PFDN6 contributes to colorectal cancer progression via transcriptional regulation

Objective Colorectal cancer (CRC) is a common cancer worldwide. Although there are several treatments for cancer, the therapeutic effect on CRC remains unsatisfactory, and it is imperative to identify new therapeutic targets.Design Prefoldin (PFDN) is mainly used in the cytoskeleton assembly during...

Full description

Saved in:
Bibliographic Details
Main Authors: Ning Li, Jing Han, Lan Jiang, Lei Liu, Xiao Sun, Nan Zheng, Fenghua Xu, LingYang Kong, WenXiang Hui, Wenxin Han, ZhiFeng Xiao, DongFeng Chen
Format: Article
Language:English
Published: BMJ Publishing Group 2024-05-01
Series:eGastroenterology
Online Access:https://egastroenterology.bmj.com/content/2/2/e100001.full
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850105515810488320
author Ning Li
Jing Han
Lan Jiang
Lei Liu
Xiao Sun
Nan Zheng
Fenghua Xu
LingYang Kong
WenXiang Hui
Wenxin Han
ZhiFeng Xiao
DongFeng Chen
author_facet Ning Li
Jing Han
Lan Jiang
Lei Liu
Xiao Sun
Nan Zheng
Fenghua Xu
LingYang Kong
WenXiang Hui
Wenxin Han
ZhiFeng Xiao
DongFeng Chen
author_sort Ning Li
collection DOAJ
description Objective Colorectal cancer (CRC) is a common cancer worldwide. Although there are several treatments for cancer, the therapeutic effect on CRC remains unsatisfactory, and it is imperative to identify new therapeutic targets.Design Prefoldin (PFDN) is mainly used in the cytoskeleton assembly during the folding of actin and tubulin monomers. However, whether PFDN subunits are involved in regulating the development of CRC remains to be elucidated. In this study, molecular biology, cell culture, transcriptome sequencing and other experimental techniques, combined with bioinformatics, were used to verify the regulatory effects of PFDN6 on CRC.Results PFDN6 expression is elevated in patients with CRC and is closely associated with the development of CRC. Knockdown of PFDN6 reduced the tumour cell number, promoted apoptosis, and inhibited the migration and invasion of CRC cells in HCT-116 and RKO cell lines. Mechanistically, differentially expressed genes and related signalling pathways in RKO cells after PFDN6 knockdown were analysed by transcriptome sequencing.Conclusion PFDN6 was found to regulate the generation and development of CRC by targeting ZNF575. These results open new avenues for therapeutic interventions for patients with CRC.
format Article
id doaj-art-dc28f66ce3ae41f1887fb2016da2a6b3
institution OA Journals
issn 2766-0125
2976-7296
language English
publishDate 2024-05-01
publisher BMJ Publishing Group
record_format Article
series eGastroenterology
spelling doaj-art-dc28f66ce3ae41f1887fb2016da2a6b32025-08-20T02:39:03ZengBMJ Publishing GroupeGastroenterology2766-01252976-72962024-05-012210.1136/egastro-2023-100001PFDN6 contributes to colorectal cancer progression via transcriptional regulationNing Li0Jing Han1Lan Jiang2Lei Liu3Xiao Sun4Nan Zheng5Fenghua Xu6LingYang Kong7WenXiang Hui8Wenxin Han9ZhiFeng Xiao10DongFeng Chen11Department of Respiratory and Critical Care Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China4 Department of Pathology, Zhongshan Hospital Fudan University, Shanghai, P. R. China1 Key Laboratory of Modem Teaching Technology Ministry of Education, Shaanxi Normal University, Xi`an, Shaanxi, China2 Department of Gastroenterology, Third Military Medical University, Chongqing, China1 Key Laboratory of Modem Teaching Technology Ministry of Education, Shaanxi Normal University, Xi`an, Shaanxi, China3 Department of Pharmacy, Second Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China1 Key Laboratory of Modem Teaching Technology Ministry of Education, Shaanxi Normal University, Xi`an, Shaanxi, China1 Key Laboratory of Modem Teaching Technology Ministry of Education, Shaanxi Normal University, Xi`an, Shaanxi, China1 Key Laboratory of Modem Teaching Technology Ministry of Education, Shaanxi Normal University, Xi`an, Shaanxi, China1 Key Laboratory of Modem Teaching Technology Ministry of Education, Shaanxi Normal University, Xi`an, Shaanxi, China2 Department of Gastroenterology, Third Military Medical University, Chongqing, China2 Department of Gastroenterology, Third Military Medical University, Chongqing, ChinaObjective Colorectal cancer (CRC) is a common cancer worldwide. Although there are several treatments for cancer, the therapeutic effect on CRC remains unsatisfactory, and it is imperative to identify new therapeutic targets.Design Prefoldin (PFDN) is mainly used in the cytoskeleton assembly during the folding of actin and tubulin monomers. However, whether PFDN subunits are involved in regulating the development of CRC remains to be elucidated. In this study, molecular biology, cell culture, transcriptome sequencing and other experimental techniques, combined with bioinformatics, were used to verify the regulatory effects of PFDN6 on CRC.Results PFDN6 expression is elevated in patients with CRC and is closely associated with the development of CRC. Knockdown of PFDN6 reduced the tumour cell number, promoted apoptosis, and inhibited the migration and invasion of CRC cells in HCT-116 and RKO cell lines. Mechanistically, differentially expressed genes and related signalling pathways in RKO cells after PFDN6 knockdown were analysed by transcriptome sequencing.Conclusion PFDN6 was found to regulate the generation and development of CRC by targeting ZNF575. These results open new avenues for therapeutic interventions for patients with CRC.https://egastroenterology.bmj.com/content/2/2/e100001.full
spellingShingle Ning Li
Jing Han
Lan Jiang
Lei Liu
Xiao Sun
Nan Zheng
Fenghua Xu
LingYang Kong
WenXiang Hui
Wenxin Han
ZhiFeng Xiao
DongFeng Chen
PFDN6 contributes to colorectal cancer progression via transcriptional regulation
eGastroenterology
title PFDN6 contributes to colorectal cancer progression via transcriptional regulation
title_full PFDN6 contributes to colorectal cancer progression via transcriptional regulation
title_fullStr PFDN6 contributes to colorectal cancer progression via transcriptional regulation
title_full_unstemmed PFDN6 contributes to colorectal cancer progression via transcriptional regulation
title_short PFDN6 contributes to colorectal cancer progression via transcriptional regulation
title_sort pfdn6 contributes to colorectal cancer progression via transcriptional regulation
url https://egastroenterology.bmj.com/content/2/2/e100001.full
work_keys_str_mv AT ningli pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT jinghan pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT lanjiang pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT leiliu pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT xiaosun pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT nanzheng pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT fenghuaxu pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT lingyangkong pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT wenxianghui pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT wenxinhan pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT zhifengxiao pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation
AT dongfengchen pfdn6contributestocolorectalcancerprogressionviatranscriptionalregulation