Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer

Abstract Ovarian cancer (OC) is a highly fatal gynecological malignancy primarily attributable to late-stage detection and restricted treatment options. Aberrant glycolysis, exemplified by the Warburg effect, facilitates tumor development, immunological evasion, and alteration of the microenvironmen...

Full description

Saved in:
Bibliographic Details
Main Authors: Mingwei Wang, Qiaohui Ying, Yuncan Xing, Shuchang Dai, Jue Wang, Zhong Liu
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
Subjects:
Online Access:https://doi.org/10.1038/s41598-025-12350-7
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849235411703955456
author Mingwei Wang
Qiaohui Ying
Yuncan Xing
Shuchang Dai
Jue Wang
Zhong Liu
author_facet Mingwei Wang
Qiaohui Ying
Yuncan Xing
Shuchang Dai
Jue Wang
Zhong Liu
author_sort Mingwei Wang
collection DOAJ
description Abstract Ovarian cancer (OC) is a highly fatal gynecological malignancy primarily attributable to late-stage detection and restricted treatment options. Aberrant glycolysis, exemplified by the Warburg effect, facilitates tumor development, immunological evasion, and alteration of the microenvironment. Identifying glycolysis-related biomarkers could provide novel insights into prognosis and potential therapeutic targets for OC.The transcriptomic and clinical information of OC patients were obtained from The Cancer Genome Atlas (TCGA), Genotype-Tissue Expression (GTEx), and Gene Expression Omnibus (GEO) databases. Differentially expressed glycolysis-related genes (GRGs) were identified and analyzed for their prognostic significance. Consensus clustering was employed to identify glycolysis subtypes, followed by pathway enrichment and immune infiltration analyses. A ten-gene GRG signature was developed with LASSO-Cox regression and verified in various cohorts. Single-cell RNA sequence and drug susceptibility analysis were performed to explore tumor microenvironment heterogeneity and potential therapeutic agents.A total of 457 differentially expressed GRGs were discovered, of which 30 were substantially linked with OC prognosis. Three molecular subtypes were characterized, with cluster C exhibiting the worst prognosis and activation of tumor-associated pathways. A prognostic model comprising ten genes (LMCD1, L1CAM, MYCN, GALT, IDO1, RPL18, XBP1, LPAR3, RUNX3, PLCG1) was developed and validated, demonstrating robust predictive efficacy across various cohorts. Immune analysis revealed substantial disparities in immune infiltration among risk groups, whereas single-cell analysis identified several critical genes essential for metabolism, proliferation, and interactions within the tumor microenvironment.This work highlights the prognostic and therapeutic significance of GRGs in OC. The ten-gene GRG signature serves as a reliable framework for risk assessment and the formulation of individualized treatment regimens. Nonetheless, further experimental validation and extensive clinical research are necessary to enable the application of these findings in clinical practice. These results highlight the potential of targeting glycolytic pathways as a promising approach to improve the management and treatment outcomes of OC.
format Article
id doaj-art-db12be7f63db4e5c8aa0008bee287576
institution Kabale University
issn 2045-2322
language English
publishDate 2025-07-01
publisher Nature Portfolio
record_format Article
series Scientific Reports
spelling doaj-art-db12be7f63db4e5c8aa0008bee2875762025-08-20T04:02:46ZengNature PortfolioScientific Reports2045-23222025-07-0115111810.1038/s41598-025-12350-7Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancerMingwei Wang0Qiaohui Ying1Yuncan Xing2Shuchang Dai3Jue Wang4Zhong Liu5Institute of Blood Transfusion, Chinese Academy of Medical Sciences and Peking Union Medical CollegeInstitute of Oral Basic Research, School and Hospital of Stomatology, Cheeloo College of Medicine, Shandong UniversityNational Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical CollegeInstitute of Blood Transfusion, Chinese Academy of Medical Sciences and Peking Union Medical CollegeInstitute of Blood Transfusion, Chinese Academy of Medical Sciences and Peking Union Medical CollegeInstitute of Blood Transfusion, Chinese Academy of Medical Sciences and Peking Union Medical CollegeAbstract Ovarian cancer (OC) is a highly fatal gynecological malignancy primarily attributable to late-stage detection and restricted treatment options. Aberrant glycolysis, exemplified by the Warburg effect, facilitates tumor development, immunological evasion, and alteration of the microenvironment. Identifying glycolysis-related biomarkers could provide novel insights into prognosis and potential therapeutic targets for OC.The transcriptomic and clinical information of OC patients were obtained from The Cancer Genome Atlas (TCGA), Genotype-Tissue Expression (GTEx), and Gene Expression Omnibus (GEO) databases. Differentially expressed glycolysis-related genes (GRGs) were identified and analyzed for their prognostic significance. Consensus clustering was employed to identify glycolysis subtypes, followed by pathway enrichment and immune infiltration analyses. A ten-gene GRG signature was developed with LASSO-Cox regression and verified in various cohorts. Single-cell RNA sequence and drug susceptibility analysis were performed to explore tumor microenvironment heterogeneity and potential therapeutic agents.A total of 457 differentially expressed GRGs were discovered, of which 30 were substantially linked with OC prognosis. Three molecular subtypes were characterized, with cluster C exhibiting the worst prognosis and activation of tumor-associated pathways. A prognostic model comprising ten genes (LMCD1, L1CAM, MYCN, GALT, IDO1, RPL18, XBP1, LPAR3, RUNX3, PLCG1) was developed and validated, demonstrating robust predictive efficacy across various cohorts. Immune analysis revealed substantial disparities in immune infiltration among risk groups, whereas single-cell analysis identified several critical genes essential for metabolism, proliferation, and interactions within the tumor microenvironment.This work highlights the prognostic and therapeutic significance of GRGs in OC. The ten-gene GRG signature serves as a reliable framework for risk assessment and the formulation of individualized treatment regimens. Nonetheless, further experimental validation and extensive clinical research are necessary to enable the application of these findings in clinical practice. These results highlight the potential of targeting glycolytic pathways as a promising approach to improve the management and treatment outcomes of OC.https://doi.org/10.1038/s41598-025-12350-7Ovarian cancerGlycolysis-related genesPrognostic modelTumor microenvironmentWarburg effect
spellingShingle Mingwei Wang
Qiaohui Ying
Yuncan Xing
Shuchang Dai
Jue Wang
Zhong Liu
Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer
Scientific Reports
Ovarian cancer
Glycolysis-related genes
Prognostic model
Tumor microenvironment
Warburg effect
title Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer
title_full Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer
title_fullStr Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer
title_full_unstemmed Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer
title_short Metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer
title_sort metabolic reprogramming and prognostic insights in molecular landscapes driven by glycolysis in ovarian cancer
topic Ovarian cancer
Glycolysis-related genes
Prognostic model
Tumor microenvironment
Warburg effect
url https://doi.org/10.1038/s41598-025-12350-7
work_keys_str_mv AT mingweiwang metabolicreprogrammingandprognosticinsightsinmolecularlandscapesdrivenbyglycolysisinovariancancer
AT qiaohuiying metabolicreprogrammingandprognosticinsightsinmolecularlandscapesdrivenbyglycolysisinovariancancer
AT yuncanxing metabolicreprogrammingandprognosticinsightsinmolecularlandscapesdrivenbyglycolysisinovariancancer
AT shuchangdai metabolicreprogrammingandprognosticinsightsinmolecularlandscapesdrivenbyglycolysisinovariancancer
AT juewang metabolicreprogrammingandprognosticinsightsinmolecularlandscapesdrivenbyglycolysisinovariancancer
AT zhongliu metabolicreprogrammingandprognosticinsightsinmolecularlandscapesdrivenbyglycolysisinovariancancer