The progress of the microbe-gut-brain axis in sepsis-associated encephalopathy

Sepsis-associated encephalopathy (SAE) is a diffuse brain dysfunction that is caused by sepsis without direct brain injury or central nervous system infection and is manifested as anxiety-like behavior and cognitive dysfunction. The microbiota-gut-brain axis, on the other hand, is a bidirectional co...

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Main Authors: Chen He, Hui Shi, Zhijie Yu, Chunhan Ma, Zhiqiang Jiao, Jin Li, Fei Yang
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-05-01
Series:Frontiers in Cellular and Infection Microbiology
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Online Access:https://www.frontiersin.org/articles/10.3389/fcimb.2025.1587463/full
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author Chen He
Chen He
Hui Shi
Zhijie Yu
Chunhan Ma
Zhiqiang Jiao
Jin Li
Fei Yang
Fei Yang
author_facet Chen He
Chen He
Hui Shi
Zhijie Yu
Chunhan Ma
Zhiqiang Jiao
Jin Li
Fei Yang
Fei Yang
author_sort Chen He
collection DOAJ
description Sepsis-associated encephalopathy (SAE) is a diffuse brain dysfunction that is caused by sepsis without direct brain injury or central nervous system infection and is manifested as anxiety-like behavior and cognitive dysfunction. The microbiota-gut-brain axis, on the other hand, is a bidirectional communication network between the gut and the brain that modulates host behavior and cognitive function in many ways and is of central importance in the preservation of general health and homeostasis. Given the functional roles attributed to the microbiota-gut-brain axis (MGBA), contemporary research is progressively focused on elucidating relationships between SAE and alterations in compositional and quantitative intestinal microbiota profiles. This review consolidates interdisciplinary insights from immunology, microbiology, neuroendocrine signaling, and neural pathophysiology to evaluate the mechanistic contribution of the MGBA to the relief of cognitive impairments in SAE. By unifying these perspectives, with the aim of preventing or enhancing SAE-related neurological dysfunction for the formulation of MGBA-targeted therapeutic strategies.
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institution Kabale University
issn 2235-2988
language English
publishDate 2025-05-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Cellular and Infection Microbiology
spelling doaj-art-cf1b819fcb154d958cf9f50902c406e92025-08-20T03:52:47ZengFrontiers Media S.A.Frontiers in Cellular and Infection Microbiology2235-29882025-05-011510.3389/fcimb.2025.15874631587463The progress of the microbe-gut-brain axis in sepsis-associated encephalopathyChen He0Chen He1Hui Shi2Zhijie Yu3Chunhan Ma4Zhiqiang Jiao5Jin Li6Fei Yang7Fei Yang8Chifeng Clinical Medical College of Inner Mongolia Medical University, Hohhot, ChinaDepartment of Critical Care Medicine, Chifeng Municipal Hospital, Chifeng, ChinaDepartment of Critical Care Medicine, Chifeng Municipal Hospital, Chifeng, ChinaDepartment of Emergency Medicine, Chifeng Municipal Hospital, Chifeng, ChinaChifeng Clinical Medical College of Inner Mongolia Medical University, Hohhot, ChinaChifeng Clinical Medical College of Inner Mongolia Medical University, Hohhot, ChinaChifeng Clinical Medical College of Inner Mongolia Medical University, Hohhot, ChinaChifeng Clinical Medical College of Inner Mongolia Medical University, Hohhot, ChinaDepartment of Critical Care Medicine, Chifeng Municipal Hospital, Chifeng, ChinaSepsis-associated encephalopathy (SAE) is a diffuse brain dysfunction that is caused by sepsis without direct brain injury or central nervous system infection and is manifested as anxiety-like behavior and cognitive dysfunction. The microbiota-gut-brain axis, on the other hand, is a bidirectional communication network between the gut and the brain that modulates host behavior and cognitive function in many ways and is of central importance in the preservation of general health and homeostasis. Given the functional roles attributed to the microbiota-gut-brain axis (MGBA), contemporary research is progressively focused on elucidating relationships between SAE and alterations in compositional and quantitative intestinal microbiota profiles. This review consolidates interdisciplinary insights from immunology, microbiology, neuroendocrine signaling, and neural pathophysiology to evaluate the mechanistic contribution of the MGBA to the relief of cognitive impairments in SAE. By unifying these perspectives, with the aim of preventing or enhancing SAE-related neurological dysfunction for the formulation of MGBA-targeted therapeutic strategies.https://www.frontiersin.org/articles/10.3389/fcimb.2025.1587463/fullsepsis-associated encephalopathymicrobe-gut-brain axisgut microbiotaneuroimmuneneuroendocrine
spellingShingle Chen He
Chen He
Hui Shi
Zhijie Yu
Chunhan Ma
Zhiqiang Jiao
Jin Li
Fei Yang
Fei Yang
The progress of the microbe-gut-brain axis in sepsis-associated encephalopathy
Frontiers in Cellular and Infection Microbiology
sepsis-associated encephalopathy
microbe-gut-brain axis
gut microbiota
neuroimmune
neuroendocrine
title The progress of the microbe-gut-brain axis in sepsis-associated encephalopathy
title_full The progress of the microbe-gut-brain axis in sepsis-associated encephalopathy
title_fullStr The progress of the microbe-gut-brain axis in sepsis-associated encephalopathy
title_full_unstemmed The progress of the microbe-gut-brain axis in sepsis-associated encephalopathy
title_short The progress of the microbe-gut-brain axis in sepsis-associated encephalopathy
title_sort progress of the microbe gut brain axis in sepsis associated encephalopathy
topic sepsis-associated encephalopathy
microbe-gut-brain axis
gut microbiota
neuroimmune
neuroendocrine
url https://www.frontiersin.org/articles/10.3389/fcimb.2025.1587463/full
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