Inflammation and Oxidative Stress in Obesity-Related Glomerulopathy

Obesity-related glomerulopathy is an increasing cause of end-stage renal disease. Obesity has been considered a state of chronic low-grade systemic inflammation and chronic oxidative stress. Augmented inflammation in adipose and kidney tissues promotes the progression of kidney damage in obesity. Ad...

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Main Authors: Jinhua Tang, Haidong Yan, Shougang Zhuang
Format: Article
Language:English
Published: Wiley 2012-01-01
Series:International Journal of Nephrology
Online Access:http://dx.doi.org/10.1155/2012/608397
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author Jinhua Tang
Haidong Yan
Shougang Zhuang
author_facet Jinhua Tang
Haidong Yan
Shougang Zhuang
author_sort Jinhua Tang
collection DOAJ
description Obesity-related glomerulopathy is an increasing cause of end-stage renal disease. Obesity has been considered a state of chronic low-grade systemic inflammation and chronic oxidative stress. Augmented inflammation in adipose and kidney tissues promotes the progression of kidney damage in obesity. Adipose tissue, which is accumulated in obesity, is a key endocrine organ that produces multiple biologically active molecules, including leptin, adiponectin, resistin, that affect inflammation, and subsequent deregulation of cell function in renal glomeruli that leads to pathological changes. Oxidative stress is also associated with obesity-related renal diseases and may trigger the initiation or progression of renal damage in obesity. In this paper, we focus on inflammation and oxidative stress in the progression of obesity-related glomerulopathy and possible interventions to prevent kidney injury in obesity.
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spelling doaj-art-dbef0c6251fd45338500de3457a0a2e72025-08-20T03:24:20ZengWileyInternational Journal of Nephrology2090-214X2090-21582012-01-01201210.1155/2012/608397608397Inflammation and Oxidative Stress in Obesity-Related GlomerulopathyJinhua Tang0Haidong Yan1Shougang Zhuang2Department of Nephrology, Shanghai East Hospital, Tongji University School of Medicine, Shanghai 200120, ChinaDepartment of Nephrology, Shanghai East Hospital, Tongji University School of Medicine, Shanghai 200120, ChinaDepartment of Nephrology, Shanghai East Hospital, Tongji University School of Medicine, Shanghai 200120, ChinaObesity-related glomerulopathy is an increasing cause of end-stage renal disease. Obesity has been considered a state of chronic low-grade systemic inflammation and chronic oxidative stress. Augmented inflammation in adipose and kidney tissues promotes the progression of kidney damage in obesity. Adipose tissue, which is accumulated in obesity, is a key endocrine organ that produces multiple biologically active molecules, including leptin, adiponectin, resistin, that affect inflammation, and subsequent deregulation of cell function in renal glomeruli that leads to pathological changes. Oxidative stress is also associated with obesity-related renal diseases and may trigger the initiation or progression of renal damage in obesity. In this paper, we focus on inflammation and oxidative stress in the progression of obesity-related glomerulopathy and possible interventions to prevent kidney injury in obesity.http://dx.doi.org/10.1155/2012/608397
spellingShingle Jinhua Tang
Haidong Yan
Shougang Zhuang
Inflammation and Oxidative Stress in Obesity-Related Glomerulopathy
International Journal of Nephrology
title Inflammation and Oxidative Stress in Obesity-Related Glomerulopathy
title_full Inflammation and Oxidative Stress in Obesity-Related Glomerulopathy
title_fullStr Inflammation and Oxidative Stress in Obesity-Related Glomerulopathy
title_full_unstemmed Inflammation and Oxidative Stress in Obesity-Related Glomerulopathy
title_short Inflammation and Oxidative Stress in Obesity-Related Glomerulopathy
title_sort inflammation and oxidative stress in obesity related glomerulopathy
url http://dx.doi.org/10.1155/2012/608397
work_keys_str_mv AT jinhuatang inflammationandoxidativestressinobesityrelatedglomerulopathy
AT haidongyan inflammationandoxidativestressinobesityrelatedglomerulopathy
AT shougangzhuang inflammationandoxidativestressinobesityrelatedglomerulopathy