Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic Pain

Chronic pain affects ~20% of the worldwide population. The clinical management of chronic pain is mostly palliative and results in limited success. Current treatments mostly target the symptoms or neuronal signaling of chronic pain. It has been increasingly recognized that glial cells, such as micro...

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Main Authors: Temugin Berta, Jee Eun Lee, Chul-Kyu Park
Format: Article
Language:English
Published: Wiley 2017-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2017/9383184
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author Temugin Berta
Jee Eun Lee
Chul-Kyu Park
author_facet Temugin Berta
Jee Eun Lee
Chul-Kyu Park
author_sort Temugin Berta
collection DOAJ
description Chronic pain affects ~20% of the worldwide population. The clinical management of chronic pain is mostly palliative and results in limited success. Current treatments mostly target the symptoms or neuronal signaling of chronic pain. It has been increasingly recognized that glial cells, such as microglia, and inflammatory signaling play a major role in the pathogenesis of chronic pain. Caspases (CASPs) are a family of protease enzymes involved in apoptosis and inflammation. They are pivotal components in a variety of neurological diseases. However, little is known about the role of CASPs in microglial modulation as to chronic pain. In particular, our recent studies have shown that CASP6 regulates chronic pain via microglial inflammatory signaling. Inhibition of microglia and CASP signaling might provide a new strategy for the prevention and treatment of chronic pain.
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series Mediators of Inflammation
spelling doaj-art-d3ebc9d19722470c8a08a6d92e59df7b2025-08-20T03:24:14ZengWileyMediators of Inflammation0962-93511466-18612017-01-01201710.1155/2017/93831849383184Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic PainTemugin Berta0Jee Eun Lee1Chul-Kyu Park2Pain Research Center, Department of Anesthesiology, University of Cincinnati Medical Center, Cincinnati, OH, USADepartment of Physiology, College of Medicine, Gachon University, Incheon 21999, Republic of KoreaDepartment of Physiology, College of Medicine, Gachon University, Incheon 21999, Republic of KoreaChronic pain affects ~20% of the worldwide population. The clinical management of chronic pain is mostly palliative and results in limited success. Current treatments mostly target the symptoms or neuronal signaling of chronic pain. It has been increasingly recognized that glial cells, such as microglia, and inflammatory signaling play a major role in the pathogenesis of chronic pain. Caspases (CASPs) are a family of protease enzymes involved in apoptosis and inflammation. They are pivotal components in a variety of neurological diseases. However, little is known about the role of CASPs in microglial modulation as to chronic pain. In particular, our recent studies have shown that CASP6 regulates chronic pain via microglial inflammatory signaling. Inhibition of microglia and CASP signaling might provide a new strategy for the prevention and treatment of chronic pain.http://dx.doi.org/10.1155/2017/9383184
spellingShingle Temugin Berta
Jee Eun Lee
Chul-Kyu Park
Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic Pain
Mediators of Inflammation
title Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic Pain
title_full Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic Pain
title_fullStr Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic Pain
title_full_unstemmed Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic Pain
title_short Unconventional Role of Caspase-6 in Spinal Microglia Activation and Chronic Pain
title_sort unconventional role of caspase 6 in spinal microglia activation and chronic pain
url http://dx.doi.org/10.1155/2017/9383184
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