Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceils

The effect of hydrocortisone on the production of interleukin-6 (IL-6) in human peripheral blood mononuclear cells was studied. Using our newly developed radioimmunoassay system for IL-6 of which specificity, reproducibility, sensitivity and usefulness have been demonstrated. IL-6 production in peri...

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Main Authors: Y. Hirooka, T. Mitsuma, T. Nogimori, Y. Ishizukis
Format: Article
Language:English
Published: Wiley 1992-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/S0962935192000036
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author Y. Hirooka
T. Mitsuma
T. Nogimori
Y. Ishizukis
author_facet Y. Hirooka
T. Mitsuma
T. Nogimori
Y. Ishizukis
author_sort Y. Hirooka
collection DOAJ
description The effect of hydrocortisone on the production of interleukin-6 (IL-6) in human peripheral blood mononuclear cells was studied. Using our newly developed radioimmunoassay system for IL-6 of which specificity, reproducibility, sensitivity and usefulness have been demonstrated. IL-6 production in peripheral blood mononuclear cells of ten normal subjects revealed that in lipopolysaccharide (LPS, 10μg/ml)-stimulation, the mean ± SD of IL-6 was 2.71 ± 0.85 ng/ml. No detectable amount of IL-6 was observed in the absence of LPS and in the presence of hydrocortisone alone. Hydrocortisone (10−10 M to 10−3 M) inhibited LPS-stimulated IL-6 production in a dose-dependent manner. However, there was a wide variation in the response to hydrocortisone, namely, ranging from steroid-sensitive to steroid-resistant. Based on the concentration required to inhibit 50% of LPS-stimulated IL-6 production, three of ten subjects were at 10−6 M, three at 10−5 M and the rest at 10−4 M, respectively. The dramatic anti-inflammatory and immunosuppressive effects of glucocorticosteroids can be life-saving in autoimmune diseases. The present findings suggested that there existed the differences in susceptibility to glucocorticosteroids even among normal subjects, providing some implications for the drug treatment, and also gave further evidence that there may exist an immunoregulatory feedback circuit between the immune and neuroendocrine systems.
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publishDate 1992-01-01
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series Mediators of Inflammation
spelling doaj-art-d2426cc133c04c52a56be40d4d67afcf2025-08-20T03:24:07ZengWileyMediators of Inflammation0962-93511466-18611992-01-011191310.1155/S0962935192000036Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceilsY. Hirooka0T. Mitsuma1T. Nogimori2Y. Ishizukis3The Fourth Department of Internal Medicine, Aichi Medical University, Yazako, Nagakute-cho, Aichi-gun, Aichi-ken, 480-11, JapanThe Fourth Department of Internal Medicine, Aichi Medical University, Yazako, Nagakute-cho, Aichi-gun, Aichi-ken, 480-11, JapanDepartment of Medicine, Konanshowa Hospital, Konan, Aichi, JapanIshizuki Thyroid Clinic, Nagoya, JapanThe effect of hydrocortisone on the production of interleukin-6 (IL-6) in human peripheral blood mononuclear cells was studied. Using our newly developed radioimmunoassay system for IL-6 of which specificity, reproducibility, sensitivity and usefulness have been demonstrated. IL-6 production in peripheral blood mononuclear cells of ten normal subjects revealed that in lipopolysaccharide (LPS, 10μg/ml)-stimulation, the mean ± SD of IL-6 was 2.71 ± 0.85 ng/ml. No detectable amount of IL-6 was observed in the absence of LPS and in the presence of hydrocortisone alone. Hydrocortisone (10−10 M to 10−3 M) inhibited LPS-stimulated IL-6 production in a dose-dependent manner. However, there was a wide variation in the response to hydrocortisone, namely, ranging from steroid-sensitive to steroid-resistant. Based on the concentration required to inhibit 50% of LPS-stimulated IL-6 production, three of ten subjects were at 10−6 M, three at 10−5 M and the rest at 10−4 M, respectively. The dramatic anti-inflammatory and immunosuppressive effects of glucocorticosteroids can be life-saving in autoimmune diseases. The present findings suggested that there existed the differences in susceptibility to glucocorticosteroids even among normal subjects, providing some implications for the drug treatment, and also gave further evidence that there may exist an immunoregulatory feedback circuit between the immune and neuroendocrine systems.http://dx.doi.org/10.1155/S0962935192000036
spellingShingle Y. Hirooka
T. Mitsuma
T. Nogimori
Y. Ishizukis
Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceils
Mediators of Inflammation
title Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceils
title_full Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceils
title_fullStr Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceils
title_full_unstemmed Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceils
title_short Effect of hydrocortisone on interleukin-6 production in human ,peripheral blood rnononuclear ceils
title_sort effect of hydrocortisone on interleukin 6 production in human peripheral blood rnononuclear ceils
url http://dx.doi.org/10.1155/S0962935192000036
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AT tmitsuma effectofhydrocortisoneoninterleukin6productioninhumanperipheralbloodrnononuclearceils
AT tnogimori effectofhydrocortisoneoninterleukin6productioninhumanperipheralbloodrnononuclearceils
AT yishizukis effectofhydrocortisoneoninterleukin6productioninhumanperipheralbloodrnononuclearceils