Telomere length associations with cognition depend on Alzheimer's disease biomarkers

Abstract Introduction While telomere shortening, a marker of cellular aging, may impact the progression of age‐related neurodegenerative diseases, its association with cognition is unclear, particularly in the context of Alzheimer's disease (AD) pathology. Methods Telomere, cognitive, and CSF d...

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Main Authors: Emily R. Mahoney, Logan Dumitrescu, Mabel Seto, Kelly N.H. Nudelman, Rachel F. Buckley, Katie A. Gifford, Andrew J. Saykin, Angela J. Jefferson, Timothy J. Hohman, Alzheimer's Disease Neuroimaging Initiative
Format: Article
Language:English
Published: Wiley 2019-01-01
Series:Alzheimer’s & Dementia: Translational Research & Clinical Interventions
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Online Access:https://doi.org/10.1016/j.trci.2019.11.003
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author Emily R. Mahoney
Logan Dumitrescu
Mabel Seto
Kelly N.H. Nudelman
Rachel F. Buckley
Katie A. Gifford
Andrew J. Saykin
Angela J. Jefferson
Timothy J. Hohman
Alzheimer's Disease Neuroimaging Initiative
author_facet Emily R. Mahoney
Logan Dumitrescu
Mabel Seto
Kelly N.H. Nudelman
Rachel F. Buckley
Katie A. Gifford
Andrew J. Saykin
Angela J. Jefferson
Timothy J. Hohman
Alzheimer's Disease Neuroimaging Initiative
author_sort Emily R. Mahoney
collection DOAJ
description Abstract Introduction While telomere shortening, a marker of cellular aging, may impact the progression of age‐related neurodegenerative diseases, its association with cognition is unclear, particularly in the context of Alzheimer's disease (AD) pathology. Methods Telomere, cognitive, and CSF data from 482 participants in the AD Neuroimaging Initiative (148 cognitively normal, 283 mild cognitive impairment, 51 AD) was leveraged to assess telomere length associations with cognition (measured by memory and executive function) and interactions with CSF amyloid‐β, tau, and APOE‐ε4. Secondary analyses assessed brain volume and thickness outcomes. Results Longer telomeres at baseline were associated with faster executive function decline. Amyloid‐β and tau interacted with telomere length on cognition, with longer telomeres related to faster decline among biomarker‐positive individuals. Discussion Telomere associations with cognition shift with AD progression, with longer telomeres related to worse outcomes as pathology increases, highlighting the need for further investigation of telomere length along the AD neuropathological cascade.
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series Alzheimer’s & Dementia: Translational Research & Clinical Interventions
spelling doaj-art-ce24736d0fc1497fbc9f2eac8fa14d6f2025-08-20T02:09:55ZengWileyAlzheimer’s & Dementia: Translational Research & Clinical Interventions2352-87372019-01-015188389010.1016/j.trci.2019.11.003Telomere length associations with cognition depend on Alzheimer's disease biomarkersEmily R. Mahoney0Logan Dumitrescu1Mabel Seto2Kelly N.H. Nudelman3Rachel F. Buckley4Katie A. Gifford5Andrew J. Saykin6Angela J. Jefferson7Timothy J. Hohman8Alzheimer's Disease Neuroimaging InitiativeVanderbilt Memory and Alzheimer's Center, Vanderbilt University Medical CenterNashvilleTNUSAVanderbilt Memory and Alzheimer's Center, Vanderbilt University Medical CenterNashvilleTNUSAVanderbilt Memory and Alzheimer's Center, Vanderbilt University Medical CenterNashvilleTNUSAIndiana Alzheimer Disease Center, Indiana University School of MedicineIndianapolisINUSADepartment of NeurologyMassachusetts General HospitalBostonMAUSAVanderbilt Memory and Alzheimer's Center, Vanderbilt University Medical CenterNashvilleTNUSADepartment of Radiology & Imaging SciencesIndiana University School of MedicineIndianapolisINUSAVanderbilt Memory and Alzheimer's Center, Vanderbilt University Medical CenterNashvilleTNUSAVanderbilt Memory and Alzheimer's Center, Vanderbilt University Medical CenterNashvilleTNUSAAbstract Introduction While telomere shortening, a marker of cellular aging, may impact the progression of age‐related neurodegenerative diseases, its association with cognition is unclear, particularly in the context of Alzheimer's disease (AD) pathology. Methods Telomere, cognitive, and CSF data from 482 participants in the AD Neuroimaging Initiative (148 cognitively normal, 283 mild cognitive impairment, 51 AD) was leveraged to assess telomere length associations with cognition (measured by memory and executive function) and interactions with CSF amyloid‐β, tau, and APOE‐ε4. Secondary analyses assessed brain volume and thickness outcomes. Results Longer telomeres at baseline were associated with faster executive function decline. Amyloid‐β and tau interacted with telomere length on cognition, with longer telomeres related to faster decline among biomarker‐positive individuals. Discussion Telomere associations with cognition shift with AD progression, with longer telomeres related to worse outcomes as pathology increases, highlighting the need for further investigation of telomere length along the AD neuropathological cascade.https://doi.org/10.1016/j.trci.2019.11.003APOEExecutive functionCognitionCSF biomarkersTelomeres
spellingShingle Emily R. Mahoney
Logan Dumitrescu
Mabel Seto
Kelly N.H. Nudelman
Rachel F. Buckley
Katie A. Gifford
Andrew J. Saykin
Angela J. Jefferson
Timothy J. Hohman
Alzheimer's Disease Neuroimaging Initiative
Telomere length associations with cognition depend on Alzheimer's disease biomarkers
Alzheimer’s & Dementia: Translational Research & Clinical Interventions
APOE
Executive function
Cognition
CSF biomarkers
Telomeres
title Telomere length associations with cognition depend on Alzheimer's disease biomarkers
title_full Telomere length associations with cognition depend on Alzheimer's disease biomarkers
title_fullStr Telomere length associations with cognition depend on Alzheimer's disease biomarkers
title_full_unstemmed Telomere length associations with cognition depend on Alzheimer's disease biomarkers
title_short Telomere length associations with cognition depend on Alzheimer's disease biomarkers
title_sort telomere length associations with cognition depend on alzheimer s disease biomarkers
topic APOE
Executive function
Cognition
CSF biomarkers
Telomeres
url https://doi.org/10.1016/j.trci.2019.11.003
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