology, Luoyang, ChinaInstitute of Drug Metabolism and Pharmaceutical Analysis, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou, ChinaAs a multidimensional metabolic disorder, the disability and death rate of type 2 diabetes mellitus (T2DM) has increased over time. T2DM covers a wide range of pathological manifestations ranging from hyperglycemia to multi-organ failure, and it has the potential to evolve into acute complications, including ketosis and chronic complications such as peripheral neuropathy, retinopathy, and nephropathy. T2DM mainly occurs in microvascular and large vessels and thus it is restricted for the clinician to diagnose and prescribe. However, the pathological mechanism and clinical diagnosis are inadequate. High-throughput metabolomics, characterized by non-invasive diagnostic techniques to identify potential biomarkers and distinct stages of T2DM, has been increasingly recognized as a vigorous tool with latent capacity for clinical translation. The pathological stratification of T2DM can significantly reduce disability and mortality rates. By tracing the metabolome and associated pathways from impaired fasting blood glucose or impaired glucose tolerance to severe organ failure, the chief contributions of large, independent population-based cohorts are summarized herein. These results facilitate understanding the pathophysiology and mechanism and supports research in accurate diagnosis, risk prediction, curative effect, distinct stages, and prognosis judgment of T2DM.https://www.frontiersin.org/articles/10.3389/fendo.2025.1501305/fullclinical metabolomicstype 2 diabetes mellitus (T2DM)complicationsbiomarkersstratification
spellingShingle Chuanxin Liu
Hetao Chen
Hetao Chen
Yujin Ma
Lei Zhang
Lulu Chen
Lulu Chen
Jiarui Huang
Zizhe Zhao
Hongwei Jiang
Jiao Kong
Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkers
Frontiers in Endocrinology
clinical metabolomics
type 2 diabetes mellitus (T2DM)
complications
biomarkers
stratification
title Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkers
title_full Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkers
title_fullStr Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkers
title_full_unstemmed Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkers
title_short Clinical metabolomics in type 2 diabetes mellitus: from pathogenesis to biomarkers
title_sort clinical metabolomics in type 2 diabetes mellitus from pathogenesis to biomarkers
topic clinical metabolomics
type 2 diabetes mellitus (T2DM)
complications
biomarkers
stratification
url https://www.frontiersin.org/articles/10.3389/fendo.2025.1501305/full
work_keys_str_mv AT chuanxinliu clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT hetaochen clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT hetaochen clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT yujinma clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT leizhang clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT luluchen clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT luluchen clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT jiaruihuang clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT zizhezhao clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT hongweijiang clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers
AT jiaokong clinicalmetabolomicsintype2diabetesmellitusfrompathogenesistobiomarkers