miR-361-5p contributes to the pathogenesis of Alzheimer’s disease

Abstract Alzheimer’s disease (AD) is a progressive neurodegenerative disorder. This study investigated the roles of HOMER1, ATAD1, and miR-361 in AD pathogenesis using microarray (GSE106241, GSE157239; n = 60) and RT-PCR (n = 100; 50 AD patients, 50 controls from Northwest Iran) analyses. Decreased...

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Main Authors: Abbas Jalaiei, Jalal Gharesouran, Shahram Arsang-Jang, Mahnaz Talebi, Maryam Rezazadeh, Soudeh Ghafouri-Fard
Format: Article
Language:English
Published: Nature Portfolio 2025-08-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-17112-z
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author Abbas Jalaiei
Jalal Gharesouran
Shahram Arsang-Jang
Mahnaz Talebi
Maryam Rezazadeh
Soudeh Ghafouri-Fard
author_facet Abbas Jalaiei
Jalal Gharesouran
Shahram Arsang-Jang
Mahnaz Talebi
Maryam Rezazadeh
Soudeh Ghafouri-Fard
author_sort Abbas Jalaiei
collection DOAJ
description Abstract Alzheimer’s disease (AD) is a progressive neurodegenerative disorder. This study investigated the roles of HOMER1, ATAD1, and miR-361 in AD pathogenesis using microarray (GSE106241, GSE157239; n = 60) and RT-PCR (n = 100; 50 AD patients, 50 controls from Northwest Iran) analyses. Decreased expression of HOMER1 and ATAD1, key regulators of glutamatergic synapses, and miR-361, a potential regulator of both, was observed in AD brain tissue (GSE106241, categorized into seven Braak stages), suggesting a link between their dysregulation, impaired synaptic function, and increased neuroinflammation. However, blood-based RT-PCR showed no significant difference in HOMER1 or ATAD1. miR-361 was significantly lower in AD patients (adjusted p < 0.043). These findings, limited by sample size and lacking a formal power analysis, require further investigation to validate their potential as peripheral biomarkers for AD. Future studies with larger sample sizes are warranted.
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institution Kabale University
issn 2045-2322
language English
publishDate 2025-08-01
publisher Nature Portfolio
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series Scientific Reports
spelling doaj-art-cfeaf42d83bf4c82bb8bb5477b45d56e2025-08-24T11:19:18ZengNature PortfolioScientific Reports2045-23222025-08-0115111110.1038/s41598-025-17112-zmiR-361-5p contributes to the pathogenesis of Alzheimer’s diseaseAbbas Jalaiei0Jalal Gharesouran1Shahram Arsang-Jang2Mahnaz Talebi3Maryam Rezazadeh4Soudeh Ghafouri-Fard5Department of Medical Genetics, Faculty of Medicine, Tabriz University of Medical SciencesDepartment of Medical Genetics, Faculty of Medicine, Tabriz University of Medical SciencesCancer Gene Therapy Research Center, Zanjan University of Medical SciencesNeurosciences Research Center (NSRC), Tabriz University of Medical SciencesDepartment of Medical Genetics, Faculty of Medicine, Tabriz University of Medical SciencesDepartment of Medical Genetics, Shahid Beheshti University of Medical SciencesAbstract Alzheimer’s disease (AD) is a progressive neurodegenerative disorder. This study investigated the roles of HOMER1, ATAD1, and miR-361 in AD pathogenesis using microarray (GSE106241, GSE157239; n = 60) and RT-PCR (n = 100; 50 AD patients, 50 controls from Northwest Iran) analyses. Decreased expression of HOMER1 and ATAD1, key regulators of glutamatergic synapses, and miR-361, a potential regulator of both, was observed in AD brain tissue (GSE106241, categorized into seven Braak stages), suggesting a link between their dysregulation, impaired synaptic function, and increased neuroinflammation. However, blood-based RT-PCR showed no significant difference in HOMER1 or ATAD1. miR-361 was significantly lower in AD patients (adjusted p < 0.043). These findings, limited by sample size and lacking a formal power analysis, require further investigation to validate their potential as peripheral biomarkers for AD. Future studies with larger sample sizes are warranted.https://doi.org/10.1038/s41598-025-17112-zAlzheimer’s diseaseHOMER1ATAD1MIR-361Glutamatergic synapses
spellingShingle Abbas Jalaiei
Jalal Gharesouran
Shahram Arsang-Jang
Mahnaz Talebi
Maryam Rezazadeh
Soudeh Ghafouri-Fard
miR-361-5p contributes to the pathogenesis of Alzheimer’s disease
Scientific Reports
Alzheimer’s disease
HOMER1
ATAD1
MIR-361
Glutamatergic synapses
title miR-361-5p contributes to the pathogenesis of Alzheimer’s disease
title_full miR-361-5p contributes to the pathogenesis of Alzheimer’s disease
title_fullStr miR-361-5p contributes to the pathogenesis of Alzheimer’s disease
title_full_unstemmed miR-361-5p contributes to the pathogenesis of Alzheimer’s disease
title_short miR-361-5p contributes to the pathogenesis of Alzheimer’s disease
title_sort mir 361 5p contributes to the pathogenesis of alzheimer s disease
topic Alzheimer’s disease
HOMER1
ATAD1
MIR-361
Glutamatergic synapses
url https://doi.org/10.1038/s41598-025-17112-z
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AT mahnaztalebi mir3615pcontributestothepathogenesisofalzheimersdisease
AT maryamrezazadeh mir3615pcontributestothepathogenesisofalzheimersdisease
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