Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis

Peroxisome proliferator-activated receptor-γ (PPARγ) is a ligand-activated transcription factor belonging to the nuclear receptor superfamily, which plays a central role in regulating lipid and glucose metabolism. However, accumulating evidence demonstrates that PPARγ agonists have potential to redu...

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Main Authors: Huang-Jun Liu, Hai-Han Liao, Zheng Yang, Qi-Zhu Tang
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:PPAR Research
Online Access:http://dx.doi.org/10.1155/2016/2198645
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author Huang-Jun Liu
Hai-Han Liao
Zheng Yang
Qi-Zhu Tang
author_facet Huang-Jun Liu
Hai-Han Liao
Zheng Yang
Qi-Zhu Tang
author_sort Huang-Jun Liu
collection DOAJ
description Peroxisome proliferator-activated receptor-γ (PPARγ) is a ligand-activated transcription factor belonging to the nuclear receptor superfamily, which plays a central role in regulating lipid and glucose metabolism. However, accumulating evidence demonstrates that PPARγ agonists have potential to reduce inflammation, influence the balance of immune cells, suppress oxidative stress, and improve endothelial function, which are all involved in the cellular and molecular mechanisms of cardiac fibrosis. Thus, in this review we discuss the role of PPARγ in various cardiovascular conditions associated with cardiac fibrosis, including diabetes mellitus, hypertension, myocardial infarction, heart failure, ischemia/reperfusion injury, atrial fibrillation, and several other cardiovascular disease (CVD) conditions, and summarize the developmental status of PPARγ agonists for the clinical management of CVD.
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institution Kabale University
issn 1687-4757
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publishDate 2016-01-01
publisher Wiley
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series PPAR Research
spelling doaj-art-db9219d18ea642acb40ea3f2592847de2025-08-20T03:24:21ZengWileyPPAR Research1687-47571687-47652016-01-01201610.1155/2016/21986452198645Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac FibrosisHuang-Jun Liu0Hai-Han Liao1Zheng Yang2Qi-Zhu Tang3Department of Cardiology, Renmin Hospital of Wuhan University, Wuhan 430060, ChinaDepartment of Cardiology, Renmin Hospital of Wuhan University, Wuhan 430060, ChinaDepartment of Cardiology, Renmin Hospital of Wuhan University, Wuhan 430060, ChinaDepartment of Cardiology, Renmin Hospital of Wuhan University, Wuhan 430060, ChinaPeroxisome proliferator-activated receptor-γ (PPARγ) is a ligand-activated transcription factor belonging to the nuclear receptor superfamily, which plays a central role in regulating lipid and glucose metabolism. However, accumulating evidence demonstrates that PPARγ agonists have potential to reduce inflammation, influence the balance of immune cells, suppress oxidative stress, and improve endothelial function, which are all involved in the cellular and molecular mechanisms of cardiac fibrosis. Thus, in this review we discuss the role of PPARγ in various cardiovascular conditions associated with cardiac fibrosis, including diabetes mellitus, hypertension, myocardial infarction, heart failure, ischemia/reperfusion injury, atrial fibrillation, and several other cardiovascular disease (CVD) conditions, and summarize the developmental status of PPARγ agonists for the clinical management of CVD.http://dx.doi.org/10.1155/2016/2198645
spellingShingle Huang-Jun Liu
Hai-Han Liao
Zheng Yang
Qi-Zhu Tang
Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis
PPAR Research
title Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis
title_full Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis
title_fullStr Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis
title_full_unstemmed Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis
title_short Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis
title_sort peroxisome proliferator activated receptor γ is critical to cardiac fibrosis
url http://dx.doi.org/10.1155/2016/2198645
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AT haihanliao peroxisomeproliferatoractivatedreceptorgiscriticaltocardiacfibrosis
AT zhengyang peroxisomeproliferatoractivatedreceptorgiscriticaltocardiacfibrosis
AT qizhutang peroxisomeproliferatoractivatedreceptorgiscriticaltocardiacfibrosis