Structures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine design

Abstract Respiratory syncytial virus (RSV) is a leading cause of severe lower respiratory tract disease in infants and older adults. The attachment glycoprotein (RSV G) binds to the chemokine receptor CX3CR1 to promote viral entry and modulate host immunity. Antibodies against RSV G are a known corr...

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Main Authors: Maria G. Juarez, Sara M. O’Rourke, John V. Dzimianski, Delia Gagnon, Gabriel Penunuri, Vitor H. B. Serrão, Russell B. Corbett-Detig, Lawrence M. Kauvar, Rebecca M. DuBois
Format: Article
Language:English
Published: Nature Portfolio 2025-03-01
Series:Scientific Reports
Online Access:https://doi.org/10.1038/s41598-025-92886-w
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author Maria G. Juarez
Sara M. O’Rourke
John V. Dzimianski
Delia Gagnon
Gabriel Penunuri
Vitor H. B. Serrão
Russell B. Corbett-Detig
Lawrence M. Kauvar
Rebecca M. DuBois
author_facet Maria G. Juarez
Sara M. O’Rourke
John V. Dzimianski
Delia Gagnon
Gabriel Penunuri
Vitor H. B. Serrão
Russell B. Corbett-Detig
Lawrence M. Kauvar
Rebecca M. DuBois
author_sort Maria G. Juarez
collection DOAJ
description Abstract Respiratory syncytial virus (RSV) is a leading cause of severe lower respiratory tract disease in infants and older adults. The attachment glycoprotein (RSV G) binds to the chemokine receptor CX3CR1 to promote viral entry and modulate host immunity. Antibodies against RSV G are a known correlate of protection. Previously, several broadly reactive, high-affinity anti-RSV G human monoclonal antibodies were isolated from RSV-exposed individuals and were shown to be protective in vitro and in vivo. Here, we determined the structures of three of these antibodies in complex with RSV G and defined distinct conformational epitopes comprised of highly conserved RSV G residues. Binding competition and structural studies demonstrated that this highly conserved region displays two non-overlapping antigenic sites. Analyses of anti-RSV G antibody sequences reveal that antigenic site flexibility may promote the elicitation of diverse antibody germlines. Together, these findings provide a foundation for next-generation RSV prophylactics, and they expand concepts in vaccine design for the elicitation of germline lineage-diverse, broadly reactive, high-affinity antibodies.
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spelling doaj-art-d2ceaad0afc14ee9983289f2b4084d682025-08-20T02:56:06ZengNature PortfolioScientific Reports2045-23222025-03-0115111510.1038/s41598-025-92886-wStructures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine designMaria G. Juarez0Sara M. O’Rourke1John V. Dzimianski2Delia Gagnon3Gabriel Penunuri4Vitor H. B. Serrão5Russell B. Corbett-Detig6Lawrence M. Kauvar7Rebecca M. DuBois8Department of Molecular, Cell, and Developmental Biology, University of California Santa CruzDepartment of Biomolecular Engineering, University of California Santa CruzDepartment of Biomolecular Engineering, University of California Santa CruzDepartment of Biomolecular Engineering, University of California Santa CruzDepartment of Biomolecular Engineering, University of California Santa CruzDepartment of Chemistry & Biochemistry, University of California Santa CruzDepartment of Biomolecular Engineering, University of California Santa CruzTrellis Bioscience, IncDepartment of Biomolecular Engineering, University of California Santa CruzAbstract Respiratory syncytial virus (RSV) is a leading cause of severe lower respiratory tract disease in infants and older adults. The attachment glycoprotein (RSV G) binds to the chemokine receptor CX3CR1 to promote viral entry and modulate host immunity. Antibodies against RSV G are a known correlate of protection. Previously, several broadly reactive, high-affinity anti-RSV G human monoclonal antibodies were isolated from RSV-exposed individuals and were shown to be protective in vitro and in vivo. Here, we determined the structures of three of these antibodies in complex with RSV G and defined distinct conformational epitopes comprised of highly conserved RSV G residues. Binding competition and structural studies demonstrated that this highly conserved region displays two non-overlapping antigenic sites. Analyses of anti-RSV G antibody sequences reveal that antigenic site flexibility may promote the elicitation of diverse antibody germlines. Together, these findings provide a foundation for next-generation RSV prophylactics, and they expand concepts in vaccine design for the elicitation of germline lineage-diverse, broadly reactive, high-affinity antibodies.https://doi.org/10.1038/s41598-025-92886-w
spellingShingle Maria G. Juarez
Sara M. O’Rourke
John V. Dzimianski
Delia Gagnon
Gabriel Penunuri
Vitor H. B. Serrão
Russell B. Corbett-Detig
Lawrence M. Kauvar
Rebecca M. DuBois
Structures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine design
Scientific Reports
title Structures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine design
title_full Structures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine design
title_fullStr Structures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine design
title_full_unstemmed Structures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine design
title_short Structures of respiratory syncytial virus G bound to broadly reactive antibodies provide insights into vaccine design
title_sort structures of respiratory syncytial virus g bound to broadly reactive antibodies provide insights into vaccine design
url https://doi.org/10.1038/s41598-025-92886-w
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