Intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer

Abstract Metastatic prostate cancer (PCa) is not curable due to its ability to acquire therapy resistance. Theoretically, acquired therapy resistance can be driven by changes to previously sensitive cancer cells or their environment and/or by outgrowth of a subpopulation of cancer cells with primary...

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Main Authors: Dena P. Rhinehart, Jiaying Lai, David E. Sanin, Varsha Vakkala, Adrianna Mendes, Christopher Bailey, Emmanuel S. Antonarakis, Channing J. Paller, Xiaojun Wu, Tamara L. Lotan, Rachel Karchin, Laura A. Sena
Format: Article
Language:English
Published: Nature Portfolio 2024-12-01
Series:npj Precision Oncology
Online Access:https://doi.org/10.1038/s41698-024-00773-w
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author Dena P. Rhinehart
Jiaying Lai
David E. Sanin
Varsha Vakkala
Adrianna Mendes
Christopher Bailey
Emmanuel S. Antonarakis
Channing J. Paller
Xiaojun Wu
Tamara L. Lotan
Rachel Karchin
Laura A. Sena
author_facet Dena P. Rhinehart
Jiaying Lai
David E. Sanin
Varsha Vakkala
Adrianna Mendes
Christopher Bailey
Emmanuel S. Antonarakis
Channing J. Paller
Xiaojun Wu
Tamara L. Lotan
Rachel Karchin
Laura A. Sena
author_sort Dena P. Rhinehart
collection DOAJ
description Abstract Metastatic prostate cancer (PCa) is not curable due to its ability to acquire therapy resistance. Theoretically, acquired therapy resistance can be driven by changes to previously sensitive cancer cells or their environment and/or by outgrowth of a subpopulation of cancer cells with primary resistance. Direct demonstration of the latter mechanism in patients with PCa is lacking. Here we present a case report as proof-of-principle that outgrowth of a subpopulation of cancer cells lacking the genomic target and present prior to therapy initiation can drive acquired resistance to targeted therapy and threaten survival in patients with PCa.
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publishDate 2024-12-01
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series npj Precision Oncology
spelling doaj-art-cfa69be4bab4442d968155aec14047a42025-08-20T02:30:51ZengNature Portfolionpj Precision Oncology2397-768X2024-12-01811510.1038/s41698-024-00773-wIntratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancerDena P. Rhinehart0Jiaying Lai1David E. Sanin2Varsha Vakkala3Adrianna Mendes4Christopher Bailey5Emmanuel S. Antonarakis6Channing J. Paller7Xiaojun Wu8Tamara L. Lotan9Rachel Karchin10Laura A. Sena11The Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityInstitute for Computational Medicine at Johns Hopkins UniversityThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityUniversity of Minnesota Masonic Cancer CenterThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityInstitute for Computational Medicine at Johns Hopkins UniversityThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins UniversityAbstract Metastatic prostate cancer (PCa) is not curable due to its ability to acquire therapy resistance. Theoretically, acquired therapy resistance can be driven by changes to previously sensitive cancer cells or their environment and/or by outgrowth of a subpopulation of cancer cells with primary resistance. Direct demonstration of the latter mechanism in patients with PCa is lacking. Here we present a case report as proof-of-principle that outgrowth of a subpopulation of cancer cells lacking the genomic target and present prior to therapy initiation can drive acquired resistance to targeted therapy and threaten survival in patients with PCa.https://doi.org/10.1038/s41698-024-00773-w
spellingShingle Dena P. Rhinehart
Jiaying Lai
David E. Sanin
Varsha Vakkala
Adrianna Mendes
Christopher Bailey
Emmanuel S. Antonarakis
Channing J. Paller
Xiaojun Wu
Tamara L. Lotan
Rachel Karchin
Laura A. Sena
Intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer
npj Precision Oncology
title Intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer
title_full Intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer
title_fullStr Intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer
title_full_unstemmed Intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer
title_short Intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer
title_sort intratumoral heterogeneity drives acquired therapy resistance in a patient with metastatic prostate cancer
url https://doi.org/10.1038/s41698-024-00773-w
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