Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac Fibrosis

Most cardiac diseases caused by inflammation are associated with fibrosis in the heart. Fibrosis is characterized by the accumulation of fibroblasts and excess deposition of extracellular matrix (ECM), which results in the distorted organ architecture and function. Recent studies revealed that cardi...

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Main Authors: Yasuhiro Yoshimatsu, Tetsuro Watabe
Format: Article
Language:English
Published: Wiley 2011-01-01
Series:International Journal of Inflammation
Online Access:http://dx.doi.org/10.4061/2011/724080
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author Yasuhiro Yoshimatsu
Tetsuro Watabe
author_facet Yasuhiro Yoshimatsu
Tetsuro Watabe
author_sort Yasuhiro Yoshimatsu
collection DOAJ
description Most cardiac diseases caused by inflammation are associated with fibrosis in the heart. Fibrosis is characterized by the accumulation of fibroblasts and excess deposition of extracellular matrix (ECM), which results in the distorted organ architecture and function. Recent studies revealed that cardiac fibroblasts are heterogeneous with multiple origins. Endothelial-mesenchymal transition (EndMT) plays important roles in the formation of cardiac fibroblasts during pathological settings. EndMT is regulated by signaling pathways mediated by cytokines including transforming growth factor (TGF)-β. Better understanding of the mechanisms of the formation of cardiac fibroblasts via EndMT may provide an opportunity to develop therapeutic strategies to cure heart diseases.
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spelling doaj-art-ccedc531cc5a468980b52f6bbcd7615b2025-08-20T02:19:52ZengWileyInternational Journal of Inflammation2042-00992011-01-01201110.4061/2011/724080724080Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac FibrosisYasuhiro Yoshimatsu0Tetsuro Watabe1Department of Molecular Pathology, Graduate School of Medicine, The University of Tokyo, Hongo, Bunkyo-ku,Tokyo 113-0033, JapanDepartment of Molecular Pathology, Graduate School of Medicine, The University of Tokyo, Hongo, Bunkyo-ku,Tokyo 113-0033, JapanMost cardiac diseases caused by inflammation are associated with fibrosis in the heart. Fibrosis is characterized by the accumulation of fibroblasts and excess deposition of extracellular matrix (ECM), which results in the distorted organ architecture and function. Recent studies revealed that cardiac fibroblasts are heterogeneous with multiple origins. Endothelial-mesenchymal transition (EndMT) plays important roles in the formation of cardiac fibroblasts during pathological settings. EndMT is regulated by signaling pathways mediated by cytokines including transforming growth factor (TGF)-β. Better understanding of the mechanisms of the formation of cardiac fibroblasts via EndMT may provide an opportunity to develop therapeutic strategies to cure heart diseases.http://dx.doi.org/10.4061/2011/724080
spellingShingle Yasuhiro Yoshimatsu
Tetsuro Watabe
Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac Fibrosis
International Journal of Inflammation
title Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac Fibrosis
title_full Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac Fibrosis
title_fullStr Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac Fibrosis
title_full_unstemmed Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac Fibrosis
title_short Roles of TGF-β Signals in Endothelial-Mesenchymal Transition during Cardiac Fibrosis
title_sort roles of tgf β signals in endothelial mesenchymal transition during cardiac fibrosis
url http://dx.doi.org/10.4061/2011/724080
work_keys_str_mv AT yasuhiroyoshimatsu rolesoftgfbsignalsinendothelialmesenchymaltransitionduringcardiacfibrosis
AT tetsurowatabe rolesoftgfbsignalsinendothelialmesenchymaltransitionduringcardiacfibrosis