High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes

Hepatocellular carcinoma is one of the most prevalent neoplasms and the leading cause of cancer-related mortality worldwide. Mitochondrial ribosomal protein S23 is encoded by a nuclear gene and participates in mitochondrial protein translation. Mitochondrial ribosomal protein S23 overexpression has...

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Main Authors: Meng Pu, Jianlin Wang, Qike Huang, Ge Zhao, Congcong Xia, Runze Shang, Zhuochao Zhang, Zhenyuan Bian, Xishegn Yang, Kaishan Tao
Format: Article
Language:English
Published: SAGE Publishing 2017-07-01
Series:Tumor Biology
Online Access:https://doi.org/10.1177/1010428317709127
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author Meng Pu
Jianlin Wang
Qike Huang
Ge Zhao
Congcong Xia
Runze Shang
Zhuochao Zhang
Zhenyuan Bian
Xishegn Yang
Kaishan Tao
author_facet Meng Pu
Jianlin Wang
Qike Huang
Ge Zhao
Congcong Xia
Runze Shang
Zhuochao Zhang
Zhenyuan Bian
Xishegn Yang
Kaishan Tao
author_sort Meng Pu
collection DOAJ
description Hepatocellular carcinoma is one of the most prevalent neoplasms and the leading cause of cancer-related mortality worldwide. Mitochondrial ribosomal protein S23 is encoded by a nuclear gene and participates in mitochondrial protein translation. Mitochondrial ribosomal protein S23 overexpression has been found in many types of cancer. In this study, we explored mitochondrial ribosomal protein S23 expression in primary hepatocellular carcinoma tissues compared with matched adjacent non-tumoral liver tissues using mitochondrial ribosomal protein S23 messenger RNA and protein levels collected from public databases and clinical samples. Immunohistochemistry was performed to analyze the relationship between mitochondrial ribosomal protein S23 and various clinicopathological features. The results indicated that mitochondrial ribosomal protein S23 was significantly overexpressed in hepatocellular carcinoma. High mitochondrial ribosomal protein S23 expression was correlated with the tumor size and tumor–metastasis–node stage. Moreover, patients with high mitochondrial ribosomal protein S23 expression levels presented poorer survival rates. Mitochondrial ribosomal protein S23 was an independent prognostic factor for survival, especially at the early stage of hepatocellular carcinoma. In addition, the downregulation of mitochondrial ribosomal protein S23 decreased the proliferation of hepatocellular carcinoma in vitro and in vivo. In conclusion, we verified for the first time that mitochondrial ribosomal protein S23 expression was upregulated in hepatocellular carcinoma. High mitochondrial ribosomal protein S23 levels can predict poor clinical outcomes in hepatocellular carcinoma, and this protein plays a key role in tumor proliferation. Therefore, mitochondrial ribosomal protein S23 may be a potential therapeutic target for hepatocellular carcinoma.
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spelling doaj-art-d37d6b9c9cc54d419d356e958da22c5a2025-08-20T03:18:09ZengSAGE PublishingTumor Biology1423-03802017-07-013910.1177/1010428317709127High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomesMeng Pu0Jianlin Wang1Qike Huang2Ge Zhao3Congcong Xia4Runze Shang5Zhuochao Zhang6Zhenyuan Bian7Xishegn Yang8Kaishan Tao9Department of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Thyroid, Breast and Vascular Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi’an, ChinaHepatocellular carcinoma is one of the most prevalent neoplasms and the leading cause of cancer-related mortality worldwide. Mitochondrial ribosomal protein S23 is encoded by a nuclear gene and participates in mitochondrial protein translation. Mitochondrial ribosomal protein S23 overexpression has been found in many types of cancer. In this study, we explored mitochondrial ribosomal protein S23 expression in primary hepatocellular carcinoma tissues compared with matched adjacent non-tumoral liver tissues using mitochondrial ribosomal protein S23 messenger RNA and protein levels collected from public databases and clinical samples. Immunohistochemistry was performed to analyze the relationship between mitochondrial ribosomal protein S23 and various clinicopathological features. The results indicated that mitochondrial ribosomal protein S23 was significantly overexpressed in hepatocellular carcinoma. High mitochondrial ribosomal protein S23 expression was correlated with the tumor size and tumor–metastasis–node stage. Moreover, patients with high mitochondrial ribosomal protein S23 expression levels presented poorer survival rates. Mitochondrial ribosomal protein S23 was an independent prognostic factor for survival, especially at the early stage of hepatocellular carcinoma. In addition, the downregulation of mitochondrial ribosomal protein S23 decreased the proliferation of hepatocellular carcinoma in vitro and in vivo. In conclusion, we verified for the first time that mitochondrial ribosomal protein S23 expression was upregulated in hepatocellular carcinoma. High mitochondrial ribosomal protein S23 levels can predict poor clinical outcomes in hepatocellular carcinoma, and this protein plays a key role in tumor proliferation. Therefore, mitochondrial ribosomal protein S23 may be a potential therapeutic target for hepatocellular carcinoma.https://doi.org/10.1177/1010428317709127
spellingShingle Meng Pu
Jianlin Wang
Qike Huang
Ge Zhao
Congcong Xia
Runze Shang
Zhuochao Zhang
Zhenyuan Bian
Xishegn Yang
Kaishan Tao
High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes
Tumor Biology
title High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes
title_full High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes
title_fullStr High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes
title_full_unstemmed High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes
title_short High MRPS23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes
title_sort high mrps23 expression contributes to hepatocellular carcinoma proliferation and indicates poor survival outcomes
url https://doi.org/10.1177/1010428317709127
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