Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐Met

Abstract The current study aimed to evaluate the expression and role of miR‐212 in the progression of pituitary adenoma (PA), thereby providing a theoretical basis and potential therapy methods for PA patients. Our data showed that miR‐212 levels were significantly reduced in PA tissues than normal...

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Main Authors: Hong‐Yan Wang, Huai‐Qing Gao
Format: Article
Language:English
Published: Wiley 2020-02-01
Series:Kaohsiung Journal of Medical Sciences
Subjects:
Online Access:https://doi.org/10.1002/kjm2.12137
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author Hong‐Yan Wang
Huai‐Qing Gao
author_facet Hong‐Yan Wang
Huai‐Qing Gao
author_sort Hong‐Yan Wang
collection DOAJ
description Abstract The current study aimed to evaluate the expression and role of miR‐212 in the progression of pituitary adenoma (PA), thereby providing a theoretical basis and potential therapy methods for PA patients. Our data showed that miR‐212 levels were significantly reduced in PA tissues than normal pituitary tissues. However, no significant difference was identified in the serum of PA patients and healthy control. In addition, the expression of miR‐212 in invasive PA was significantly lower than that in noninvasive and normal pituitary tissues. Moreover, the level of miR‐212 was decreased with the increase of tumor invasion. Meanwhile, the expression of miR‐212 in giant adenomas was significantly lower than that in macroadenomas and microadenomas. Furthermore, inhibition of miR‐212 significantly enhanced the proliferation and invasive capacity of GH3 cells. Dual luciferase reporter assay and western blot analysis confirmed that c‐Met was a target gene of miR‐212. More importantly, upregulation of c‐Met significantly prompted PA cell proliferation mainly as a result of the enhanced level of phosphorylation of AKT. This effect could be abolished when c‐Met was silenced in GH3 cells. In summary, reduced miR‐212 expression in PA contributed to abnormal cancer cell proliferation and invasion mainly by targeting c‐Met.
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spelling doaj-art-ccdf5aa98227430b92ea72dc9f94e6092025-08-20T02:08:45ZengWileyKaohsiung Journal of Medical Sciences1607-551X2410-86502020-02-01362818810.1002/kjm2.12137Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐MetHong‐Yan Wang0Huai‐Qing Gao1Department of Neurology Zibo Central Hospital Zibo Shandong Province ChinaDepartment of Neurology Zibo Central Hospital Zibo Shandong Province ChinaAbstract The current study aimed to evaluate the expression and role of miR‐212 in the progression of pituitary adenoma (PA), thereby providing a theoretical basis and potential therapy methods for PA patients. Our data showed that miR‐212 levels were significantly reduced in PA tissues than normal pituitary tissues. However, no significant difference was identified in the serum of PA patients and healthy control. In addition, the expression of miR‐212 in invasive PA was significantly lower than that in noninvasive and normal pituitary tissues. Moreover, the level of miR‐212 was decreased with the increase of tumor invasion. Meanwhile, the expression of miR‐212 in giant adenomas was significantly lower than that in macroadenomas and microadenomas. Furthermore, inhibition of miR‐212 significantly enhanced the proliferation and invasive capacity of GH3 cells. Dual luciferase reporter assay and western blot analysis confirmed that c‐Met was a target gene of miR‐212. More importantly, upregulation of c‐Met significantly prompted PA cell proliferation mainly as a result of the enhanced level of phosphorylation of AKT. This effect could be abolished when c‐Met was silenced in GH3 cells. In summary, reduced miR‐212 expression in PA contributed to abnormal cancer cell proliferation and invasion mainly by targeting c‐Met.https://doi.org/10.1002/kjm2.12137c‐MetinvasionmiR‐212pituitary adenoma
spellingShingle Hong‐Yan Wang
Huai‐Qing Gao
Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐Met
Kaohsiung Journal of Medical Sciences
c‐Met
invasion
miR‐212
pituitary adenoma
title Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐Met
title_full Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐Met
title_fullStr Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐Met
title_full_unstemmed Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐Met
title_short Reduction of miR‐212 contributes to pituitary adenoma cell invasion via targeting c‐Met
title_sort reduction of mir 212 contributes to pituitary adenoma cell invasion via targeting c met
topic c‐Met
invasion
miR‐212
pituitary adenoma
url https://doi.org/10.1002/kjm2.12137
work_keys_str_mv AT hongyanwang reductionofmir212contributestopituitaryadenomacellinvasionviatargetingcmet
AT huaiqinggao reductionofmir212contributestopituitaryadenomacellinvasionviatargetingcmet