Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modifications

Cervical cancer (CC), one of the most prevalent and detrimental gynaecologic cancers, evolves through genetic and epigenetic alterations resulting in the promotion of oncogenic activity and dysfunction of tumour-suppressing mechanisms. Despite medical advancement, the prognosis for advanced-stage pa...

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Main Authors: Akshat D. Modi, Hira Zahid, Ashlyn Chase Southerland, Dharmeshkumar M. Modi
Format: Article
Language:English
Published: Cambridge University Press 2024-01-01
Series:Expert Reviews in Molecular Medicine
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Online Access:https://www.cambridge.org/core/product/identifier/S1462399424000206/type/journal_article
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author Akshat D. Modi
Hira Zahid
Ashlyn Chase Southerland
Dharmeshkumar M. Modi
author_facet Akshat D. Modi
Hira Zahid
Ashlyn Chase Southerland
Dharmeshkumar M. Modi
author_sort Akshat D. Modi
collection DOAJ
description Cervical cancer (CC), one of the most prevalent and detrimental gynaecologic cancers, evolves through genetic and epigenetic alterations resulting in the promotion of oncogenic activity and dysfunction of tumour-suppressing mechanisms. Despite medical advancement, the prognosis for advanced-stage patients remains extremely low due to high recurrence rates and resistance to existing treatments. Thereby, the search for potential prognostic biomarkers is heightened to unravel new modalities of CC pathogenesis and to develop novel anti-cancer therapies. Epitranscriptomic modifications, reversible epigenetic RNA modifications, regulate various biological processes by deciding RNA fate to mediating RNA interactions. This narrative review provides insight into the cellular and molecular roles of endogenous RNA-editing proteins and their associated epitranscriptomic modifications, especially N6-methyladenosine (m6A), 5-methylcytosine (m5C) and N1-methyladenosine (m1A), in governing the development, progression and metastasis of CC. We discussed the in-depth epitranscriptomic mechanisms underlying the regulation of over 50 RNAs responsible for tumorigenesis, proliferation, migration, invasion, survival, autophagy, stemness, epithelial-mesenchymal transition, metabolism (glucose, lipid, glutamate and glutamine), resistance (drug and radiation), angiogenesis and recurrence of CC. Additionally, we provided a concise overview of the therapeutic potential of targeting the altered expression of endogenous RNA-editing proteins and aberrant deposition of RNA modifications on both coding and non-coding RNAs in CC.
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spelling doaj-art-dbeaf258a3a841aeb89e11db3673eae22025-08-20T12:57:31ZengCambridge University PressExpert Reviews in Molecular Medicine1462-39942024-01-012610.1017/erm.2024.20Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modificationsAkshat D. Modi0https://orcid.org/0000-0003-1086-3670Hira Zahid1Ashlyn Chase Southerland2Dharmeshkumar M. Modi3https://orcid.org/0000-0002-4011-0533Department of Biological Sciences, University of Toronto, Scarborough, CanadaDepartment of Biology, University of Toronto, Mississauga, CanadaDepartment of Health Sciences, California State University, Los Angeles, USADepartment of Pharmacy, Silver Oak University, Ahmedabad, IndiaCervical cancer (CC), one of the most prevalent and detrimental gynaecologic cancers, evolves through genetic and epigenetic alterations resulting in the promotion of oncogenic activity and dysfunction of tumour-suppressing mechanisms. Despite medical advancement, the prognosis for advanced-stage patients remains extremely low due to high recurrence rates and resistance to existing treatments. Thereby, the search for potential prognostic biomarkers is heightened to unravel new modalities of CC pathogenesis and to develop novel anti-cancer therapies. Epitranscriptomic modifications, reversible epigenetic RNA modifications, regulate various biological processes by deciding RNA fate to mediating RNA interactions. This narrative review provides insight into the cellular and molecular roles of endogenous RNA-editing proteins and their associated epitranscriptomic modifications, especially N6-methyladenosine (m6A), 5-methylcytosine (m5C) and N1-methyladenosine (m1A), in governing the development, progression and metastasis of CC. We discussed the in-depth epitranscriptomic mechanisms underlying the regulation of over 50 RNAs responsible for tumorigenesis, proliferation, migration, invasion, survival, autophagy, stemness, epithelial-mesenchymal transition, metabolism (glucose, lipid, glutamate and glutamine), resistance (drug and radiation), angiogenesis and recurrence of CC. Additionally, we provided a concise overview of the therapeutic potential of targeting the altered expression of endogenous RNA-editing proteins and aberrant deposition of RNA modifications on both coding and non-coding RNAs in CC.https://www.cambridge.org/core/product/identifier/S1462399424000206/type/journal_articleanti-cancer therapiescervical cancerdiagnostic biomarkersepitranscriptomicsgene expressionm1Am5Cm6ARNA modificationsRNA-editing proteins
spellingShingle Akshat D. Modi
Hira Zahid
Ashlyn Chase Southerland
Dharmeshkumar M. Modi
Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modifications
Expert Reviews in Molecular Medicine
anti-cancer therapies
cervical cancer
diagnostic biomarkers
epitranscriptomics
gene expression
m1A
m5C
m6A
RNA modifications
RNA-editing proteins
title Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modifications
title_full Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modifications
title_fullStr Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modifications
title_full_unstemmed Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modifications
title_short Epitranscriptomics and cervical cancer: the emerging role of m6A, m5C and m1A RNA modifications
title_sort epitranscriptomics and cervical cancer the emerging role of m6a m5c and m1a rna modifications
topic anti-cancer therapies
cervical cancer
diagnostic biomarkers
epitranscriptomics
gene expression
m1A
m5C
m6A
RNA modifications
RNA-editing proteins
url https://www.cambridge.org/core/product/identifier/S1462399424000206/type/journal_article
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