Fighting vessel dysmorphia to improve glioma chemotherapy

Abstract High‐grade gliomas are aggressive and abundantly vascular tumors, and as in most cancer types, blood vessels in advanced lesions are highly abnormal. Poor perfusion and vascular leakage in tumor tissue resulting in hypoxia, necrosis, and high interstitial fluid pressure can hamper the effic...

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Main Authors: Marja Lohela, Kari Alitalo
Format: Article
Language:English
Published: Springer Nature 2017-11-01
Series:EMBO Molecular Medicine
Online Access:https://doi.org/10.15252/emmm.201708431
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author Marja Lohela
Kari Alitalo
author_facet Marja Lohela
Kari Alitalo
author_sort Marja Lohela
collection DOAJ
description Abstract High‐grade gliomas are aggressive and abundantly vascular tumors, and as in most cancer types, blood vessels in advanced lesions are highly abnormal. Poor perfusion and vascular leakage in tumor tissue resulting in hypoxia, necrosis, and high interstitial fluid pressure can hamper the efficient delivery of chemotherapy. Tumor angiogenesis is known to be supported by host leukocytes recruited to the tumor microenvironment, but the mechanisms leading to dysfunctional vascular network formation are incompletely understood. In this issue of EMBO Molecular Medicine, Mathivet et al (2017) present an elegant study, where longitudinal intravital imaging gives new insight on how recruitment and polarization of tumor‐associated macrophages regulate aberrant angiogenesis in experimental gliomas. They show that macrophage targeting results in vessel normalization and improved chemotherapy response, suggesting that the combination of these therapeutic modalities could improve the outcome of glioma treatment in the clinic.
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institution Kabale University
issn 1757-4676
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language English
publishDate 2017-11-01
publisher Springer Nature
record_format Article
series EMBO Molecular Medicine
spelling doaj-art-d3db6931b0e7484d91b8c84d79231c462025-08-20T03:46:13ZengSpringer NatureEMBO Molecular Medicine1757-46761757-46842017-11-019121626162810.15252/emmm.201708431Fighting vessel dysmorphia to improve glioma chemotherapyMarja Lohela0Kari Alitalo1Biomedicum Imaging Unit, University of HelsinkiTranslational Cancer Biology Program, University of HelsinkiAbstract High‐grade gliomas are aggressive and abundantly vascular tumors, and as in most cancer types, blood vessels in advanced lesions are highly abnormal. Poor perfusion and vascular leakage in tumor tissue resulting in hypoxia, necrosis, and high interstitial fluid pressure can hamper the efficient delivery of chemotherapy. Tumor angiogenesis is known to be supported by host leukocytes recruited to the tumor microenvironment, but the mechanisms leading to dysfunctional vascular network formation are incompletely understood. In this issue of EMBO Molecular Medicine, Mathivet et al (2017) present an elegant study, where longitudinal intravital imaging gives new insight on how recruitment and polarization of tumor‐associated macrophages regulate aberrant angiogenesis in experimental gliomas. They show that macrophage targeting results in vessel normalization and improved chemotherapy response, suggesting that the combination of these therapeutic modalities could improve the outcome of glioma treatment in the clinic.https://doi.org/10.15252/emmm.201708431
spellingShingle Marja Lohela
Kari Alitalo
Fighting vessel dysmorphia to improve glioma chemotherapy
EMBO Molecular Medicine
title Fighting vessel dysmorphia to improve glioma chemotherapy
title_full Fighting vessel dysmorphia to improve glioma chemotherapy
title_fullStr Fighting vessel dysmorphia to improve glioma chemotherapy
title_full_unstemmed Fighting vessel dysmorphia to improve glioma chemotherapy
title_short Fighting vessel dysmorphia to improve glioma chemotherapy
title_sort fighting vessel dysmorphia to improve glioma chemotherapy
url https://doi.org/10.15252/emmm.201708431
work_keys_str_mv AT marjalohela fightingvesseldysmorphiatoimprovegliomachemotherapy
AT karialitalo fightingvesseldysmorphiatoimprovegliomachemotherapy