Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parameters

BackgroundHuman papillomavirus (HPV) infection, especially high-risk types like HPV16 and HPV18, is a primary cause of cervical cancer. The p53 gene influences cellular response to DNA damage and has a functional polymorphism (rs1042522, p.Arg72Pro) that affects susceptibility to degradation by HPV...

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Main Authors: Cheng Sun, Jun Zhang, Lili Pan, Shuang Yao, Fenghua Zhang, Linjuan Ji, Miaomei Yu, Guanghua Luo, Xiping Jiang
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-05-01
Series:Frontiers in Oncology
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Online Access:https://www.frontiersin.org/articles/10.3389/fonc.2025.1541928/full
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author Cheng Sun
Jun Zhang
Lili Pan
Shuang Yao
Fenghua Zhang
Linjuan Ji
Miaomei Yu
Guanghua Luo
Guanghua Luo
Xiping Jiang
author_facet Cheng Sun
Jun Zhang
Lili Pan
Shuang Yao
Fenghua Zhang
Linjuan Ji
Miaomei Yu
Guanghua Luo
Guanghua Luo
Xiping Jiang
author_sort Cheng Sun
collection DOAJ
description BackgroundHuman papillomavirus (HPV) infection, especially high-risk types like HPV16 and HPV18, is a primary cause of cervical cancer. The p53 gene influences cellular response to DNA damage and has a functional polymorphism (rs1042522, p.Arg72Pro) that affects susceptibility to degradation by HPV E6 protein. This study aims to analyze the relationship among p53 genotypes, high-risk HPV infection, and hematological parameters in cervical cancer development and to develop a predictive model.MethodsThis retrospective cross-sectional study collected cervical cancer specimens and brush samples from patients at the First People’s Hospital of Changzhou between January 2020 and August 2024. HPV types and p53 genotyping were performed using PCR. Inflammatory markers like neutrophil-to-lymphocyte ratio (NLR), systemic immune-inflammation index (SII), and platelet-to-lymphocyte ratio (PLR) were calculated. Statistical analyses including logistic regression and LASSO were used to construct a predictive model.ResultsThe study included 147 female patients with cervical cancer and controls. HPV16 and HPV18 had high infection rates. In the log-additive model, each additional p53 C allele reduced the risk by 48% (OR = 0.52, 95% CI: 0.27-0.98, P = 0.038). Significant interactions were found between p53 genotypes and HPV18 infection on cervical cancer risk (P = 0.026). Cervical cancer patients showed reduced red blood cell count and hemoglobin. The predictive model, including p53 genotype, HPV16, HPV18, and hematological parameters, had an AUC of 0.920 (95% CI: 0.875–0.965).ConclusionThe study identified significant differences in p53 genotypes, HPV infection, and hematological parameters between cervical cancer patients and controls. The predictive model demonstrated high discriminatory ability for cervical cancer risk assessment. The interaction between HPV18 and p53 genotypes suggests a potential protective effect of the p53 C allele. Larger studies are needed to validate these findings.
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spelling doaj-art-d06ed7563e784a4aa06a829af4295dc62025-08-20T03:07:26ZengFrontiers Media S.A.Frontiers in Oncology2234-943X2025-05-011510.3389/fonc.2025.15419281541928Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parametersCheng Sun0Jun Zhang1Lili Pan2Shuang Yao3Fenghua Zhang4Linjuan Ji5Miaomei Yu6Guanghua Luo7Guanghua Luo8Xiping Jiang9Department of Gynecology, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaClinical Medical Research Center, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaClinical Medical Research Center, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaClinical Medical Research Center, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaDepartment of Gynecology, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaDepartment of Gynecology, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaClinical Medical Research Center, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaClinical Medical Research Center, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaChangzhou Medical Center, Nanjing Medical University, Changzhou, ChinaDepartment of Gynecology, The First People’s Hospital of Changzhou and the Third Affiliated Hospital of Soochow University, Changzhou, ChinaBackgroundHuman papillomavirus (HPV) infection, especially high-risk types like HPV16 and HPV18, is a primary cause of cervical cancer. The p53 gene influences cellular response to DNA damage and has a functional polymorphism (rs1042522, p.Arg72Pro) that affects susceptibility to degradation by HPV E6 protein. This study aims to analyze the relationship among p53 genotypes, high-risk HPV infection, and hematological parameters in cervical cancer development and to develop a predictive model.MethodsThis retrospective cross-sectional study collected cervical cancer specimens and brush samples from patients at the First People’s Hospital of Changzhou between January 2020 and August 2024. HPV types and p53 genotyping were performed using PCR. Inflammatory markers like neutrophil-to-lymphocyte ratio (NLR), systemic immune-inflammation index (SII), and platelet-to-lymphocyte ratio (PLR) were calculated. Statistical analyses including logistic regression and LASSO were used to construct a predictive model.ResultsThe study included 147 female patients with cervical cancer and controls. HPV16 and HPV18 had high infection rates. In the log-additive model, each additional p53 C allele reduced the risk by 48% (OR = 0.52, 95% CI: 0.27-0.98, P = 0.038). Significant interactions were found between p53 genotypes and HPV18 infection on cervical cancer risk (P = 0.026). Cervical cancer patients showed reduced red blood cell count and hemoglobin. The predictive model, including p53 genotype, HPV16, HPV18, and hematological parameters, had an AUC of 0.920 (95% CI: 0.875–0.965).ConclusionThe study identified significant differences in p53 genotypes, HPV infection, and hematological parameters between cervical cancer patients and controls. The predictive model demonstrated high discriminatory ability for cervical cancer risk assessment. The interaction between HPV18 and p53 genotypes suggests a potential protective effect of the p53 C allele. Larger studies are needed to validate these findings.https://www.frontiersin.org/articles/10.3389/fonc.2025.1541928/fullcervical cancerblood routine parametersp53high-risk HPVnomogram
spellingShingle Cheng Sun
Jun Zhang
Lili Pan
Shuang Yao
Fenghua Zhang
Linjuan Ji
Miaomei Yu
Guanghua Luo
Guanghua Luo
Xiping Jiang
Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parameters
Frontiers in Oncology
cervical cancer
blood routine parameters
p53
high-risk HPV
nomogram
title Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parameters
title_full Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parameters
title_fullStr Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parameters
title_full_unstemmed Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parameters
title_short Innovative nomogram for cervical cancer prediction: integrating high-risk HPV infection, p53 genotype, and blood routine parameters
title_sort innovative nomogram for cervical cancer prediction integrating high risk hpv infection p53 genotype and blood routine parameters
topic cervical cancer
blood routine parameters
p53
high-risk HPV
nomogram
url https://www.frontiersin.org/articles/10.3389/fonc.2025.1541928/full
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