Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytes

Objective This study investigates how astaxanthin (AST) counters tert-butyl hydroperoxide (tBHP)-induced cellular damage in C28/I2 chondrocytes, focusing on the circ-HP1BP3/miR-139-5p/SOD1 signaling pathway and its use in sustained-release microspheres for osteoarthritis treatment.Methods We employe...

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Main Authors: Wenwei Liang, Gang Liu, Weibo Zhou, Wei Chen, Yaojun Lu, Hao Wu, Yao Qin, Chunhui Zhu
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Redox Report
Subjects:
Online Access:https://www.tandfonline.com/doi/10.1080/13510002.2024.2422271
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author Wenwei Liang
Gang Liu
Weibo Zhou
Wei Chen
Yaojun Lu
Hao Wu
Yao Qin
Chunhui Zhu
author_facet Wenwei Liang
Gang Liu
Weibo Zhou
Wei Chen
Yaojun Lu
Hao Wu
Yao Qin
Chunhui Zhu
author_sort Wenwei Liang
collection DOAJ
description Objective This study investigates how astaxanthin (AST) counters tert-butyl hydroperoxide (tBHP)-induced cellular damage in C28/I2 chondrocytes, focusing on the circ-HP1BP3/miR-139-5p/SOD1 signaling pathway and its use in sustained-release microspheres for osteoarthritis treatment.Methods We employed a variety of techniques including real-time quantitative PCR, Western blot, ELISA, and dual-luciferase reporter gene assays to explore AST's molecular effects. Additionally, the efficacy of AST-loaded sustained-release microspheres was evaluated in vitro and in a mouse model of osteoarthritis.Results AST significantly enhanced SOD1 expression, reducing apoptosis and inflammation in damaged cells. The AST-loaded microspheres showed promising in vitro drug release, improved cell viability, and reduced oxidative stress. In the osteoarthritis mouse model, they effectively decreased joint inflammation and increased the expression of chondrocyte markers.Conclusion Astaxanthin effectively mitigates oxidative stress and inflammation in chondrocytes via the circ-HP1BP3/miR-139-5p/SOD1 pathway. The development of AST-loaded microspheres offers a novel and promising approach for osteoarthritis therapy, potentially extending to osteoarthritis treatment.
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institution OA Journals
issn 1351-0002
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language English
publishDate 2024-12-01
publisher Taylor & Francis Group
record_format Article
series Redox Report
spelling doaj-art-ce04bd9da9c4486384ea230cd30ee31b2025-08-20T02:21:07ZengTaylor & Francis GroupRedox Report1351-00021743-29282024-12-0129110.1080/13510002.2024.2422271Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytesWenwei Liang0Gang Liu1Weibo Zhou2Wei Chen3Yaojun Lu4Hao Wu5Yao Qin6Chunhui Zhu7Department of Orthopedics, The First Affiliated Hospital of Nanjing Medical University, Nanjing, People’s Republic of ChinaDepartment of Orthopedics, The People's Hospital of Puyang, Puyang, People’s Republic of ChinaTrauma Center, The Affiliated Changzhou Second People's Hospital of Nanjing Medical University, Changzhou, People’s Republic of ChinaTrauma Center, The Affiliated Changzhou Second People's Hospital of Nanjing Medical University, Changzhou, People’s Republic of ChinaTrauma Center, The Affiliated Changzhou Second People's Hospital of Nanjing Medical University, Changzhou, People’s Republic of ChinaTrauma Center, The Affiliated Changzhou Second People's Hospital of Nanjing Medical University, Changzhou, People’s Republic of ChinaDepartment of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, People’s Republic of ChinaTrauma Center, The Affiliated Changzhou Second People's Hospital of Nanjing Medical University, Changzhou, People’s Republic of ChinaObjective This study investigates how astaxanthin (AST) counters tert-butyl hydroperoxide (tBHP)-induced cellular damage in C28/I2 chondrocytes, focusing on the circ-HP1BP3/miR-139-5p/SOD1 signaling pathway and its use in sustained-release microspheres for osteoarthritis treatment.Methods We employed a variety of techniques including real-time quantitative PCR, Western blot, ELISA, and dual-luciferase reporter gene assays to explore AST's molecular effects. Additionally, the efficacy of AST-loaded sustained-release microspheres was evaluated in vitro and in a mouse model of osteoarthritis.Results AST significantly enhanced SOD1 expression, reducing apoptosis and inflammation in damaged cells. The AST-loaded microspheres showed promising in vitro drug release, improved cell viability, and reduced oxidative stress. In the osteoarthritis mouse model, they effectively decreased joint inflammation and increased the expression of chondrocyte markers.Conclusion Astaxanthin effectively mitigates oxidative stress and inflammation in chondrocytes via the circ-HP1BP3/miR-139-5p/SOD1 pathway. The development of AST-loaded microspheres offers a novel and promising approach for osteoarthritis therapy, potentially extending to osteoarthritis treatment.https://www.tandfonline.com/doi/10.1080/13510002.2024.2422271AstaxanthinC28/I2 cellstBHPCirc-HP1BP3Mir-139-5pSOD1
spellingShingle Wenwei Liang
Gang Liu
Weibo Zhou
Wei Chen
Yaojun Lu
Hao Wu
Yao Qin
Chunhui Zhu
Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytes
Redox Report
Astaxanthin
C28/I2 cells
tBHP
Circ-HP1BP3
Mir-139-5p
SOD1
title Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytes
title_full Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytes
title_fullStr Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytes
title_full_unstemmed Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytes
title_short Astaxanthin mediated repair of tBHP-Induced cellular injury in chondrocytes
title_sort astaxanthin mediated repair of tbhp induced cellular injury in chondrocytes
topic Astaxanthin
C28/I2 cells
tBHP
Circ-HP1BP3
Mir-139-5p
SOD1
url https://www.tandfonline.com/doi/10.1080/13510002.2024.2422271
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