Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infections

Introduction: Staphylococcus aureus-induced bloodstream infections (BSIs) remain a prevalent clinical challenge and the underlying pathogenesis is still poorly understood. The aim of this study was to investigate the inflammatory responses and histopathological changes in BSIs in mice. Methodology:...

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Main Authors: Dan Wu, Shusheng Zhou, Shijing Hu, Bao Liu
Format: Article
Language:English
Published: The Journal of Infection in Developing Countries 2017-04-01
Series:Journal of Infection in Developing Countries
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Online Access:https://jidc.org/index.php/journal/article/view/7800
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author Dan Wu
Shusheng Zhou
Shijing Hu
Bao Liu
author_facet Dan Wu
Shusheng Zhou
Shijing Hu
Bao Liu
author_sort Dan Wu
collection DOAJ
description Introduction: Staphylococcus aureus-induced bloodstream infections (BSIs) remain a prevalent clinical challenge and the underlying pathogenesis is still poorly understood. The aim of this study was to investigate the inflammatory responses and histopathological changes in BSIs in mice. Methodology: Male C57BL/6 mice were inoculated with S. aureus intravenously to induce BSIs. The survival rate, weight loss, and murine sepsis scores (MSS) were monitored in BSI and phosphate-buffered saline (PBS) control mice. Blood samples and tissue homogenates were plated on agar plates to determine the bacterial burden. Inflammatory proteins and cytokines were determined by enzyme-linked immunosorbent assay (ELISA) kits. Histopathologic changes were assessed by pathological inflammation score (PIS) and macroscopic and microscopic examinations. Results: BSI mice induced by 4.5 × 108 CFU/mL S. aureus showed ~70% survival rate, higher sepsis scores, significantly decreased body weight, elevated levels of white blood cell (WBC) counts, C-reactive protein (CRP), procalcitonin (PCT), interleukin (IL)-1β, IL-6, and tumor necrosis factor (TNF)-α. Prominent correlations were found between elevated CRP and PCT levels as well as among IL-1β, IL-6, and TNF-α. Pathological changes and higher PIS were also observed in BSI mice. Conclusions: Our results demonstrate that inflammatory proteins (PCT and CRP) and cytokines (IL-6, IL-1β and TNF-α) play an important role in the inflammatory responses and histopathological changes in S. aureus-induced BSIs.
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spelling doaj-art-cf0605bc70064e2d8fbf83e55ca8001b2025-08-20T02:27:20ZengThe Journal of Infection in Developing CountriesJournal of Infection in Developing Countries1972-26802017-04-01110410.3855/jidc.7800Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infectionsDan Wu0Shusheng Zhou1Shijing Hu2Bao Liu3Nanjing First Hospital, Nanjing Medical University, Nanjing, ChinaAffiliated Provincial Hospital of Anhui Medical University, Hefei, ChinaAffiliated Provincial Hospital of Anhui Medical University, Hefei, ChinaAffiliated Provincial Hospital of Anhui Medical University, Hefei, ChinaIntroduction: Staphylococcus aureus-induced bloodstream infections (BSIs) remain a prevalent clinical challenge and the underlying pathogenesis is still poorly understood. The aim of this study was to investigate the inflammatory responses and histopathological changes in BSIs in mice. Methodology: Male C57BL/6 mice were inoculated with S. aureus intravenously to induce BSIs. The survival rate, weight loss, and murine sepsis scores (MSS) were monitored in BSI and phosphate-buffered saline (PBS) control mice. Blood samples and tissue homogenates were plated on agar plates to determine the bacterial burden. Inflammatory proteins and cytokines were determined by enzyme-linked immunosorbent assay (ELISA) kits. Histopathologic changes were assessed by pathological inflammation score (PIS) and macroscopic and microscopic examinations. Results: BSI mice induced by 4.5 × 108 CFU/mL S. aureus showed ~70% survival rate, higher sepsis scores, significantly decreased body weight, elevated levels of white blood cell (WBC) counts, C-reactive protein (CRP), procalcitonin (PCT), interleukin (IL)-1β, IL-6, and tumor necrosis factor (TNF)-α. Prominent correlations were found between elevated CRP and PCT levels as well as among IL-1β, IL-6, and TNF-α. Pathological changes and higher PIS were also observed in BSI mice. Conclusions: Our results demonstrate that inflammatory proteins (PCT and CRP) and cytokines (IL-6, IL-1β and TNF-α) play an important role in the inflammatory responses and histopathological changes in S. aureus-induced BSIs. https://jidc.org/index.php/journal/article/view/7800Staphylococcus aureusbloodstream infectionsinflammationcytokinesHistopathology
spellingShingle Dan Wu
Shusheng Zhou
Shijing Hu
Bao Liu
Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infections
Journal of Infection in Developing Countries
Staphylococcus aureus
bloodstream infections
inflammation
cytokines
Histopathology
title Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infections
title_full Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infections
title_fullStr Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infections
title_full_unstemmed Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infections
title_short Inflammatory responses and histopathological changes in a mouse model of Staphylococcus aureus-induced bloodstream infections
title_sort inflammatory responses and histopathological changes in a mouse model of staphylococcus aureus induced bloodstream infections
topic Staphylococcus aureus
bloodstream infections
inflammation
cytokines
Histopathology
url https://jidc.org/index.php/journal/article/view/7800
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AT shushengzhou inflammatoryresponsesandhistopathologicalchangesinamousemodelofstaphylococcusaureusinducedbloodstreaminfections
AT shijinghu inflammatoryresponsesandhistopathologicalchangesinamousemodelofstaphylococcusaureusinducedbloodstreaminfections
AT baoliu inflammatoryresponsesandhistopathologicalchangesinamousemodelofstaphylococcusaureusinducedbloodstreaminfections